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At least 19 recordsLinked to original sources

Visual allesthesia.

A patient with a right parietooccipital arteriovenous malformation experienced paroxysmal illusory left homonymous transpositions of objects viewed in the right homonymous field. The illusory images were palinoptic, persisting for up to 15 minutes after the real objects were no longer in view. She had left homonymous hemiachromatopsia and a right parietooccipital epileptogenic focus. Episodes of visual allesthesia were accompanied by other manifestations of seizures. Administration of anticonvulsant medications resulted in cessation of electroencephalographic epileptogenic activity and disappearance of all clinical manifestations of seizures, including visual allesthesias. Cerebral irritation and seizures, a defective but not blind half-field of vision, and experience of palinoptic images are essential components of some cases of visual allesthesia. The phenomenon may result from interhemispheric transfer of a visual percept from a normal to an irritated parietooccipital lobe, which then retains it as a palinoptic image.

Adult↗

Allochiria vs allesthesia. Is there a misperception?

Allochiria is the mislocation of sensory stimuli to the corresponding opposite half of the body or space. Obersteiner (1882) introduced the term allochiria (Greek allos = other + chiria = hand), and more than 20 authors employed it in this context over the next 25 years. Stewart (1894) described a related phenomenon in which stimuli are displaced to a different point on the same extremity. He noted that the displacements were different than allochiria and coined the term allachaesthesia (ie, allesthesia) (Greek allaché = elsewhere + aisthésis = perception). Despite this historical background, Jones (1907) redefined both terms in an attempt to increase diagnostic specificity and attributed allochiria to hysteria. Jones' reinterpretation does not appear to be justified historically, etymologically, or scientifically and has resulted in contradictory definitions of allochiria and allesthesia in present-day medical dictionaries and neurologic textbooks. We advocate a return to usage consistent with the original descriptions and word derivations.

Europe↗

Effects of a low dose of naltrexone on glucose-induced allesthesia and hunger in humans.

In order to study the effects of a low dose of the opioid antagonist naltrexone on ingestive behavior for sucrose in humans, preference for sucrose solutions and feelings of hunger were scored on visual analogical scale by 14 healthy subjects with or without naltrexone. Effects of intragastric glucose load or water, and naltrexone (25 mg) or placebo were tested. At this low dose, naltrexone alone had a slight effect on allesthesia, and it produced a strong potentiation of glucose-induced allesthesia.

Adult↗

Negative allesthesia and decreased endogenous opiate system activity in anorexia nervosa.

The combined effects of an intragastric load of glucose compared to water and of naltrexone compared to placebo were tested on preference for sucrose in six anorectic patients. While in normal subjects, glucose-induced negative allesthesia is known to disappear upon loss of weight, it persisted in anorexia nervosa (AN) despite a major weight loss; furthermore, in contrast with its effects in normoponderal subjects, naltrexone at the dose of 25 mg did not decrease the preference for sucrose nor did it enhance glucose-induced allesthesia. Basal plasma beta endorphin level determined by radioimmunoassay was higher in AN than in normal subjects (75 +/- 6.1 pmoles/l vs. 13 +/- 3.8 pmoles/l) (p less than 0.001). It is suggested that a decrease in endogenous system opiate activity might be associated with food refusal and body weight loss in anorexia nervosa.

Adolescent↗

Visual allesthesia in manual pointing: some evidence for a sensorimotor cerebral organization.

The authors of this paper first summarize some of the theoretical frames put forward to account for allesthesia. Then, pointing performances in a case of visual allesthesia is reported; unexpectedly, pointing behavior for identical targets was different according to hand. The most salient feature was that a same visual target elicited relatively short RTs with adequate pointing using the left hand versus much longer reaction times with allesthesic pointing using the right hand. Discussion is focused on the presented theoretical frames. In view of the results, authors are led to hypothesize that, in sensorimotor cerebral organization, some aspects of signal processing are dependent upon some premotor aspects of response elaboration.

Attention↗

[A case of falcotentorial meningioma with visual allesthesia].

Visual allesthesia, in which visual images are transposed from one homonymous half-field of visual field to another, is a rare phenomenon. Palinopsia is the persistence or recurrence of visual images after the exciting stimulus object has been removed. Some authors have reported these phenomena, but these pathophysiology has not been understood. We report a right-handed 63-year-old woman, with a right falcotentorial meningioma. She paroxysmally experienced illusory transpositions of objects viewed in the right homonymous visual field into the left, and she recognized her face in it. The illusory images were palinoptic, persisting for up to a few minutes after the real objects were no longer in view. Administration of anticonvulsant medications resulted in the decrease of frequency of this phenomenon. Radiological and surgical findings revealed that the tumor was compressing the very localized visual cortex, especially the Brodmann's area 18 and 19. After resection of the tumor, visual allesthesia and palinopsia completely disappeared. These areas are associated with memory and recognition of visual images as visuopsychic area. This case provides some insight into the mechanisms of this phenomenon, and we consider that it could be caused by seizure activity in this lesion.

Female↗

[A case of visual allesthesia].

A case of visual allesthesia is reported. A thirty-year-old right-handed man with a right temporo-occipital arteriovenous malformation underwent the surgery for extirpation of the lesion. Postoperatively he had left homonymous hemianopsia. Approximately 6 months after the surgery, he experienced the initial spell of generalized convulsion, followed by the episodes of visual allesthesia. He experienced illusory left hemonymous transpositions of objects viewed in the right homonymous field. The transposition occurred from normal to the defective visual field. The illusory image was palinoptic, persisting for up to several minutes after the real object was no longer in view. It was equal in shape with the original, and caused monocular diplopia. MRI showed a large area of postoperative changes. The lesion was located from the posterior half of the middle and inferior gyri of the right temporal lobe to the occipital and a part of the parietal cortex. And it extended into the right fusiform and lingual gyri. The right lateral geniculate body and the posterior part of thalamus were also involved. We speculated that this phenomenon may result from irritation of the affected brain and its surrounding areas, since the episode took place following the seizure.

Adult↗

Palinopsia and visual allesthesia.

Two cases of palinopsia are reported. The first patient had palinopsia and visual allesthesia secondary to an occipital calcified cysticercus. Symptoms were more prominent during treatment for his parasitic infection. The second case was a patient with long standing vascular headaches associated for many years with episodes of visual persistence; he had a right occipital lobe infarct; he reported deformation of images, illusion of movement and macropsia in his left homonymous hemianoptic visual field. Different hypotheses for explaining palinopsia have been proposed. An analysis of our cases does not prove or disprove any of the current hypotheses. However, the current hypotheses do not necessarily contradict each other. Implications of the two cases reported are analyzed.

Adult↗

Visual allesthesia.

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Dominance, Cerebral↗

Comparisons of hemi-inattention produced by unilateral lesions of the posterior parietal cortex or medial agranular prefrontal cortex in rats: neglect, extinction, and the role of stimulus distance.

Neglect in human and non-human primates has been demonstrated following unilateral lesions of both posterior parietal and prefrontal areas. While it has now been well established that a unilateral lesion of the rodent analog of dorsolateral prefrontal cortex, medial agranular cortex (AGm), results in neglect, the effects of unilateral damage restricted to rodent posterior parietal cortex (PPC) have not been examined in detail. The current study assessed rats with unilateral lesions of PPC or AGm on their ability to orient to unilateral and bilateral stimulation. Since it has been proposed in both the primate and rodent literatures that frontal areas may be responsible for the perception of near space while parietal areas may be responsible for far space, stimuli were presented at two different distances. Lesions of PPC and AGm resulted in severe neglect relative to control operates, with both PPC and AGm operates manifesting severe hemi-inattention and allesthesia relative to control operates. After behavioral recovery from neglect there was no evidence of extinction to bilateral simultaneous stimulation. While neglect to visual stimuli predominated in unilateral PPC operates, unilateral AGm operates had severe neglect in all modalities. In addition, while both left and right PPC operates showed contralesional neglect, AGm operates demonstrated the lateralized differences in neglect reported in previous studies. All groups demonstrated an approximately equivalent level of neglect to stimuli presented at the two different distances, and thus failed to support the suggestion of a peripersonal-extrapersonal dichotomy between frontal and parietal areas in rodents.

Animals↗

The proximal tap or "central Tinel" sign in central dysesthetic syndrome after spinal cord injury.

Diffuse, chronic, and dysesthetic pain following spinal cord injury (SCI) has been described by several authors under different terms. As illustrated by the two patients described here, central dysesthetic syndrome (CDS) can be mistaken for musculoskeletal, peripheral neuropathic or visceral disease in SCI patients. In these patients, an added clue to the central neuropathic nature of their symptoms was allesthesia and allodynia to light touch or tapping over areas rostral to the level of injury; this may be called the proximal tap or "central Tinel" sign.

Humans↗