The "third coma" in diabetes: diabetic coma without ketoacidosis.
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Acidosis↗
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Results for “Coma”
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[Treatment of acido-ketosic diabetic coma and hyperosmolar coma in diabetics].
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Diabetic Coma↗
[Diabetic coma. Pathophysiology and treatment of diabetic coma].
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Diabetic Coma↗
[Emergencies in general medicine. Myxedematous coma, hypothyroid coma].
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Coma↗
[Science of symptomatology: coma. Etiology and mechanisms involved in coma].
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Coma↗
[Science of symptomatology: coma. Classification and physiopathology of coma].
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Coma↗
[Science of symptomatology: coma. Nursing observation and care of patients in coma].
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Coma↗
[Emergency nursing standard. Diabetic coma and hepatic coma].
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Diabetic Coma↗
Treatment of hepatic coma. A. Pathophysiology of hepatic coma.
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Blood Transfusion↗
Insulin coma and hibernation coma.
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Convulsive Therapy↗
[Modes of the origin of hepatic coma in liver cirrhosis. I. Incidence of hepatic coma in the course of liver cirrhosis].
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Hepatic Encephalopathy↗
Synergism between ammonia and fatty acids in the production of coma: implications for hepatic coma.
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Ammonia↗
Hepatic coma in cirrhosis of the liver. The course and prognosis of 100 consecutive coma episodes.
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Aged↗
Synergism between mercaptans and ammonia or fatty acids in the production of coma: a possible role for mercaptans in the pathogenesis of hepatic coma.
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Ammonia↗
Treatment of hepatic coma. Treatment of acute hepatic coma with plasmaphoresis and plasma exchange.
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Acute Disease↗
[Science of symptomatology: coma. Nursing problems and management of patients with hepatic coma].
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Hepatic Encephalopathy↗
[The cell separator in the therapy of hepatic coma: preliminary experience in cases of viral hepatitis coma].
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Cell Separation↗
[Acute hepatic coma. Experimental study on the predictive value of clinical-chemical findings for the prognosis of acute hepatic coma].
In order to determine the validity of clinical-chemical parameters for the prognosis of hepatic failure, 28 pigs were subjected to liver ischemia for 40--160 minutes duration. The following parameters were studied: GOT, GPT, gamma-GT, LAP, LDH, GlDH, AP and isoenzymes, total bilirubin, potassium, sodium and chloride. In a statistical comparison in the surviving animals, an unexplainable increase in GlDH activity was observed. In the other clinical-chemical parameters none was seen to be of use for the prognosis for either life or death in acute hepatic failure.
Acute Disease↗