[Coronary spasm: myth or reality?].
Explore the source record for details and available documents.
SEARCH · PubMed Health
Explore indexed PubMed citations for clinical trials, systematic reviews and public health research. Read source abstracts and follow each citation to its original PubMed record.
Quote a phrase for an exact phrase match. Source license links do not imply unrestricted reuse.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
A 56-year-old man had coronary artery spasm. The initial manifestation of the disease was syncope that occurred at night. Coronary artery spasm was documented by Holter recording and cardiac catheterization. The patient was effectively treated with nitrates and calcium-blocking agents.
Ergometrine test was undertaken on the 1--3d day after hospitalization in 49 patients with unstable angina pectoris. Pain or ECG changes were recorded in 90% of patients. Reactions with the rise of the ST segment and changes of the T wave on ECG were interpreted as "spastic" and were seen in 43% of cases. Myocardial infarction supervened during hospitalization in 19% of patients with "spastic reactions", and in 14.3% of other patients. Selective coronarography was performed in 33 patients. During coronarography the local spasm of one coronary artery supervening always at the site of organic stenosis was seen in 10 patients (30%). Spastic reactions in angina pectoris can be elicited relatively frequently, but nonspecific reactions are possible, with low tolerance of haemodynamic shifts caused by ergometrine. In the initial angina pectoris, immediately preceding myocardial infarction ergometrine may cause no reaction. An increased tendency of atherosclerotic coronaries to spasm plays a definite role in the development of unstable angina pectoris, in a part of patients only (or only in a certain phase of its course). Such reactions are not associated with a considerably increased danger of development of myocardial infarction.
A total of 128 patients with unstable angina pectoris (UAP) have been examined. Personal data of clinical examinations and the literature data enabled the authors to elaborate diagnostic criteria of UAP. Classification of UAP with consideration of its history, the clinical course of the disease and changes in the coronary arteries found with coronarography is offered. Almost half of patients with UAP begin to feel it while in perfect health. In 1/3 of cases only therapeutic treatment made the symptoms regress. It is established that UAP occurs mainly due to the changes in the system of left coronary artery. Very often stenosis of the main branch of this artery is observed.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.
Explore the source record for details and available documents.