Fatal salicylate intoxication and stramonium poisoning.
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Considering the effect of crop-weed competition the rate of weed growing, the competitiveness of the occurring weed species and the duration of competition are determining factors. Experiments were carried out on fields in order to collect data on the effect of early weed competition on maize, including the competition for nutrients and the possible rate of nutrient removal by weeds. From 7 sampling areas of the 9.2 ha field weeds and maize samples were collected 1 month after the sowing of maize. We determined the total numbers and the species numbers of weeds by plots. The removed plant species and maize were weighed then dried until the weight balance was reached. The samples were tested for N, P, K and Ca. Comparison was done with the weight and nutrient element content of maize plants taken from the treated, weed-free area. At the same time comparative analyses were made with the mass and nutrient contents of maize plants. There were 12 occurring weed species in this experiment. Based on the rate of weed cover the following species were dominant: Datum stramonium L., Cannabis sativa L., Amaranthus chlorostachis Willd., Chenopodium album L., Chenopodium hybridum L. Our experiments revealed that in the areas being likely to produce high weed populations and showing a considerable high nutrient removal by weeds, the competition between weed plants and maize occurs at an earlier stage of the vegetation period of maize than on fields with moderate weed populations. Weeds have utilised significant amount of nutrients which has been many fold of maize in case of unit area.
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Jimson weed is a hallucinogenic plant that is common in rural areas. Consumption of any part of the plant can result in severe anticholinergic toxicity. The clinical presentation of jimson weed toxicity is similar to that seen in cases of atropine poisoning. Treatment is aimed at removing plant material from the gastrointestinal tract, keeping the patient safe and reversing severe anticholinergic sequelae.
The efficiency of in vitro translation of potato virus X (PVX) RNA within vRNP complexes assembled from genomic RNA and viral CP was examined. The vRNP particles contain the 5'-proximal RNA segments encapsidated by helically arranged CP head-like portions heterogeneous in length and the CP-free RNA tail. Translation of RNA is completely repressed upon incubation with PVX CP and is accompanied by vRNP particles production. By contrast, translation is activated in vRNPs in vitro assembled using two CP forms, differing in the principals of their N-terminal peptides modification. The N-terminal peptide of PVX CP represents the major phosphorylation site(s) for Thr/Ser-specific protein kinases. It was shown that: (i) CP phosphorylation results in a translational activation of vRNP; (ii) removal of N-terminal peptide from CP abolished activation and CP retains the translation repressing ability. It was suggested that substitution of Ser/Thr residues by non-phosphorylated Ala/Gly in N-terminal peptide of the mutant CP will led to a complete inhibition of vRNP translation. However, opposite results were obtained in our experiments: (i) RNA of such mutant virus (PVX-ST) was efficiently translated within the virions; (ii) RNA of a wild-type (wt) PVX also efficiently translated in mixedly assembled vRNP "wt PVX RNA + PVX-ST CP"; (iii) opposite result (repression of translation) was obtained with "mixed" vRNP (PVX-ST RNA + wtPVX CP). Therefore, the N-terminal peptide located at the surface of the particle and of the particles plays a key role in translation activation of the RNA encapsidated in vRNP and native virions.
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