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At least 145 records · Page 8Linked to original sources
PubMed1990

Comparison of six different regimens for the control of atrophic rhinitis in swine.

Effectiveness of six different programs of atrophic rhinitis (AR) control was evaluated under field conditions. Efficacy of treatments was recognized on the basis of--comparative evaluation of the average daily gain (ADG), morphometric examination of turbinate bones and computer conchal morphometry (TPR). Usefulness of the evaluated regimes differed significantly. TPR appeared to be the most objective method for AR evaluation. Correlation between severity of turbinate atrophy and ADG was only partial.

Animals↗
PubMed1985

[Production of a specific gamma globulin against infectious atrophic rhinitis in swine].

A specific gamma globulin against infectious atrophic rhinitis in swine was produced following a pattern that was worked out for the hyperimmunization of serum-producing swine. Used were strains of Bordetella bronchiseptica and Mycoplasma hyorhinis isolated by the authors. The hyperimmune serum obtained was processed after Cohn's method which was improved by Ponamarova and Nechaeva. The preparation obtained was innocuous and immunogenic for both test animals and pigs. It rendered protection when applied at the rate of 0.4 cm3 per kg body mass. At 1 cm3, i/m, it was found to act therapeutically.

Animals↗
PubMed1978

Silastic implant in atrophic rhinitis - a review of 10 cases.

Ten patients with atrophic rhinitis were treated surgically with silastic implants. The details and principles of the surgical technique are emphasized. These patients have been observed for up to nine years. They all improved, with 70 per cent of them showing a "marked improvement" and the remaining 30 per cent showing a satisfactory one.

Adult↗
PubMed1983

Virulence of Pasteurella multocida in atrophic rhinitis of gnotobiotic pigs infected with Bordetella bronchiseptica.

Bordetella bronchiseptica and Pasteurella multocida have been impLicated in the aetiology of atrophic rhinitis of pigs but the precise cause and pathogenesis of field outbreaks have still to be clarified. The virulence of 11 strains of P multocida was investigated by intraperitoneal injection of culture filtrates in BALB/C mice, or by infection of gnotobiotic piglets given B bronchiseptica five days previously. Three of four type D strains of P multocida were lethal for mice and caused severe turbinate lesions and shortening of the snout with B bronchiseptica in gnotobiotic pigs; large numbers of P multocida and B bronchiseptica persisted for 64 days in the nasal cavity of these pigs. The fourth strain caused moderately severe turbinate lesions in gnotobiotic pigs infected with B bronchiseptica; small numbers of P multocida were found in these pigs and the lesions were attributed mainly to B bronchiseptica. Filtrates from seven strains of P multocida (four type A and three type D) were not lethal for mice and these strains with B bronchiseptica caused moderately severe turbinate lesions in gnotobiotic pigs; five of them colonised the nasal cavity reasonably well for 35 days but the lesions were attributed mainly to B bronchiseptica. The turbinate bones had regenerated by 64 days in pigs given type A strains of P multocida whereas the lesions persisted in pigs given type D strains. Antibodies to P multocida were detected in sera from infected gnotobiotic pigs by acid agglutination but not by indirect haemagglutination tests; neutralising activity to the mouse lethal toxin was detected in serum from one of five piglets at 64 days. The lethal toxin was inactivated at 56 degrees C for 30 minutes, by incubation with protease K for two hours and by 0.2 per cent formalin for 18 hours at 37 degrees C but not by trypsin; it was precipitated by 30 to 40 per cent saturation with ammonium sulphate and remained in the supernatant after centrifugation at 150,000 g. It was concluded that infection with virulent, type D strains of P multocida and B bronchiseptica could explain severe outbreaks of atrophic rhinitis; large numbers of both organisms persisted in the nasal cavity of gnotobiotic pigs with severe lesions; and that a soluble, heat-labile toxin may be an important virulence determinant in the type D strains of P multocida that cause severe atrophic rhinitis.

Animals↗
PubMed1998

Closure of the nasal vestibule in atrophic rhinitis--a new non-surgical technique.

A new technique is described for closing the nasal vestibule in cases of secondarily-acquired atrophic rhinitis. This involves occlusion of the nasal vestibule with an obturator made from dimethylpolysiloxane. Being a non-invasive method it is specifically indicated in the management of cases of secondarily-acquired atrophic rhinitis where any surgical treatment is contra-indicated. We describe its use in a case each of unilateral and bilateral secondary atrophic rhinitis.

Adult↗
PubMed1990

Detection and distribution of toxigenic Pasteurella multocida in pig herds with different degrees of atrophic rhinitis.

Four herds of pigs were selected which had different degrees of clinical atrophic rhinitis and used different specific counter-measures. In two of them, the clinical signs occurred spasmodically and were slight. The sows, suckling pigs and growing pigs in all the herds were sampled for toxigenic Pasteurella multocida. In one of the slightly affected herds (herd D), the weaners were moved to a second farm for finishing. No toxigenic P multocida were found at the breeding farm, but 50 per cent of the large growing pigs were positive. It seemed that the organism had entered only at the finishing farm and that the mild clinical signs were due to the infection starting in older pigs than usual. In the second mildly affected herd, 47 per cent of the sows and 42 per cent of the growers were infected. Three toxigenic isolates from this herd produced as severe turbinate damage experimentally in specific-pathogen-free pigs as a stock pathogenic strain. Except in herd D, toxigenic P multocida were found in all the age groups of pigs sampled. However, the pattern of distribution of the organism within the herds was not obviously correlated with the severity of the disease. In a fifth herd there were obvious cases of clinical atrophic rhinitis, with marked turbinate atrophy, from which toxi-genic P multocida were recovered in abundance. Subsequently, the clinical disease disappeared and, despite extensive and repeated sampling, the organism was not found again.

Animals↗