Respiratory acidosis in chronic pulmonary heart disease and the red cell pattern.
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Echocardiography and pulmonary scintigraphy demonstrated post-embolic chronic cor pulmonale in a six-year-old woman presenting with dyspnoea. The patient's symptoms deteriorated during subsequent thromboembolic episodes, despite treatment consisting of oral anticoagulants and diuretics. After discussing the pathophysiology and diagnostic methods, the authors emphasize the limits of medical treatment of post-embolic chronic cor pulmonale and the value of two surgical treatments: thromboendarterectomy and lung transplantation. They also recall the precise selection criteria for lung transplantation.
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With the exception of the decompensated state there is no special therapy in the pulmonary heart disease. Therapy is mainly directed to the original lung disease. Regarding the frequency of the ventilative-hypoxic type of the cor pulmonale chronicum the main thing of this lecture is the improvement of oxygen supply and thereby the lowering of pulmonary artery pressure pathologically raised by drug-induced bronchial dilatation.
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