ENLARGEMENT OF ATRIAL SEPTAL DEFECT. A SIMPLIFIED TECHNIQUE.
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Due to the broad behaviour variability of the tricuspid atresia by the defects to which it is associated, the authors propose another classification, based on the position of the great vessels and the characteristics of the pulmonary flow, in addition to the extent of the intracardiac septal defects (atrial septal defect and ventricular septal defect) and the presence of patent ductus arterious. Eleven patients with tricuspid atresia with situs solitus and heart normo-placed are studied for diagnosis confirmation by catheterism or autopsy. Electrocardiograms are studied and observed in two patients with transposition of the great vessels and pulmonary oligohemia, a more evident right auricular enlargement (P of greater voltage in DII and AP heading right in the front plane), the AQRS deviated to the right, up and backwards, with less evidence of the left ventricular power than in other varieties of tricuspid atresia. These characteristics gave a diagnosis useful for this variation. On those patients without transposition and pulmonary flow increase, it is observed that growing appearance of the left auricle is more evident and so is the power of the free wall of the right ventricle (high R in right precordial deviations), the AQRS heads left as in most of the cases, but forward and upwards also. This electrocardiographic information is considered to have great importance to establish a diagnosis of the variations of tricuspid atresia due to associated malformations. Common varieties have already been extensively described by other authors and are not subject to special study. Bibliography on this matter is reviewed.
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A hitherto undescribed cardiac malformation, characterized by persistence of the primitive aortic conus and normal positioning of the great vessels, is reported and used to discuss the theory of bulbar resorption and the hypothesis of conal growth. The persistence of the primitive aortic conus is explained by the absence of bulbar contraction. The present selective inhibition of bulbar involution in normal positioning of the great vessels shows it to be a phenomenon formally independent of the transposition. Thus, the concept explaining the formal genesis of transposition is explained by arrest of torsion of the bulbar truncus is reconfirmed.
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