PubMed Health⌕ Search

SEARCH · PubMed Health

Results for “Broilers”

Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 91 records · Page 5Linked to original sources

A comparison of the immune performance of a 1991 commercial broiler with a 1957 randombred strain when fed "typical" 1957 and 1991 broiler diets.

The general objective of the present study was to assess the contribution that changes in genetic selection and dietary regimen have made on the immune performance of broilers. Chicks were hatched from 1991 and 1957 strains and placed on diets thought to be typical of those fed during 1957 and 1991. Immune responses were measured as total, IgM, and IgG antibody production, macrophage, and natural killer (NK) cell functions. Significant differences were observed between strains in antibody production. For example, 1957 males fed 1957 diets had the highest total (P < .0001), IgM (P < .0016), and IgG (P < .015) anti-sheep red blood cell antibodies as compared with all other strain-diet-sex groups. Both strains behaved similarly in terms of inflammatory macrophage recruitment, substrate adherence potential, and in the phagocytosis of sheep red blood cells. A greater percentage of the 1991 strain birds exhibited NK cell activity than all other groups. These studies suggest that genetic selection towards enhanced performance traits has negatively influenced the adaptive arm of the immune system (antibody production) with little or no effect on the nonadaptive components (macrophage and NK functions).

Animal Feed↗

Supplementation of broiler diets with all-rac-alpha- or a mixture of natural source RRR-alpha-,gamma-,delta-tocopheryl acetate. 1. effect on vitamin E status of broilers in vivo and at slaughter.

A total of 300 female broiler chickens were reared from day-old to 10 d of age on the same starter diet. Then they were divided into five groups, receiving a control diet (Group 1) relatively rich in fat (14.3%) and unsaturated fatty acids (87.6%) and standardized with respect to vitamins and minerals, supplemented with 100 mg (Group 2) and 500 mg (Group 4) of RRR-alpha-,gamma-,delta-tocopheryl acetate/kg feed (40.6% alpha-, 41.1% gamma-, 18.3% delta-) or 100 mg (Group 3) and 500 mg (Group 5) all-rac-alpha-tocopheryl acetate/kg feed until slaughter at 6 wk of age. No differences between the supplemented groups were observed with respect to weight gain, feed consumption, packed cell volume (PCV), plasma enzyme activities of creatine kinase (CK) and glutathione peroxidase (GSH-Px), fatty acid composition, and enzyme activities of citrate synthase (CS), and total lactate dehydrogenase (LDH), and 3-OH-acyl-coenzyme A-dehydrogenase (HAD) of breast (Pectoralis major) and thigh (Gastrocnemius interna) muscle. Increasing levels of alpha-, gamma-, and delta-tocopherol were found in blood plasma with increasing dietary levels of these tocopherols. Only alpha-tocopherol was detectable in skeletal muscle and in higher concentrations in thigh than in breast muscle. Hemolysis in vitro and plasma activity of aspartate aminotransferase (ASAT) were lower (P < .01) in Groups 2 and 4 than in Groups 3 and 5. Interactions were observed between dietary type and concentration of tocopherols for plasma CK, GSH-Px, Na+, and K+. No measurable excretion of ethane and pentane was observed in any of the groups. The findings indicate that the oxidative stress in the live animals was minimal. The mixture of natural source RRR-alpha-,gamma-,delta-tocopherols was as efficient in protecting the live chickens as the all-rac-alpha-tocopheryl acetate, when provided on a weight basis as judged from the chosen in vivo parameters of vitamin E status.

Abattoirs↗

Inclusion of oxidized vegetable oil in broiler diets. Its influence on nutrient balance and on the antioxidative status of broilers.

Over a period of 4 wk, 24 10-d-old broiler hens were fed diets containing 11% vegetable oil (9% rapeseed oil, 2% soybean oil), which was added either fresh (1 meq O2/kg oil) or oxidized (156 meq O2/kg oil). The effects of the dietary treatments on nutrient digestibility were examined in a balance experiment. The antioxidative status of the animals was evaluated using plasma concentrations of thiobarbituric acid-reactive substances (TBARS), erythrocyte hemolysis in vitro, selenium-dependent and selenium-independent activity of glutathione peroxidase in liver cell cytosolic fractions, and concentrations of tocopherols and other fat-soluble compounds with antioxidative properties (lutein, beta-carotene, and retinol) in plasma and various tissues (skeletal muscle, cardiac muscle, liver, and abdominal fat). Compared to the fresh oil, the concentrations of linoleic and linolenic acid were slightly lower in oxidized oil. The concentration of alpha-tocopherol in the diet with fresh oil was an average of 80.8 mg/kg diet, whereas the diet with oxidized oil only provided 44 mg/kg. The dietary selenium content averaged 0.48 mg/kg in both diets. During the experiment, none of the animals showed symptoms of diarrhea or vitamin E deficiency. The intake of oxidized oil caused a growth depression after 2 wk. The retention of fat (P = 0.07), energy (P = 0.09), and alpha-tocopherol (P < 0.01) was lower in the group fed oxidized fat. Furthermore, these animals showed significantly higher plasma concentrations of TBARS (P < 0.01), and lower concentrations of tocopherols, lutein, beta-carotene, and retinol in plasma and tissues.

Adipose Tissue↗

Broiler pulmonary hypertension syndrome. III. Commercial broiler strains differ in their susceptibility.

Broilers of 4 different commercial strains were kept during winter at an altitude of 1,350 m. The birds were slaughtered at weekly intervals and their relative right ventricular mass (pulmonary arterial pressure index = API values) determined. In addition, the incidence of ascites was recorded. Two of the strains suffered high losses from ascites, while the other 2 showed a certain degree of resistance. The former 2 groups had a higher mean API and a greater percentage of high API values in clinically normal birds than the other 2 groups. The group with the lowest incidence of ascites achieved the highest mean live mass at 51 days. There was also a decrease in mean API with age in clinically normal birds. There was no difference in the incidence of ascites between males and females.

Age Factors↗

Protein and energy relationships in the broiler chicken. 12. Dietary protein and triiodothyronine (T3) effects on the response of broilers to isoproterenol and cyclic adenosine monophosphate in vitro.

Indian River male broiler chickens (7-d-old) were fed on diets containing 120, 210 or 300 g crude protein/kg + 0 or 1 mg triiodothyronine (T3)/kg diet (Expt 1) and 120, 150, 180 or 210 g crude protein/kg + 0 or 1 mg T3/kg diet (Expt 2) to determine the effects of crude protein level and T3 on growth and metabolism. Body composition of chickens was determined by a combination of dissection of muscle and abdominal fat pads, and chemical extraction (Expt 1). In vitro lipogenesis (IVL) was determined in both experiments by incubating liver explants for 2 h at 37 degrees in the presence of 10(-4) M-dibutyryl cyclic AMP (cAMP) or 10(-5) M isoproterenol (ISO) and 10(-2) M-[2-14C]acetate. Acetate incorporation into total lipid was an indication of IVL. Activity ratios for each of these additions relative to control (-cAMP-ISO) were calculated to ascertain basal v. inhibited rates of IVL. The relative muscle mass was increased by increasing crude protein from 120 to 210 g/kg diet but not from 210 to 300 g/kg diet. Dietary T3 decreased total body lipid regardless of the dietary crude-protein level. Increasing dietary crude protein decreased (P < 0.05) basal IVL (-cAMP-ISO) but not IVL (+cAMP). Dietary T3 decreased basal IVL in birds fed on the diets containing 120 and 210 g crude protein/kg but had little effect on the two inhibited states of lipogenesis (+cAMP or +ISO). The component of lipogenesis sensitive to in vitro inhibition is also the component under dietary control.

Acetates↗

Use of a sperm analyzer for evaluating broiler breeder males. 2. Selection of young broiler breeder roosters for the sperm quality index increases fertile egg production.

Previous research has shown that the sperm quality index (SQI) of rooster semen is indicative of overall semen quality. The objectives of the present experiments were to determine the correlation of the SQI with semen characteristics and fertility and to determine if selection of young males for the SQI would improve fertility. In Experiment 1 semen was collected from 35 Peterson males and was analyzed individually for sperm concentration and viability. To determine fertility, 100 microL of diluted semen was inseminated into 10 hens for each rooster. Positive correlations of the SQI with total and live sperm concentrations as well as fertility were found. A negative correlation of the SQI with the percentage of dead sperm was observed. In Experiment 2, four semen samples were collected at 2- to 3-d intervals from each of 142, 27-wk-old Peterson roosters to determine their SQI. Males were then allocated to six treatment groups based on their average SQI readings as follows: 0 to 150, 151 to 200, 201 to 250, 251 to 300, 301 to 350, and >350. For each SQI group, semen was collected weekly for 8 wk, pooled, and used at a rate of 50 microL/hen to inseminate 40 hens. The percentage of fertilized eggs increased linearly across the SQI groups, from a minimum of 65% for the 0 to 150 SQI group to a maximum of 98% for the >350 SQI group. The SQI groups of 301 to 350 and >350 produced the slowest decline in fertility over days postinsemination. Therefore, selection of males for the SQI at an early age appears to improve flock fertility.

Animals↗

Sequential feeding of whole wheat to growing broiler chickens.

1. Four broiler feeding trials were performed to examine the suitability of a whole wheat sequential feeding regimen for commercial broiler production. The sequential feeding programme gave a continuous cycle of ad libitum access to only whole wheat followed by the same time of access to only a pelleted diet. The pelleted diet provided a concentration of nutrients to balance that provided by the whole wheat. This was called a balancer diet. 2. The first trial used 144 cage-reared broilers from 28 to 49 d of age. Four different times of access (4, 8, 12 and 24 h) to the two alternate foods were compared. A whole wheat choice-feeding treatment and a complete single diet treatment were also compared. Whole wheat accounted for over 40% of the broilers' total food intakes when they were given the sequential feeding treatments of 8 h or greater. The whole wheat intakes of the birds given the 4 h sequential feeding and the choice-feeding were only 20 and 5% respectively. There was a non linear relationship between the weight gains of the broilers and the length of the sequential feeding period (P < 0.01). The growth rates of the broilers given sequential feeding were lowest (P < 0.05) in the 4-h feeding periods but highest (P < 0.05) in the 8-h periods. Weight gains decreased (P < 0.01) linearly as the sequential feeding periods were increased above 8 h. 3. A second trial, using 144 cage-reared broilers, examined the effect of different balancer compositions or different wheat varieties in 8-h sequential feeding. The broilers selected more whole wheat in their diet when they were given balancers with increased cereal contents. However, these broilers did not eat enough whole wheat to compensate for the reduced cereal content of the balancers and their overall diets had lower energy:protein ratios. The two different wheat samples did not result in any differences (P > 0.05) in the proportion of whole wheat selected by the broilers. 4. A third trial compared the diet selections, weight gains, food intakes and water excretions of 72 cage-reared broilers given whole wheat feeding regimens. The growth rates of the broilers given a loose mix of whole wheat and a pelleted balancer diet were similar (P > 0.05) to broilers given a complete single diet. The growth rates of these two groups were 7% greater (P < 0.05) than broilers given choice-feeding or 8-h sequential feeding.(ABSTRACT TRUNCATED AT 400 WORDS)

Animal Feed↗

Persistence of vancomycin-resistant enterococci (VRE) in broiler houses after the avoparcin ban.

The glycopeptide growth promoter avoparcin was banned from animal production in the EU in 1997 due to concern for the spread of vancomycin-resistant enterococci (VRE) from food animals to humans. In recent Norwegian and Danish studies, extensive occurrence of VRE on broiler farms and in broiler flocks after the avoparcin ban has been reported. The present study was undertaken to investigate the epidemiology of VRE on broiler farms in the absence of the selective pressure exerted by avoparcin. Environmental samples were obtained from five broiler houses after depopulation, cleaning, and disinfection of the houses between rotations, and two consecutive broiler flocks from each house were sampled by taking cloacal swabs from the broilers at the time of slaughter. A total of 69 vancomycin-resistant Enterococcus faecium isolates obtained from broiler flocks and broiler houses were subjected to molecular typing by pulsed-field gel electrophoresis (PFGE). Forty-one PFGE-profiles were observed. VRE with indistinguishable or highly similar PFGE profiles were isolated from consecutive broiler flocks and from environmental samples from the houses in which the flocks were reared, whereas VRE-isolates from different broiler houses and from flocks reared in different houses appeared to be genetically unrelated. These findings indicated that VRE was transmitted between consecutive broiler flocks by clones of resistant bacteria surviving in the broiler houses despite cleaning and disinfection between rotations. Thus, the extensive occurrence of VRE in broiler flocks after the avoparcin ban may be explained by persistence of VRE in the broiler house environment.

Animal Feed↗