Vitamin C deficiency and connective tissue. II. Determinations of hexosamine, uronic acid and hydroxyproline in guinea pig carcasses.
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Animals↗
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Results for “ASCORBIC ACID DEFICIENCY”
Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.
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[Effect of ethanol on the detoxification of hydrogen peroxide induced by ascorbic acid in normal erythrocytes and those with glucose-6-phosphate dehydrogenase deficiency].
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Ascorbic Acid↗
The dynamics of serum lipoprotein patterns in vitamin C-saturated and vitamin C-deficient guinea-pigs after carbon tetrachloride poisoning.
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Ascorbic Acid↗
Serum cholesterol in vitamin C deficiency in man.
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Ascorbic Acid Deficiency↗
Vitamin-C deficiency in the elderly and its manifestations.
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Aged↗
Early signs of vitamin A deficiency in Bulgaria.
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Adolescent↗
[The effect of vitamin C deficiency on calcium metabolism in guinea pigs].
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Animals↗
[Role of vitamin C deficiency in production of vascular diseases of the brain].
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Ascorbic Acid Deficiency↗
[Modification of blood viscosity in vitamin C deficiency and in certain other states].
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Ascorbic Acid↗
[Experimental arthritis and vitamin C deficiency. III].
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Animals↗
[Experimental arthritis and vitamin C deficiency. IV].
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Animals↗
[Detection of systemic vitamin C deficiency on the basis of color field changes].
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Ascorbic Acid Deficiency↗
[Pathogenesis of vitamin deficiency in patients with chronic gastritis and peptic ulcer].
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Ascorbic Acid Deficiency↗
Some observations on the effects of vitamin C deficiency on bone.
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Animals↗
[Chronic latent deficiency of vitamin C: metabolic and pathophysiologic consequences].
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Adult↗
[Post-heparin lipolytic activity of plasma, secretion rate of triglycerides and kinetics of plasma triglycerides in guinea pigs with chronic borderline vitamin C deficiency].
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Animals↗
Correction of anemia and iron deficiency in vegetarians by administration of ascorbic acid.
Twenty-eight strict vegetarians were given 500 mg ascorbic acid twice daily after lunch and dinner for two months. Hemoglobin and certain iron status parameters were measured before and after the treatment. Ascorbate treatment increased mean hemoglobin by 8%, serum iron by 17% and transferrin saturation by 23% and decreased total iron binding capacity by 7%. All these changes were statistically significant. The rise in serum ferritin was 12%. The serum protein or copper level did not indicate their dietary deficiency, while initial serum ascorbate level were low which rose by 60% on therapy. It is concluded that ascorbate supplementation is a better method of improving hematologic and iron status than iron salt administration.
Adolescent↗
Oral contraceptives and ascorbic acid.
Plasma, leukocyte, and platelet ascorbic acid levels are decreased in women ingesting oral contraceptive steroids. Studies have shown that it is the estrogenic component of the oral contraceptive agents that is associated with the decresased ascorbic acid concentrations. Urinary excretion of ascorbic acid does not appear to be increased by the steroids. Although serum levels of copper are increased by estrogens and oral contraceptives, ascorbic acid catabolism does not appear to be increased (unpublished). Our preliminary data on tissue uptake of ascorbic acid suggest that changes in tissue distribution are one possible answer for the observed effects of the steroids on blood levels of ascorbic acid.
Adrenal Glands↗