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At least 1,063 records · Page 59Linked to original sources
PubMed1975

Retinal fat emboli as sequela to acute pancreatitis.

A clinical picture of Purtscher's retinopathy was observed in three patients with acute pancreatitis. In two patients, fluorescein angiography revealed arteriolar obstruction. Since fat emboli occur in pancreatitis, and since fat emboli may produce the picture of Purtscher's retinopathy, we proposed that fat emboli caused the retinal findings in these patients.

Acute Disease↗
PubMed1976

Techniques of argon laser photocoagulation of diabetic disk new vessels.

Four argon laser techniques have been evaluated in the eradication of diabetic disk neovascularization: focal (nonfeeder) photocoagulation, feeder-frond photocoagulation alone, panretinal photocoagulation alone, and feeder-frond treatment combined with panretinal photocoagulation. Focal photocoagulation was dangerous and the least effective technique in preventing recurrence of neovascularization. Feeder-frond photocoagulation was effective initially, but was associated with a high incidence of recurrent neovascularization. Panretinal photocoagulation and panretinal photocoagulation combined with feeder-frond treatment produced results superior to those obtained with the two earlier techniques. The best results were obtained with combined feeder-frond and panretinal photocoagulation done concurrently. Early diagnosis, aggressive photocoagulation therapy, and close follow-up were essential in order to obtain good results.

Adolescent↗
PubMed1976

Rapid resolution of venous stasis retinopathy after carotid endarterectomy.

A 60-year-old woman with ocular hypertension developed unilateral venous stasis retinopathy and neovascularization of the optic disk as the first signs of carotid occlusive disease. The retinopathy and the neovascularization disappeared rapidly and dramatically after carotid endarterectomy. Neovascularization in venous stasis retinopathy must be maintained by hypoxia or hypoperfusion, possibly through some intermediate substance such as a vasoproliferative factor.

Carotid Artery Thrombosis↗
PubMed1976

Severe proliferative retinopathy as the only sign of sickle cell hemoglobin C disease.

A 48-year-old black woman developed severe bilateral hypoxic proliferative retinopathy without other clinical manifestations. The hemoglobin level was 10.6 to 11.5 g/100 ml, reticulocyte level was 2.2%, targeted and sickled red blood cells were seen on blood smears, and hemoglobins S and C were demonstrated by electrophoresis. Glucose tolerance test was normal. The development of neovascular proliferation, vitreous hemorrhage, and retinal detachment unassociated with other clinical symptoms is unusual in sickle cell hemoglobin C disease.

Anemia, Sickle Cell↗
PubMedDate not supplied

Retinal branch vein occlusion.

Obstruction of a major temporal branch vein, or one of its macular tributaries, presents a significant threat to vision. Visual acuity may be reduced by macular edema or the consequences of retinal neovascularization, and these afflictions frequently become irreversible. Since the complicating macular edema and retinal neovascularization respond, at least in part, to argon laser therapy in some other conditions, some investigators have begun to treat branch vein occlusions with this modality. However, since the pathogenesis and natural history of the disorder have not yet been elucidated by prospective studies, it is not clear whether such treatment is indicated. Clinical and experimental studies are reviewed, and treatment rationale and techniques are discussed. The authors emphasize the need for well-controlled randomized studies to evaluate the natural history of branch vein occlusion and the efficacy of photocoagulation in its treatment.

Diagnosis, Differential↗
PubMed1984

Natural course of nonaphakic cystoid macular edema.

We evaluated the longterm natural history of nonaphakic cystoid macular edema (CME) in a retrospective study of 130 out of 557 CME cases recorded in the past ten years. A listing of causes was provided and the cases divided into two groups: those with perifoveal leakage and those with deep subretinal leakage. In cases of diabetic retinopathy, 60 patients who were followed up for more than three years had noncystoid or cystoid macular edema. The occurrence and persistence of a large central foveal cyst usually resulted in a severe decrease in visual acuity. Hard exudates, present in 60% of cases, seemed to influence visual prognosis when they were inside the foveal avascular zone. In cases of venous occlusion, chronic CME increased the risk of a central cyst and was the major cause of a macular scar. In cases of uveitis and vasculitis, the restoration of macular capillary wall competence was possible when inflammation decreased. Disturbances in the macular pigment epithelium were also shown to produce poor visual acuity.

Choroid Neoplasms↗
PubMedDate not supplied

Battered child syndrome in a four year old with previous diagnosis of Reye's syndrome.

A 4-year-old Black child was admitted to hospital in coma and with retinal hemorrhages. He was diagnosed as having Reye's Syndrome and remained hospitalized for 1 month. At discharge, he was neurologically compromised with spasticity of the right arm and leg, and could not walk or sit unsupported. Three weeks later he was again seen in the emergency room with a swollen right arm and leg and was subsequently readmitted. Healing fracture of the right femur and probable healing fracture of the right humerus were diagnosed. These were thought to be due to a fall from a stroller. Almost 4 months later, the child died at his home in the care of his mother's boyfriend. Autopsy findings were consistent with a severely chronically battered child.

Battered Child Syndrome↗
PubMed1993

Outcome of patients with diabetes mellitus and acute myocardial infarction treated with thrombolytic agents. The Thrombolysis and Angioplasty in Myocardial Infarction (TAMI) Study Group.

OBJECTIVES: This study was designed to assess outcome in patients with diabetes who received thrombolytic therapy and to determine whether differences in angiographic characteristics may account for the worse outcome observed in diabetic patients. BACKGROUND: Patients with diabetes are known to have a worse outcome after acute myocardial infarction than that of patients without diabetes. METHODS: Clinical and angiographic characteristics of the 148 patients with diabetes and the 923 patients without diabetes in the Thrombolysis and Angioplasty in Myocardial Infarction (TAMI) trials were examined and analyzed. RESULTS: Patients with diabetes tended to be older (median age 59 vs. 56 years) and to have a higher incidence of hypertension and hyperlipidemia and a lower incidence of cigarette smoking. Patients with diabetes had significantly more severe anatomic disease (66% vs. 46% had multivessel disease, p < 0.0001), similar global left ventricular ejection fraction (49% vs. 51%) and worse noninfarct zone ventricular function (-0.13 vs. 0.32 SD/chord, p = 0.02) than that of nondiabetic patients. Angiographic patency rates at 90 min after thrombolytic therapy were similar in patients with and without diabetes (initial 90-min patency 71% vs. 70%). Diabetic patients had nearly twice the in-hospital mortality rate (11% vs. 6%, p < 0.02) and a higher incidence of pulmonary edema (11% vs. 4%, p = 0.001). Diabetic women had an especially high in-hospital mortality rate (21%). No retinal hemorrhages were observed. Although diabetes as an unadjusted variable was predictive of in-hospital (p < 0.02) and long-term (p = 0.003) mortality, after adjustment for baseline clinical and angiographic characteristics, diabetes was not found to have an independent influence on mortality. CONCLUSIONS: Patients with diabetes after myocardial infarction have a worse outcome than that of patients without diabetes despite similar rates of infarct vessel patency. However, diabetes was not found to be an independent risk factor for increased mortality. These findings suggest that diabetes itself is not a major risk factor for poor early outcome after thrombolytic therapy for myocardial infarction; rather, the secondary effects such as more extensive coronary artery disease account for the worse outcome.

Aged↗
PubMed1974

Retinal change induced in the primate (Macaca mulatta) by oxygen nuclei radiation.

Retinas of primates (Macaca mulatta) were exposed to oxygen nuclei at the Bevatron, Berkeley, California. Color fundus photographs and fluorescein angiograms were taken of the retinas prior to irradiation and up to 5 weeks post exposure. Animals were sacrificed at post exposure intervals for histopathologic examination of the retinas. A series of animals were exposed to 200 kVp X-ray and examined on the same regime as the first series. The results showed a low rad equivalent dose for retinal damage as compared with the X-ray series, i.e., a high quality factor, and a marked compression of the latency between exposure and onset of the retinal pathology.

Animals↗