PubMed Health⌕ Search

SEARCH · PubMed Health

Results for “Auditory Threshold”

Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.

Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.

At least 1,531 records · Page 85Linked to original sources
PubMed1988

[The auditory evoked potentials of the brain stem in the guinea pig].

The examination of the standard waves' amplitude and latency of the brain stem auditory evoked response (BAEP) was performed in 20 guinea pigs (males and females, weighing 250 to 300 g). According with the relative loudness of stimuli (90, 70, 50, 30, 10 dB SPL), the latency of BAEP waves was larger (t1 = 0.2 msec), but the conductance time between P1 to P5 was constant (3.1 to 3.6 msec). The highest wave of BAEP was P2 with an amplitude: 90 dB SPL, U = 6.5 +/- 1.2 microV; 70 dB SPL, U = 4.3 +/- 1.0 microV; 50 dB SPL, U = 3.5 +/- 0.6 microV; 30 dB SPL, U = 2.0 +/- 0.4 microV.

Acoustic Stimulation↗
PubMed1986

[Neuro-otology].

Explore the source record for details and available documents.

Audiometry↗
PubMed1985

Auditory brainstem responses to clicks in neonates.

Normative data are required when interpreting brainstem electric response audiometry results. An abnormal auditory brainstem response (ABR) cannot be identified without knowledge of the normal ABR and the variability associated with technical and physiological factors. Normative data obtained from babies in a neonatal intensive care unit in response to click stimulation are presented in this paper.

Acoustic Stimulation↗
PubMed1985

The physiology of the normal and pathological cochlear neurones--some recent advances.

This is the first of two auditory science tutorials designed for otolaryngologists, audiologists, and others dealing with deafness and its consequences. This tutorial is intended as an update on recent advances in the understanding of cochlear function both in the normal person and in patients with hearing loss of cochlear origin. It draws largely on studies involving electrophysiological recordings from cochlear nerve fibres in "animal models" of cochlear hearing loss. These data allow considerable insight into the underlying causes, for example, loudness recruitment, tinnitus, and poor speech intelligibility. Areas of scientific ignorance are also mentioned. This is not an exhaustive review of the literature but rather a general overview. The style of the tutorial is not starchily scientific, contains minimum jargon, and is intended to make easy reading.

Animals↗
PubMed1985

Effect on brainstem auditory evoked responses of posterior semicircular canal occlusion in guinea pigs.

The site of origin of benign paroxysmal positional vertigo is the posterior semicircular canal and when persistent, the condition is known as cupulolithiasis. Singular neurectomy is the current procedure of choice for the treatment of incapacitating cupulolithiasis. The neurectomy is difficult to learn and carries a significant risk of hearing loss. An alternative procedure, whereby the posterior canal is occluded, is discussed. This was performed on 11 guinea pigs and the effect on hearing was measured using brainstem evoked audiometry. Eight animals showed no hearing loss while the other three had a mild to moderate loss of hearing due to intra-operative complications. The effect of canal plugging on hearing compares favorably with the hearing results obtained with singular neurectomy. Posterior semicircular canal occlusion is suggested as a safer and technically easier treatment for incapacitating cupulolithiasis.

Animals↗
PubMed1984

[Cervically-induced symptoms of the Klippel-Feil syndrome].

Klippel-Feil syndrome is a congenital deformity disease caused by disturbances of the growing together of the mesenchymal preliminary vertebrae which are mostly located in the regions of the cervical spine and shoulder. Short neck and wryneck (torticollis), as well as limited motion of the neck and reduced mobility of the cervical spine, are the most striking findings on clinical examination. Vertigo, disturbed sound conduction and perception, as well as combined loss of hearing, tinnitus and paralysis of the facial nerve can occur although otoscopic findings may be normal. Differential diagnosis is effected by means of the typical x-ray film of the cervical spine and the vertebrobasilar transition. Functional anomalies of the vertebral arteries are determined via x-ray angiography. Neuro-otological syndromes can be explained by congenital deformities of the middle ear, cochlea, peripheral vestibular organs and internal auditory canal; mainly, however, by a "vascular" cervical syndrome, which, in this particular case, explains the cochlea symptoms. Vertigo and dizziness can also be caused by a pathological irritation of cervical proprioceptive sensory organs through deformities of the skeleton.

Audiometry, Pure-Tone↗
PubMed1994

Asymmetric frequency-following responses.

This study offers evidence of asymmetry in the frequency-following response (FFR), a brainstem evoked potential that mimics stimulus frequency. The FFRs to stimulation of one ear compared to the other are shown to have latency and amplitude dissimilarities, as well as considerable variations in waveform characteristics. The findings were obtained from eight subjects. Tone bursts at 500 Hz were delivered to the right ear, left ear, and binaurally at three sound levels. With few exceptions, subjects had asymmetric FFRs at every sensation level (SL); at some, the sound to the left ear evoked a larger response than the same sound to the right; at other levels, the right ear FFR was larger than the left. The results provide evidence that cortical processing asymmetries must, to some extent, be reflective of asymmetric mechanisms within the lower brain stem.

Acoustic Stimulation↗
PubMed1993

[Diagnosis of hearing disorders in premature infants].

In cases of severe hearing loss in babies an early rehabilitation should prevent a delay in development. More than a half of all hearing disorders in children are acquired; to a large extend these hearing disorders are caused by premature birth and can be detected by suitable screening. Two methods are used for testing: (1) reflex audiometry with a standardized stimulus generator; and (2) bedside recording of brainstem potentials. Having tested the method of bedside recording of brainstem potentials a group of 60 premature infants of varied body weights and no additional diseases was examined as "normal" control group. Only premature birth with all signs of maturation adequate for gestational age is not a greater risk for hearing disorders. In premature infants with birth weights less than 1500 g, having marked hyperbilirubinemia and extensive intracranial bleeding always increased wave V latencies has been observed. In about 10% of these children severe hearing disorders were diagnosed and after leaving the premature department were sent to a paedaudiological dispensaire.

Audiometry, Evoked Response↗
PubMed1993

Behavioral, electrophysiologic, and otoacoustic measures from a child with auditory processing dysfunction: case report.

This case was selected to highlight the importance of the test battery approach in the assessment of a child with auditory processing deficits. The utility of behavioral, electrophysiologic, acoustic immittance, and evoked otoacoustic emission procedures, as well as the problems associated with interpreting these multiple measures with differing results, is discussed. This case was confounded by the possibility that both peripheral and central auditory problems existed. The outcome stresses the importance of examining the results of multiple auditory measures in the determination of the site of lesion and habilitation strategies.

Acoustic Impedance Tests↗
PubMed1993

Auditory middle-latency response: intramodal and intermodal interactions.

Intramodal and intermodal interactions have been studied in generators of the auditory middle-latency response (MLR). The MLRs were recorded to the wide-band clicks. Similar clicks or electric shocks to the median nerve served as conditioning stimuli. A procedure was employed permitting to avoid distortions of test MLRs due to superimposition upon potentials to the conditioning stimuli. A rapid recovery of responsiveness in MLR generators was revealed in intramodal experiments. Poorly discernible test MLRs were identified at conditioning interval of 10 ms, while already at interval of 50 ms the MLR restoration was completed. No effects of shocks upon MLRs were evident in intermodal experiments. The starting knee was only altered in most test MLRs, outlining a narrow strip of intermodal interaction. The sources of the MLR are discussed and a subcortical rather than cortical, polylevel rather than monolevel, monomodal rather than polymodal nature of the MLR is proposed.

Acoustic Stimulation↗
PubMed1993

Late subjective audiometric verifications of the early auditory brainstem responses results in young hard-of-hearing children before their speech development.

The authors described the late subjective audiometric verifications of the auditory brainstem responses (ABR) results in a group of hard-of-hearing children treated in Phoniatric Department of Children Hospital Dziekanów Leśny. The audiometric examinations took place after some time of the speech and hearing rehabilitation. The rehabilitation effects make the audiometric examinations feasible. The correlation between these two type of examinations was good.

Acoustic Stimulation↗