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PubMed1995

Family cohesion as a buffer against the effects of problem-drinking fathers on psychological distress, deviant behavior, and heavy drinking in adolescents.

In this paper we examine the degree to which family cohesion buffers the effects of fathers' problem drinking at Time 1 (T1) on adolescent distress, deviance, and heavy drinking at Time 2 (T2), one year later. Data from a representative sample of 658 families were used to test the hypotheses. Mothers, fathers (if present), and adolescent children were interviewed in the home. Fathers who were present completed self-report scales measuring problem drinking. When fathers were not available, mothers' reports on fathers' drinking were used to measure fathers' problem drinking. Results from regression analysis indicate that after controlling for the effects of race, SES, age, gender, and family structure: (1) the more cohesion in the family and the fever stressful events, the less distress, deviance, and heavy drinking shown by adolescents; (2) the fathers' problem drinking affects adolescent distress and deviance when cohesion is low; but as cohesion increases, the effects of the fathers' drinking are reduced. The findings support the hypothesis that cohesion in families buffers the effects of fathers' problem drinking on adolescents.

Adolescent↗

PubMed1996

Demographic and psychosocial risk for alcohol use: ethnic differences.

OBJECTIVE: We examined the influence of demographic variables, and social (parents and peers), attitudinal and intentions variables regarding alcohol use on actual drinking behavior among Asian and white populations. METHOD: Asian (n = 148; 79 female, 69 male) and white (n = 132; 72 female, 60 male) college students completed a questionnaire. RESULTS: Confirmatory factor analyses revealed that social and attitudinal factors reflected a common construct of Psychosocial Vulnerability which, in a structural equation model, was significantly predicted by ethnicity. The white population was exposed to more psychosocial risks to alcohol use compared to the Asian population. Ethnicity, however, did not directly predict either drinking intentions or drinking behavior, after the effects on Psychosocial Vulnerability were considered. CONCLUSIONS: Our findings suggest that ethnic differences in alcohol use between Asians and whites are mainly due to different levels of exposure to risk factors. Effective prevention programs must consider, not only psychosocial factors, but also certain contextual factors such as sex and ethnicity.

Adolescent↗

PubMed1998

Alcohol use disorder is a risk factor for mortality among older public housing residents.

Epidemiologic survey data were used to examine relationships between alcohol use and abuse and the physical and mental health status of elderly public housing residents (weighted n = 865) and to determine the influence that drinking behavior had on mortality. Residents with a current alcohol disorder (4%) were more likely to rate their physical health as fair/poor but had fewer major medical illnesses, functional impairments, and other current psychiatric disorders. Individuals with a current or past alcohol disorder (22%) were more likely than others to die (odds ratio [OR] = 7.5) during the 28-month follow-up period. In multivariate analyses, women with a past alcohol disorder were more likely than lifetime abstainers to die (OR = 21.9). Drinking behavior was not predictive of death in men. The high prevalence of alcohol disorder and its strong influence on mortality in this predominantly African-American female population demonstrate the need for programs designed to prevent and treat alcoholism in public housing developments for the elderly.

Black or African American↗

PubMed1998

Mode effects on self-reported alcohol use and problem drinking: mail questionnaires and personal interviewing compared.

OBJECTIVE: Much attention is paid to the influence of different data collection methods on the quality of self-reported drinking behavior estimates. Thus far, however, the findings show inconsistencies. Therefore, a comprehensive study was conducted to compare data on alcohol use and alcohol-related problems obtained by mail survey and personal interviews. METHOD: A general population survey on alcohol was conducted among a random sample of 8,000 Dutch inhabitants of Rotterdam aged 16 to 69. A small sample (n = 500) of the total sample (N = 8,000) was personally interviewed and the others (n = 7,500) received a mailed questionnaire. The response rate was 44% (N = 3,537). Respondents of the mail survey and personal interviews are compared on overall response rate, item-nonresponse rate, background factors, self-reported alcohol use, alcohol-related problems and problem drinking. RESULTS: No notable differences in self-reported alcohol use, alcohol-related problems or problem drinking were found by data collection mode. This holds for both the total general population and for men and women separately. The overall response rate was somewhat higher for the personal interviews. No important significant differences were found in item nonresponse or background factors. CONCLUSIONS: The absence of notable differences in estimated self-reported drinking behavior by mail survey and personal interviews indicates that both data collection methods yield comparable results. This is true for both the total population and for men and women separately.

Adolescent↗

PubMed1998

Appetitive and consummatory behaviors in the control of ethanol consumption: a measure of ethanol seeking behavior.

Models of ethanol self-administration in animals have demonstrated that ethanol can reinforce a variety of behaviors, independent of ethanol's caloric or fluid properties. However, the processes that control self-administration remain unclear. Determining factors related to ethanol seeking behavior, independent of consumption, is central to the concepts of intake regulation. The model described in this article proposes a method to separate the initial appetitive (seeking) behavior from the following consummatory (drinking) behavior to assess each behavior type. Rats were trained to lever press to gain access to a drinking tube connected to a fluid bottle containing either 10% ethanol or 3% sucrose for 20 min. When the response requirement to obtain access to the tube was increased, it was found that both solutions supported the same amount of responding (breakpoint was at approximately a fixed ratio 32 requirement), indicating equal reinforcer strength. However, regardless of the response requirement, if access to the fluids occurred, intakes were not changed. This suggests that factors besides those of reinforcer efficacy are important in controlling the size of the consummatory bout. Based on these findings, we believe that this model will be useful in determining factors related to seeking behaviors and the control of drinking bout size.

Alcohol Drinking↗

PubMed1996

Drinking and blood pressure responses to central injection of L-NAME in conscious rats.

The drinking behavior and blood pressure responses to i.c.v. administration of artificial cerebrospinal fluid (aCSF) or NG-nitro-L-arginine methyl ester (L-NAME, 10, 250, or 500 micrograms), an inhibitor of nitric oxide synthase, were examined in conscious rats following either osmotic stimulation (1.0 M NaCl, 15 ml/kg, s.c.) or induction of hemorrhage (0.7 ml/min to a 20% blood volume loss). Water intake increased in all animals. L-NAME at doses of 250 and 500 micrograms, but not 10 micrograms, significantly attenuated water consumption induced by both stimuli. The mean arterial blood pressure (MABP), which increased after osmotic stimulation, was maintained at pressor levels by 250 and 500 micrograms of L-NAME, but decreased progressively and reached basal levels after treatment with aCSF and the lowest dose of L-NAME (i.e., 10 micrograms). Hemorrhage significantly decreased MABP in all rats. The fall in blood pressure associated with hemorrhage returned to control levels in animals treated with 250 and 500 micrograms of L-NAME but not in those treated with aCSF or 10 micrograms of L-NAME. These results indicate that nitric oxide is involved in the regulation of drinking behavior and may play an important role in the central control of blood pressure during osmotic stimulation and hypotensive hemorrhage.

Animals↗

PubMed1995

Trends in alcohol-related mortality among New Mexico's American Indians, Hispanics, and non-Hispanic whites.

Reduction of alcohol-related mortality is a national goal for health promotion and disease prevention. We conducted this analysis to determine whether trends in New Mexico's Hispanics, non-Hispanic Whites, and American Indians were consistent with national trends in alcohol-related mortality, and whether differences in drinking patterns could account for racial and ethnic differences in rates. Age-adjusted, race-specific, and ethnic-specific alcohol-related mortality rates and 95% confidence intervals were calculated for 5-year periods for 1958-1991 using New Mexico vital statistics data. We estimated the prevalence of acute and chronic at-risk drinking behaviors and abstinence from data collected by the Behavioral Risk Factor Surveillance System (BRFSS) for the period 1986-1992. We found that alcohol-related mortality rates varied substantially by race, ethnicity, sex, age, and calendar period. American Indians had the highest rates for both sexes. Rates increased sharply from the period 1958-1962 until the late 1970s and the early 1980s, and then began to decrease rapidly. However, during the most recent decade, the rates have followed contrasting trends in the three ethnic and racial groups. Although rates have continued to decline among non-Hispanic Whites, rates for Hispanics and American Indians have not declined, and still remain substantially higher than rates during the 1958-1962 period. Differences in at-risk drinking behaviors reported to the BRFSS do not explain the contrast in race-specific and ethnic-specific mortality rates. Although progress has been made in reducing national per capita alcohol consumption and alcohol-related mortality, certain high-risk racial and ethnic groups may not be sharing in the progress.

Adult↗

PubMed2005

[Study on the perception of influencing factors in the course of alcoholism--a comparison among alcoholics, families, and health care professionals].

(1) We prepared a questionnaire modified from that of Edwards, G. et al, to investigate the factors influencing drinking behaviors. Responses of the questionnaire were filled out by the alcoholics, their families, and health care professionals in four psychiatric hospitals having intensive alcoholic rehabilitation programmes. Clinical course of them were traced back for at least two years. (2) We examined the relationship between the course of alcoholism and the differences in perceptions among alcoholics, their families, and health care professionals. (3) We obtained sufficient responses from 76 alcoholics. Those were divided into two groups; one is a group of alcoholics with a history of continuous abstinence of over one year (50 cases), and the other is the group of less than one year of recent abstinence period (26 cases). (4) As the overall results from the participants, items on "self-help group" were favorably responded for the improvement of drinking behavior. Families tend to perceive more sensitively into the internal psychological changes of the alcoholics than the health care professionals, whereas the health care professionals tend to make general and common responses regardless of any alcoholics. We, health care professionals should be aware of such a limit to promote more efficient collaborative works with alcoholics and their families. (5) We compared these two groups ("over one year group" vs. "less than one year group") with regard to their demographic and family backgrounds, clinical features, treatment, self-help group attendance, employment, etc. (6) A significant accordance of responses among three groups (alcoholics, family, health care professionals) was found in the "over one year group" than in the "less than one year group". Such a high level of accordance is considered to reflect a favorable family adaptation along with the recovery from alcoholism, deep understanding on the part of health care professionals, progress in the treatment and in the relationship in a self-help group. (7) As regards the "less than one year" group, it includes more single individuals and less frequent attendances to self-help groups. For this group of alcoholics, group therapy in a smaller group, individualistically oriented approaches for their needs including various supportive resources in the society should be addressed.

Aged↗

PubMed1988

Unemployment and alcohol abuse: a review.

Unemployment rates in alcohol treatment programs are strikingly high, yet the drinking behavior of unemployed populations has been neglected by alcohol researchers. Stress-based and socioenvironmental theories of alcoholism coupled with empirical research on the health and social costs of unemployment have suggested that the unemployed may be "at risk" for abusing alcohol. Specifically, the unemployed are said to abuse alcohol as a means of coping with financial stress triggered by job loss. Research on job loss and alcohol abuse has been beset by methodologic problems, which prevents drawing firm conclusions. For example, little attention has been paid to variables that intervene between job loss and alcohol abuse and that may actually account for the observed associations. Investigators have too often assumed that unemployment leads to alcohol abuse without considering the reverse. Controls for pre-job loss drinking behavior are virtually absent in studies to date. If future research does confirm that job loss leads to alcohol abuse, then alcohol policy implications may include: (1) the use of job loss as a diagnostic marker by alcohol treatment professionals, (2) the establishment of cooperative arrangements between those in the employment and training and alcohol fields, (3) a re-examination of the policy which ties health insurance coverage for alcohol treatment to current employment, and (4) the targeting of prevention and treatment resources to areas of high unemployment.

Alcoholism↗

PubMed2003

Pharmacological studies of alcohol susceptibility and brain monoamine function in stroke-prone spontaneously hypertensive rats (SHRSP) and stroke-resistant spontaneously hypertensive rats (SHRSR).

Differences of alcohol drinking behavior, brain dopamine (DA) and serotonin (5-HT) levels and releases in the striatum were investigated in stroke-prone spontaneously hypertensive rats (SHRSP) and age-matched stroke-resistant spontaneously hypertensive rats (SHRSR). Voluntary alcohol (EtOH) consumption in SHRSP rats increased at 1 and 2 hours in the 4 hour time access. In the DA level, SHRSP showed decreases in the caudate-putamen (C/P) and dorsal raphe nucleus (DRN) compared with in SHRSR. 5-HT levels in the C/P, ventral tegmental area-subtantia nigra (V/S) and DRN of the SHRSP were decreased compared with that in SHRSR. The basal extracellular levels of 5-HT release in the C/P were increased in SHRSP as compared with those in SHRSR. K(+)- or EtOH-induced DA and 5-HT releases in the C/P of the SHRSP were a lower magnitude than those in SHRSR. Increased basal extracellular 5-HT releases showing low levels of 5-HT in the C/P of SHRSP mean an abnormality of serotonergic neuronal functions in a normal physiological condition. Higher voluntary alcohol drinking behavior, so called lower susceptibility to EtOH, in the SHRSP may be associated with the degenerated rewarding system including the DRN. These results suggest that the hypertensive state causes the dysfunction in the striatum of the brain rewarding system and induces the risk for increasing alcohol consumption to compensate for the alteration of serotonergic neurons.

Alcohol Drinking↗

PubMedDate not supplied

General and alcohol-specific social support following treatment.

Both general and alcohol-specific support have been shown, albeit inconsistently, to affect drinking behavior. The discrepant findings may be clarified by examining how they work together. In exploratory analysis of clients following private outpatient alcoholism treatment, we found that the two variables add uniquely to the explanation of the variance in proportion of days abstinent (PDA). Both contribute significantly in the short term (3 months posttreatment), but only alcohol-specific support helps to explain variance over the longer term (15 months posttreatment), and alcohol-specific support explains more of the variance in PDA than general support at both time periods. More complex relationships are operating when short-term treatment effects have diminished. Alcohol-specific support mediates the relationship between general support and PDA, and both general social support and alcohol-specific support are moderators of one another in their relationships to PDA. Knowing how different types of social support affect drinking behavior at different intervals following treatment may help treatment providers to better prepare their clients for the posttreatment social environment.

Adult↗

PubMed2004

Effect of champagne compared to still white wine on peripheral neurotransmitter concentrations.

To evaluate how the peripheral release of neurotransmitters such as serotonin, dopamine, cholecystokinin, and beta-endorphin is involved in drinking behavior, blood concentrations of these neurotransmitters were followed in 40 healthy young volunteers during the first hour after ingestion of a moderate dose of some common alcoholic beverages (champagne, still white wine) as compared to water. Concerning serotonin levels, two groups of subjects are statistically distinct: one with low basal serotonin levels (< 620 nmol/L) which responded with an increase in serotonin (52% in 10 minutes), and a second group with higher basal serotonin levels (> 620 nmol/L) which responded with a decrease ( 190% in 60 minutes). Variations in serotonin concentrations appear to depend upon the alcoholic content of the beverage. A rapid increase in plasma dopamine concentrations after consumption of champagne seems to be due to the nonalcoholic content of the beverage. Cholecystokinin values were not significantly different between the three beverages: the observed increase can be explained by a moderate gastric distention. Beta-endorphin levels didn't change significantly after drinking. In conclusion, some significant blood variations of serotonin and dopamine appeared even after moderately dose of champagne or still white wine. These changes might be partially responsible for the different drinking behavior.

Adult↗

PubMed1997

Effects of lifelong ethanol consumption on cerebellar layer volumes in AA and ANA rats.

Aging and chronic alcohol consumption can cause degenerative changes in the cerebellar cortex. In this study, the effects of aging and lifelong alcohol consumption on cerebellar cortical layer volumes (molecular and granular) and also white matter layer volumes were studied in alcohol-preferring (AA) and nonpreferring (ANA) rats of both sexes. The ethanol-consuming animals (EtOH) had 12% (w/v) ethanol as the only available fluid from 4 to 22 months of age, whereas the young (3 month) and old controls (24 months) had only water to drink. The volumes of molecular, granular, and white matter layers of the cerebellar vermis in folia II, IV, VII, and X were measured by using systematic sampling and a point-counting method. The volumes of the granular and white matter layers showed consistent increase between 3 and 24 months of age, whereas the volume of the molecular layer remained unchanged with increasing age. Individual ethanol intake was measured over a 1-week period at the beginning and at the end of chronic ethanol exposure. Significant (ANOVA, p = 0.000) sex difference was found in the drinking behavior in both lines, with females consuming more alcohol than males (daily ethanol consumption at 22 months of age 3.2 +/- 0.3 vs. 7.1 +/- 0.3 g/kg for AA males and females; 3.2 +/- 0.3 vs. 5.4 +/- 0.4 g/kg for ANA males and females, respectively). The only ethanol-induced effect on the cerebellum was observed in ANA-EtOH females with a 15% reduction in the volumes of the molecular and granular layer in folium II compared with age-matched controls and a significant (p < 0.05, analysis of covariance with ethanol intake as a covariate) line difference in folium II (molecular and granular layers) was observed between ANA-EtOH females and AA-EtOH females. Furthermore, the volume of the molecular layer in folium II was significantly (p < 0.05, analysis of covariance with ethanol intake and body weights as covariates) reduced for ANA-EtOH females, compared with ANA-EtOH males indicating a sex difference in the cerebellar degeneration due to chronic alcohol consumption. Of the three layers studied, the white matter layer was the most resistant layer to the effects caused by chronic alcohol consumption. In view of the fact that AA and ANA rats of both sexes differ regarding the drinking behavior and ethanol metabolism, they provide an important model for further research on ethanol-induced pathological changes in the central nervous system.

Acetaldehyde↗

PubMed1997

Models of alcohol use by young adults: an examination of various attitude-behavior theories.

OBJECTIVE: The aim of this study was to test the effectiveness of various attitude-behavior theories in explaining alcohol use among young adults. The theory of reasoned action (TRA), the theory of planned behavior and an extension of the TRA that incorporates past behavior were compared by the method of maximum-likelihood estimation, as implemented in LISREL for Windows 8.12. METHOD: Respondents consisted of 122 university students (82 female) who were questioned about their attitudes, subjective norms, perceived behavioral control, past behavior and intentions relating to drinking behavior. Students received course credit for their participation in the research. RESULTS: Overall, the results suggest that the extension of the theory of reasoned action which incorporates past behavior provides the best fit to the data. For these young adults, their intentions to drink alcohol were predicted by their past behavior as well as their perceptions of what important others think they should do (subjective norm). CONCLUSIONS: The main conclusions drawn from the research concern the importance of focusing on normative influences and past behavior in explaining young adult alcohol use. Issues regarding the relative merit of various alternative models and the need for greater clarity in the measure of attitudes are also discussed.

Adolescent↗

PubMed1999

[Conjugated forms of angiotensin II and beta-endorphin in systemic organization of goal-oriented behavior].

The effects of conjugated angiotensin II and beta-endorphin on the rat drinking behavior that was natural and formed due to preliminary learning were comparatively analyzed. Some behavioral responses were also studied after active immunization with the above agents. It is suggested that the conjugated forms of the regulatory peptides play a specific informational role in the systemic organization of goal-oriented behavior. These substances due to their conformational properties and involvement in the hierarchic organization of integrative processes in the nervous system are shown to be of great value.

Angiotensin II↗

PubMedDate not supplied

Paraventricular nucleus injections of peptide YY and neuropeptide Y preferentially enhance carbohydrate ingestion.

Neuropeptide Y (NPY) injected into the paraventricular nucleus (PVN) is known to elicit a powerful feeding response in satiated, brain-cannulated rats. The present experiment investigates the effect of peptide YY (PYY), a structurally-related peptide, on feeding behavior and, in addition, the effects of both PYY and NPY on the pattern of macronutrient selection. Injection of PYY directly into the PVN, in doses ranging from 7.8 to 235 pmol/0.3 microliters, caused a strong, dose-dependent stimulation of feeding behavior, as well as a small stimulation of drinking behavior, in satiated rats. The mean latency to eat was 9.3 min, with substantial feeding occurring within 30 min of the injection. At low doses, the increase in feeding was seen predominantly during the first hr. At the highest dose, in contrast, food intake continued to increase progressively over the next few hr. such that by 4 hr postinjection food intake was more than 20 g over vehicle baseline. In 1 hr tests with 3 pure macronutrient (protein, fat and carbohydrate) diets simultaneously available, PYY and NPY (78 pmol/0.3 microliters) both elicited a strong and selective increase in carbohydrate consumption, with little or no effect on protein or fat consumption. These results suggest that hypothalamic receptors sensitive to PYY and NPY may participate in the control of carbohydrate consumption.

Animals↗

PubMed2006

Behavioral effects of aminoadamantane class NMDA receptor antagonists on schedule-induced alcohol and self-administration of water in mice.

RATIONALE: Aminoadamantanes represent a class of NMDA glutamate receptor antagonists that reduce alcohol consumption and may prevent alcohol-induced neuronal adaptations and side effects. OBJECTIVE: Behavioral specificity of memantine and amantadine on alcohol drinking in a schedule-induced polydipsia (SIP) task was investigated in mice. METHODS: Male C57BL/6J mice were food-deprived and divided into four groups: 5% alcohol SIP, water SIP, 1 h limited access regulatory water drinking, and a control group to determine if either drug altered ethanol drinking. Behavioral specificity of memantine (5, 10, and 25 mg/kg, ip) and amantadine (20, 40, and 60 mg/kg, ip) was determined by comparing alterations in alcohol or water consumption in SIP and regulatory water drinking. Drug effects on SIP drinking-specific measures (grams per kilogram consumption) were also compared to nondrinking measures (locomotion, head-entries for food, and lick efficiency). RESULTS: Compared to saline, memantine reduced alcohol SIP drinking (10 and 25 mg/kg). Memantine increased locomotion during alcohol SIP (25 mg/kg) and during water SIP (5 and 25 mg/kg). In contrast, amantadine reduced both alcohol SIP (40 mg/kg) and water SIP (40 and 60 mg/kg). Both drugs reduced regulatory water consumption over the entire dose range tested. Blood alcohol concentrations indicated consumption of physiologically meaningful amounts of alcohol during SIP, and that changes in alcohol metabolism did not account for drug-induced reductions in alcohol drinking. CONCLUSIONS: In addition to reducing alcohol drinking, both drugs had other behavioral effects that included reductions in regulatory drinking. These results suggest that the therapeutic utility of these drugs for ameliorating human alcohol addiction remains questionable.

Alcohol Drinking↗

PubMed1986

Drinking and subsequent suppression of vasopressin is unaltered by naloxone in dogs.

The effect of the opioid antagonist naloxone on drinking and the subsequent suppression of plasma vasopressin were evaluated in seven dogs following 24 hr of water deprivation. Each animal underwent an intravenous injection of vehicle as a control and a low (0.05 mg/kg) and high (1 mg/kg) dose of naloxone. Plasma vasopressin was significantly (p less than 0.05) increased from a control value of 4.6 +/- 1.9 microU/ml to 9.9 +/- 3.1 microU/ml after the high dose of naloxone. Fluid intake was not altered by naloxone; 42 +/- 6 ml/kg for the control, 45 +/- 8 ml/kg at the low dose, and 49 +/- 7 ml/kg for the high dose. Six minutes after the onset of drinking vasopressin was reduced by 48% for the control, 41% for the low dose and 45% for the high dose, with no significant difference among treatments. Thus, in dehydrated dogs naloxone presumably blocks endogenous opioids, elevates vasopressin following dehydration, but does not affect drinking behavior or the subsequent suppression of vasopressin after drinking.

Animals↗