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At least 91 records · Page 5Linked to original sources
PubMed1987

Management of behavioral symptoms in disturbed elderly patients: comparison of trifluoperazine and haloperidol.

This double-blind trial compared the efficacy of trifluoperazine 1 mg b.i.d. and haloperidol 0.5 mg b.i.d. as initial doses in the treatment of behavioral symptoms associated with chronic brain syndromes and senile psychoses in 54 institutionalized elderly patients. Forty-four of the patients were followed through the entire 6-week trial. Improvement in symptoms as measured by Clinical Global Impressions (CGI) scores was observed in 86% (N = 19) of those who received trifluoperazine and 90% (N = 20) of those given haloperidol. Twenty-seven percent (N = 6) of the trifluoperazine patients were rated at least moderately improved on the CGI, whereas only 9% (N = 2) of those treated with haloperidol exhibited such improvement. Marginally significant trends favoring trifluoperazine over haloperidol were observed in the ratings obtained on the Brief Psychiatric Rating Scale, the Nurses' Observation Scale for Inpatient Evaluation, and the Sandoz Clinical Assessment-Geriatric. The type and incidence of adverse reactions were similar for the two treatments, with sedation being the most common complaint. None of the patients developed any extrapyramidal motor symptoms.

Aged↗
PubMed1976

[Morphologic basis of psychoses associated with vascular and senile pathology].

The paper deals with a study of 100 patients diagnosed as combined senile and vascular psychoses. It is marked that in classical cases of such combinations the disorder has 3 stages. On the first stage there is a predominance of vascular pathology. After 60 years of age (the second stage) the senile sumptomatology disguises many symptoms of vascular lesions. The third stage is characterized by a vascular catastrophe. In many cases of combined psychoses there may be complications of other somatic pathology and mainly of lung and liver disorders.

Aged↗
PubMed1979

Mortality in psychiatric hospitals in Norway 1950--74.

The present investigation comprises all deaths in Norwegian psychiatric hospitals 1950--74: 10,413 deaths. Mortality in men declined from 361 per 10,000 before 1950 to 252 per 10,000 in 1969--74 and in women from 324 per 10,000 to 215 per 10,000 during the same periods. In the organic and symptomatic psychoses (mainly senile and arteriosclerotic) mortality ranged from six to ten times that of the general population, whereas in the non-organic (functional) psychoses mortality was only twice as high as in the general population. This discrepancy in mortality between organic and non-organic psychoses, which is caused by the somatic disorders with high lethality underlying the organic psychoses, suggests that mortality should be calculated separately for organic and non-organic psychoses, which is sometimes neglected. An increasing number of hospital admissions with organic, mainly senile, psychoses is to be expected in the future, as well as an increasing proportion of non-organic patients with slight psychotic symptoms and a low and possibly decreasing mortality. between 1950 and 1974 radical changes took place in the psychiatric hospitals which could have influenced mortality. Age-adjusted death rates from cardio-vascular diseases were actually higher in 1963--68 than in 1950--62, possibly indicating that an adverse effect of drug therapy on physical activity and somatic fitness had outweighed the stress-relieving effect. A significant rise in unnatural deaths (suicides and accidents) has been observed particularly since 1963. As in previous investigations from Norway 1926--41, cancer as cause of death was equal to or below the general population in the non-organic psychoses and somewhat higher in the organic psychoses.

Accidents↗
PubMed1977

[Role of endogenous and organic factors in the origin of endoform psychoses in the presence of senile-atrophic processes in the brain].

A clinical study of 50 patients with late schizophrenia and 150 with senile dementia demonstrated an involvement of the endogenous predisposition in the origin of schizoform psychoses in senile dementia, as well as close relation of these psychoses with atrophic processes. The author suggests that a slowly developing atrophic process creates specific, especially favourable conditions for the accomplishment of the endogenous predisposition in some cases and for the exacerbation and depending on the already existing endogenous disturbances in other cases.

Aged↗
PubMedDate not supplied

[Senile dementia].

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Alzheimer Disease↗
PubMed1960

Senile dementia.

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Alzheimer Disease↗