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At least 145 records · Page 8Linked to original sources
PubMed1982

Choristoma: esophageal stenosis, due to tracheobronchial remnants.

This communication describes one case treated by the authors and reviews 27 reports of congenital esophageal stenosis caused by tracheobronchial remnants. Histologic examination of the case showed respiratory mucous glands with respiratory epithelium and hyaline cartilage. The anomalies presumably result from failure of normal embryonic separation of the respiratory tract from the primitive esophagus. Primary resection is recommended as definitive treatment. Dilatation of the stenosis affords only temporary relief.

Bronchi↗
PubMed1986

Congenital esophageal stenosis. Transendoscopic web incision.

We treated a case of congenital membranous stenosis of the esophagus successfully by transendoscopic incision. We discuss various types of true congenital esophageal stenosis and their treatment, to distinguish these rare anomalies from the more frequently encountered acquired variety.

Endoscopy↗
PubMed2003

Clinical characteristics and management of congenital esophageal stenosis: a report on 14 cases.

PURPOSE: The aim of this study was to review 14 patients with congenital esophageal stenosis (CES) from diagnostic and therapeutic points of view. METHODS: From 1976 to 1999, 14 patients with CES were treated at the authors' hospital. In each diagnostic examination, an esophagogram, 24-hour pH monitoring, a manometric study, and an endoscopy were performed. The medical, radiologic, and operative reports of these patients were reviewed. RESULTS: Esophagogram results showed an abrupt stenosis in 5 patients and a tapered stenosis in 9. After 24-hour pH monitoring and an endoscopy, we were able to rule out the possibility of gastroesophageal reflux with esophagitis in most of the patients. Of the 11 patients who initially underwent bougienage or balloon dilatation, 8 of them had recurrent stenosis. Eleven patients required surgical treatment, including the 8 who had recurrent stenosis after undergoing dilatation. Nine of the patients underwent a resection of the stenotic segment and esophago-esophagostomy. An antireflux procedure was performed on 2 patients. Another patient had an esophageal rupture, which required a jejunal interposition. A histopathologic study showed that tracheobronchial remnants were present in 6 patients, and fibromuscular thickening was found in 4 patients. CONCLUSIONS: The efficacy of dilatation seems to be limited and may even result in severe complications such as an esophageal rupture. In this study, most patients ultimately required surgical treatment. Surgical repair for CES is the authors' preferred treatment, although initial dilatation may be effective for some patients.

Abnormalities, Multiple↗
PubMed1999

[Endoesophageal prosthesis as palliative treatment for malignant esophageal stenosis: cooperative study of the Northwest of the Buenos Aires Province].

PURPOSE: To show the experience in rigid prosthesis used as a palliative treatment for malignant++ esophageal stenosis. MATERIAL AND METHOD: From November 1995 to January 1997, 14 patients between 51 and 83 years old, were referred for rigid prosthesis in a prospective way (71.5 = 2.3); 11 of them were men and 3 women. The criteria for including them was: 11 (eleven) patients suffering from esophagus cancer stage IV; 2 (two) patients suffering from stomach cancer with esophageal invasion stage IV and 1 (one) patient with mediastinum metastasis of breast cancer. Wilson-Cook rigid prosthesis was used for all these cases. The insertion technique was "the dilator method". RESULTS: Successful method: 14 (100%). Mortality: Not recorded. Disorders: In 6 (six) patients (42.8%); 3 (three) (21.4%) at an early stage: migrations and 3 (21.4%) at a later stage: 2 (two) tamponade because of food and 1 (one) magration. Time in hospital or clinic: 24 hours (100%). Improvement on Dysphagia: 14 (100%). Following: 13 (92.8%). Survivance: 18-266 days (92 = 83.6). CONCLUSIONS: 1. High percentage of success in prosthesis setting. 2. No mortality. 3. Low percentage of complications and if so, of minor importance. 4. Minimal time in hospital or clinic. 5. Quick and effective dysphagia improvement. 6. Appropriate follow up. 7. Survival similar to other palliative methods, less morbimortality and cost and better life conditions.

Aged↗
PubMed1997

[Esophageal stenosis in epidermolysis bullosa hereditaria].

Benign esophageal strictures in the upper esophagus may be caused by systemic diseases. A rare reason for esophageal strictures are the mechanobullous disorders marked by blister formation following relatively minor trauma, e.g. epidermolysis bullosa. In this report we present a 63-year-old patient who had suffered from epidermolysis bullosa simplex since birth. The epicutaneous symptoms were no longer present but an esophageal stricture had developed. The stricture was treated by X-ray-controlled balloon dilation twice, with a functionally good result. We suggest managing a recurrence recidiv of stricture with balloon dilation to minimize trauma and prevent further lesions.

Catheterization↗
PubMed1991

[Esophageal stenosis and bullous epidermolysis. Endoscopic treatment].

Esophageal lesions are often present in congenital epidermolysis bullosa. Bullae formation, ulceration and oedema ultimately will lead to stricture formation. Surgery is useful only for slowly evolutive forms. To prevent the risk of developing complete occlusion to the human of the esophagus, dilatations are performed under endoscopic control, that allows through nutrition without risk of local complication.

Dilatation↗
PubMed1995

[Self-expanding wire-netting Elastalloy endoprosthesis in malignant esophageal stenosis].

12 patients with malignant inoperable esophageal obstruction (carcinoma of the esophagus n = 7, carcinoma of the esophagogastric junction n = 3, mediastinal lymph node metastasis n = 2) and high-grade dysphagia were treated with self-expanding metal stents (Ultraflex, Microvasive) made of a nickel titanium alloy (Elastalloy). Other forms of palliation had failed in 9/12 patients. The degree of palliation was expressed as a dysphagia score (0-4) before and after stent insertion. The stents were inserted under endoscopic and fluoroscopic control. They were placed successfully and without complications in all patients. A good functional result was achieved in 11 patients (91.7%). Thus, the dysphagia score decreased significantly from 3.2 +/- 0.4 before to 0.9 +/- 1.0 immediately after stent insertion (p < 0.001). The remarkable relief of dysphagia was sustained during a mean follow up of 101 days (10-278) with a dysphagia score of 1.1 +/- 1.0 at the end of the study (p < 0.001 compared to the score before the procedure). In one patient with mediastinal lymph node metastasis the stent expanded insufficiently. 7 days after insertion it was removed endoscopically and replaced successfully by another stent with a stronger expansive force (Instent). 3 patients experienced recurrent dysphagia (food impaction n = 1 tumor ingrowth through the meshes of the stent n = 2). They were successfully treated by an endoscopical intervention (endoscopical dilatation n = 1, laser therapy n = 1, insertion of a Wallstent n = 1). At the end of the study, 6 patients were alive, 6 patients were dead with a mean survival of 56 days (10-117).

Adult↗
PubMed1977

[Special aspects of colo esophagoplasty in post-caustic esophageal stenosis for corrosive stricture of the esophagus (author's transl)].

The author reports 12 cases which illustrate unusual aspects of colo esophagoplasty in post-caustic esophageal stenosis. 8 cases respresented post-caustic pharyngolaryngeal stenosis in which he recommended anastomosis of the colon with the oropharynx according to a personal technical variant. In 1 case associated with amputation of the epiglottis, he carried out a double pharyngo-colic Y-shaped anastomosis. He classified the pharyngotomy types in relation to the 3 main nerves, the lingual, hypoglossal and superior laryngeal nerves. In 3 cases the author carried out successfully retrosternal transposition of a colonic segement 6 months, 1 year and 4 years after pre-thoracic colo esophagoplasty. He reports 1 case of intrathoracic strangling of the colonic tube which occurred 1 year after retrosternal colo esophagoplasty, and wich was cured by intra-vascular vertical clectomy. In all cases the good results were maintained for a long period.

Burns, Chemical↗
PubMed1993

H-type tracheoesophageal fistula and congenital esophageal stenosis.

An 18-month-old boy was seen in the pediatric pulmonary clinic with a history of wheezing, stridor and intolerance to solid foods. Barium esophagram revealed distal esophageal stenosis and subsequently an H-type TEF at surgery. Following the surgery to repair both lesions the child continues to do well.

Esophageal Stenosis↗
PubMed1984

[Presternal ileum and right colon for benign esophageal stenosis. Very long-term checking].

Long-term follow up results are analyzed of 10 patients treated by pre-sternal right ileocolic grafts for an esophageal stenosis of a benign nature. Operation in 7 cases had been performed 28 (1 case), 26 (2 cases), 19 (1 case), 16 (1 case), 11 (1 case) and 6 (1 case) years previously. Functional results were generally very satisfactory. Two cologastric anastomotic ulcers were heated by coloduodenal and colojejunal reimplantation respectively. One patient operated upon for stenosis due to chemicals developed malpighian cancer of the cervical esophago-ileal anastomosis 12 years later, but secondary malnutrition did not occur following a gastric bypass operation. Although overall functional results could be considered as very satisfactory, there is a risk of anastomotic cologastric ulcers developing (2 cases), but the risk can be eliminated by routine implantation of colon into duodenum or jejunum. The gastric bypass achieved failed to produce any clinical manifestations in patients operated upon, some of whom have been followed up for nearly 30 years.

Adolescent↗
PubMed1987

[Treatment of benign esophageal stenosis by prolonged dilatation: re-evaluation after 15 years].

The purpose of this paper is to re-appraise the results of a method for slow continuous dilatation of benign esophageal stenosis comparing previous experience of a decade with that accumulated during the following five-year period. A total of 78 patients were dilated, the average age was 58.1 years, and the M/F dilated, the average age was 58.1 years, and the M/F ratio 3.3/1. Reflux esophagitis was the most frequent etiology when comparing both groups (77.5% and 79.4% respectively). Esophageal hiatus hernia was the most common associated pathology in both groups (83.3% and 87.7% respectively). Gastric analysis showed average values within the limits of normal, 40% of de cases showed a hypersecretion pattern. The mean lower esophageal sphincter pressure was low, and a group of patients showed aperistalsis in more than 50% of the swallows, fact that was thought to be due to esophageal inability to clear the refluxed material. Another important fact was an increase in the frequency of Barrett's epithelium (6.5% to 9.3%). Undoubtedly the most important features were the increase in the number of good results of dilatation (87.7% to 92.3%), the decrease of morbidity (6.1% to 0.7%) and mortality (2% to 0.2%). The follow-up increased from X 27.3 to X 33.8 months.(ABSTRACT TRUNCATED AT 250 WORDS)

Adolescent↗
PubMed1985

[A case of acute promyelocytic leukemia complicated with severe esophageal stenosis caused by aclacinomycin A].

Aclacinomycin A (ACM) is different from other anthracycline antibiotics in its antitumor activity and clinical effect. We report a case of acute promyelocytic leukemia complicated with severe esophageal ulcer by BH-AC . AMP therapy. The patient was 24 years old. In 1983, he achieved complete remission with BH-AC . DMP therapy but we confirmed relapse in April, 1984. ACM caused nausea, vomiting, diarrhea, alimentary tract bleeding and sore throat, together with a complication of esophageal stenosis. In about half of 10 cases receiving BH-AC . AMP therapy in our hospital, we noticed severe bleeding in the urinary treat, genital organs and alimentary tract. Care should therefore be taken with regard to mucosal injury when ACM therapy is used.

Aclarubicin↗