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PubMed1990

Low energy expenditures in free-living populations.

Low energy intakes are reported fairly frequently in populations where the implication is that these populations are free-living and that these intakes represent the norm. Intakes of energy, if they really reflect those pertaining to the average life-style, must also be reasonably indicative of energy expenditure. It is possible to define 'low' energy expenditure on the basis of obligatory metabolism (BMR + dietary induced thermogenesis) plus the energy needed for minimal physical activity--eg 2 h of standing activities and 1 h of walking daily. On that basis, the minimal realistic level is about 1.5 x BMR, and tables are given showing typical values for men and women over a range of body weights. If these values are accepted, considerable doubt must be placed on the interpretation of many reports in the literature of people apparently existing on 'low' energy intakes.

Adult↗
PubMed1993

[Recommendations of the Council for Nutrition on infant nutrition. Norwegian National Nutrition Council].

Because of the high frequency of nursing mothers there is concensus that, in general, the diet of Norwegian newborns is good for the first six months of life. However, several publications have questioned the recommendations concerning the babies' diet from the age of six to 12 months with respect to time of weaning and introduction of cow's milk, and intake of energy, fat-soluble vitamins, iron and essential fatty acids. The Norwegian National Nutrition Council has therefore evaluated the available data, and has adjusted some of the recommendations, including the recommendations for energy intake, energy percent from fat and essential polyunsaturated fatty acids.

Breast Feeding↗
PubMed1995

Macronutrients and energy balance in obesity.

Energy balance is the difference between metabolizable energy intake and total energy expenditure. Energy intake is difficult to measure accurately; changes in body weight, for example, are not a good measure of the adequacy of energy intake, because fluctuations in body weight are common even if the overall trend is toward weight loss. It is now customary to assess energy requirements indirectly from total energy expenditure. Total energy expenditure consists of basal metabolism, postprandial thermogenesis, and physical activity. Energy expenditure is related to both body weight and body composition. A reduction in total energy expenditure accompanies weight loss, because basal metabolic rate decreases with the loss of lean tissue mass. Similarly, with weight gain, there is an increase in basal metabolic rate, because lean tissue mass grows to support the increase in fat tissue mass. Excess energy intake over energy expenditure causes weight gain and an accompanying increase in total energy expenditure. Following a period of adaptation, total energy expenditure will match energy intake and body weight will stabilize at a higher level. This same relationship holds for weight loss. Respiratory quotient (measured in steady state) is an indication of the proportion of energy expenditure derived from fat and carbohydrate oxidation. Over long periods of time, fat balance is equivalent to energy balance, as an excess of fat intake over fat oxidation causes fat storage.

Body Weight↗
PubMed1998

Increased energy expenditure in infants with cyanotic congenital heart disease.

Infants with cyanotic congenital heart disease (CCHD) often have reduced weight gain compared with infants in control groups. Our purpose was to conduct a longitudinal study of energy intake, resting energy expenditure (REE), and total energy expenditure (TEE) of a group of infants with CCHD. We hypothesized that increased REE and TEE and decreased energy intake in these infants would lead to reduced growth. Ten infants with uncorrected CCHD and 12 infants in a control group were studied at 2 weeks of age and again at 3 months. Indirect calorimetry was used to determine REE; the doubly labeled water method was used to determine TEE and intake. At 2 weeks and 3 months of age, infants with CCHD weighed significantly less than infants in the control group. No significant difference was seen in energy intake or REE between groups during either period. TEE was slightly but not statistically increased in the CCHD group at 2 weeks (72.6 +/- 17.4 vs 59.8 +/- 10.9 kcal/kg/d) and significantly increased at 3 months (93.6 +/- 23.3 vs 72.2 +/- 13.2 kcal/kg/d, P </=.03). We conclude that increased TEE but not increased REE is a primary factor in the reduced growth in infants with CCHD.

Calorimetry, Indirect↗
PubMed2000

Energy requirements derived from total energy expenditure and energy deposition during the first 2 y of life.

BACKGROUND: Current recommendations for energy intake of children are derived from observed intakes. Deriving energy requirements on the basis of energy expenditure and deposition is scientifically more rational than is using the observational approach and is now possible with data on total energy expenditure (TEE), growth, and body composition. OBJECTIVES: The objectives of this study were 1) to define energy requirements during the first 2 y of life on the basis of TEE and energy deposition; 2) to test effects of sex, age, and feeding mode on energy requirements; and 3) to determine physical activity. DESIGN: TEE, sleeping metabolic rate, anthropometry, and body composition were measured in 76 infants. TEE was measured with doubly labeled water, sleeping metabolic rate with respiratory calorimetry, and body composition with a multicomponent model. RESULTS: Total energy requirements were 2.23, 2.59, 2.97, 3. 38, 3.72, and 4.15 MJ/d at 3, 6, 9, 12, 18, and 24 mo, respectively. Energy deposition (in MJ/d) decreased significantly over time (P: = 0.001) and was lower in breast-fed than in formula-fed infants (P: = 0.01). Energy requirements were approximately 80% of current recommendations. Energy requirements differed by age (P: = 0.001), feeding group (P: = 0.03), and sex (P: = 0.03). Adjusted for weight or fat-free mass and fat mass, energy requirements still differed by feeding group but not by age or sex. Temperament and motor development did not affect TEE. CONCLUSION: The TEE and energy-deposition data of these healthy, thriving children provide strong evidence that current recommendations for energy intake in the first 2 y of life should be revised.

Anthropometry↗
PubMed1994

Appetite and the regulation of body composition.

Stability of body composition requires that energy intake equals energy expenditure when integrated over prolonged periods. As recent human studies have failed to demonstrate active changes in energy expenditure with changes in body composition, it is likely that energy intake is continually adjusted to preserve a constant total adipose tissue mass. If adipose tissue mass is regulated directly, then there must be some input reflecting this quantity to the central nervous system for the purpose of making corrective changes in appetite when total body fat content fluctuates. The nature of this input has been examined in a variety of animal experiments involving induced weight change, lipectomy, plasma transfer from obese or satiated animals to hungry animals, and parabiosis between obese and lean animals. The bulk of evidence suggests that the plasma level of one or more currently unidentified stable circulating molecules increases in proportion to total body fat content and augments the effect of meal-related satiety signals in the central nervous system. The implications of this adipose tissue-related satiety factor for the pathogenesis of obesity, and the possible nature of the factor are discussed.

Animals↗
PubMed2000

The sensitivity and specificity of the Goldberg cut-off for EI:BMR for identifying diet reports of poor validity.

OBJECTIVE: To explore the specificity and sensitivity of the Goldberg cut-off for EI:BMR for identifying diet reports of poor validity as compared with the direct comparison of energy intake with energy expenditure measured by doubly-labelled water. DESIGN: Twenty-two studies with measurements of total energy expenditure by doubly-labelled water (EE), basal metabolic rate (BMR) and energy intake (EI) provided the database (n=429). The ratio EI:EE provided the baseline definition of under- (UR), acceptable- (AR) and over-reporters (OR), respectively EI:EE <0.76, 0.76-1.24 and >1.24. Four strategies for identifying under- and over-reporters using the Goldberg cut-off were explored. Sensitivity of the cut-off was calculated as the proportion of UR correctly identified and specificity as the proportion of non-UR correctly identified. RESULTS: UR, AR and OR (by EI:EE) were 34, 62 and 4% respectively of all subjects. When a single Goldberg cut-off for the physical activity level (PAL) of 1.55 was used, for men and women respectively the sensitivity was 0.50 and 0.52 and the specificity 1. 00 and 0.99. Using a cut-off for higher PAL traded specificity for sensitivity. Using the cut-off for a PAL of 1.95, sensitivity was 0. 76 and 0.85 and the specificity 0.87 and 0.78 for men and women respectively. Using cut-offs for mean age-sex specific PAL did not improve sensitivity. When subjects were assigned to low, medium and high activity levels and cut-offs for three different PALs used, sensitivity improved to 0.74 and 0.67 without loss of specificity (0. 97 and 0.98), for men and women respectively. If activity levels for men were applied to the womens' data, sensitivity improved to 0.72. CONCLUSION: To identify diet reports of poor validity using the Goldberg cut-off for EI:BMR, information is needed on each subject's activity level.

Adolescent↗
PubMed1982

Effects of excessive caloric intake and caloric restriction on body weight and energy expenditure at rest and light exercise.

The individual response to overeating and semistarvation on energy expenditure at rest and light work before and after a test meal was investigated. This response was related to the change in dry body weight, measured as weight (W) minus total body water (TBW). Experiments were performed on 9 non-obese subjects: (a) with a normal habitual energy intake; (b) (overfeeding) with an extra energy intake of 12 MJ per day for two weeks; and (c) (semistarving) with an energy intake of only 2.1 MJ per day for 2 weeks. Measurements of VO2, VCO2, W and TBW were obtained at the end of each of the three periods. It was found that the perturbation in energy intake from normal to 20-25 MJ per day increased the energy expenditure. The magnitude of this increase was highly individual and inversely related to the change in dry body weight. Energy expenditure, measured under the four standardized conditions, after 2 weeks of starvation was lower than that obtained after the preceding overeating period. This decrease was also roughly inversely related to the change in dry body weight. The results support the idea that part of the regulation of body energy content takes place by way of a change in the efficiency of energy utilization and that the response to a perturbed energy intake varies considerably between subjects.

Adult↗
PubMed1996

Evaluation of the dietary history method used in the SENECA Study.

OBJECTIVE: To evaluate a modified dietary history method applied in elderly people aged 74-79 y. SETTING AND SUBJECTS: A total of 387 men and 420 women from seven European towns participated in an observational study. METHODS: Dietary intake was assessed with a modified dietary history and compared with a 3-day estimated record. RESULTS: The modified dietary history provided consistently higher intakes of energy and nutrients than the estimated record. For most nutrients this difference was between 10% and 20%. However, in some towns differences were much larger. Physical activity ratios calculated as reported energy intake divided by estimated basal metabolic rate (BMR) showed values less than 1.5 for data obtained with the estimated record, suggesting underreporting of energy intake. Pearson correlation coefficients between estimates by the two methods of the intakes of energy and almost all nutrients ranged from approximately 0.5 to 0.75. CONCLUSION: The size of the overall correlation coefficients indicates an acceptable agreement between the dietary history and record method in ranking individuals according to their intakes. For two towns, this agreement was not that good. Results indicate that the estimated record might not be a good reference method for older adults.

Aged↗
PubMed1995

[Empty body weight gain, protein, faty and energy deposit and utilization of metabolizable energy for energy deposit in black and white bulls. 2. Relationship between energy deposit and intake of metabolizable energy].

From 6 series of individual feeding experiments with different energy supply (EL = 1.1-1.8; EL = 538 kJ/kg LW0.75) the relationship between the intake of metabolizable energy and live weight gain empty body weight gain and energy deposition was quantified according to a nonlinear regression analysis: intake ME (MJ/d) = A.eB.ED. EBWG. LWG with A = a.LW0.75 B = b.ec.LW The estimated parameters are: [chart: see text] The regression analysis made it possible to estimate the maintenance requirement. Dependent on the variable the following values for maintenance requirements were found: [chart: see text] For the partial utilization of metabolizable energy for energy deposition an average value of 0.37 was obtained by suggestion of a maintenance requirement of 538 kJ/kg LW0.75 and a nonlinear generalization of intake of metabolizable energy. The utilization of metabolizable energy was positively influenced by the metabolizability of the energy of the diet and it was negatively influenced by the energy level. The linear quantification of the relation between the intake of metabolizable energy and the energy deposition led to an utilization of 0.40.

Animals↗
PubMed1997

Energy balance: role of genetics and activity.

It is clear from the above discussion that much remains to be learned about both energy intake and energy expenditure. Obesity in humans is not likely to be caused by one gene, as it is in certain rodent models of obesity, such as the ob/ob mouse and the fa/fa rat. It is likely to be a polygenic condition in which numerous genes interact with each other and the environment to express the obesity phenotype. It is likely that genes that affect energy intake as well as genes that affect energy expenditure are involved. The role of leptin as a putative factor in signaling the extent of the fat mass to the central nervous system and controlling both food intake and energy expenditure is unclear at this time. The genetics of energy expenditure are also not clear at this time. There are mechanisms of nutrient partitioning, such as respiratory quotient and lipoprotein lipase activity, that are being discovered to be important. In addition, insulin sensitivity is likely to play a role in the etiology of obesity. Much more investigation will be required before we have a clearer picture of how the above-named factors interact, what the genetic contribution to this is, and how important each factor is to the overall phenotypic expression of obesity.

Animals↗
PubMed1976

Nutrition of elderly men living alone. I. Intakes of energy and nutrients.

The energy and nutrient intakes of 35 men aged 70 years and over, living alone at home, were studied using five-day records. The mean energy intake was 1628 (SD 545) kcal/day. Protein and calcium intakes were generally adequate, but 60% of the old men took less than 10mg of iron per day. Intakes of vitamin C were below 15 mg/day in 34%. Thirty-four percent took less than 0.8 mg thiamine daily. Intakes of vitamin A and riboflavin were, in general, adequate, except for a small number of subjects. It is concluded that about one-third of the men had deficient vitamin C or thiamine intakes.

Aged↗