[Aneurysms of pancreaticoduodenal and hepatic arteries].
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Diagnostic and therapeutic nerve blocks are potent tools that can be utilized as one component of the armamentarium in the overall management of cancer pain. With the advent of the multidisciplinary approach to cancer pain management, the appropriate role of nerve blocks should be examined in terms of timing and risk-benefit ratio for each individual patient. Improved patient selection, prior utilization of other lower risk techniques, improved understanding of patient goals, and changes in pain experience may result in higher success. The ultimate goal is to provide comfort without sacrificing other functions important to the patient and/or family.
The authors report 4 cases of abdominal aortic hypoplasia and, reviewing the literature, recall the anatomical classifications and the clinical aspects predominated by arterial hypertension and arterial claudication. Pathogenesis does not seem to be univocal and some arguments are in favor of a congenital origin when others plead for an acquired inflammatory disease. But above forty one must consider the possible responsibility of added atheroma. The numerous operative techniques point out the difficulties encountered in the treatment of the aortic lesions and associated arterial, mainly renal lesions. The polymorphism and complexity of these lesions oblige to vary the choice of indications.
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Numerous studies in man and animals suggest that beta-adrenergic receptors stimulate pancreatic polypeptide (PP) release whereas alpha-adrenergic receptors inhibit PP release. This study was undertaken to further clarify the role of the adrenergic nervous system in regulating PP release. We evaluated the effects of stellatectomy and celiacectomy on resting and meat meal-stimulated PP release in the dog. PP release was studied in three stages--control, poststellatectomy, and poststellatectomy plus celiacectomy in five dogs. The meat meal caused a prompt and prolonged increase in plasma levels of PP. Stellatectomy did not alter the control PP response. However, celiacectomy increased basal and peak PP levels and enhanced the early and late phases of delta-integrated PP release. The data suggest that the celiac and superior mesenteric ganglia are the main source of the adrenergic innervation of the pancreas and that this innervation is primarily an inhibitor of PP release.
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