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At least 19 recordsLinked to original sources
PubMed1978

[Two surveys on farmer's lung disease and bird breeder's disease in the Pyrenees region].

Two different surveys, one strictly epidemiological in rural areas, the other retrospective and within hospitals, led to a better knowledge of the setting of the FLD and BBD in South Pyrenees region. In the country, more than 12% of patients had specific precipitins, 6% had either one or both diseases; women had anti-aviary precipitins more frequently than men. The retrospective hospital survey yielded 456 cases of patients admitted or consulting revealing numerous correlations between the different clinical, radiological and functional syndromes on the one hand and the serological positivity on the other. Serological diagnosis was most useful, as a wide variety of antigenic extracts was used (actinomycetes thermophiles, mouldy hay, bird droppings), mixed sensitizing was extremely frequent. The widely found positivity towards avian antigens seems specific to the South Pyrenees region.

Adult↗
PubMed1976

Serological investigations in 15 cases of bird fanciers disease.

Serological studies in 15 patients with bird fanciers disease are reported. Quantitative analysis of serum proteins showed elevation of IgG in almost every case. High IgE was observed in three patients with isolated asthmatic reactions. In four patients alpha1-antitrypsin was transiently diminished. Two of these had heterozygous deficiency of this protein (genotype MS and MZ). Precipitins against avian sera and avian droppings were found in all patients, but also in some exposed and nonexposed controls. Cross-reactions to avian antigens from different species were observed quite regularly in patients but not in controls. One case is presented suggesting that these cross-reactions may be of clinical importance. Precipitins against other inhaled antigens such as Aspergillus fumigatus, cereals or Micropolyspora faeni were observed significantly more frequently in patients than in controls. These precipitins showed no cross-reactions with those against avian antigens.

Adult↗
PubMed1978

Transmission of salmonid whirling disease by birds fed trout infected with Myxosoma cerebralis.

Mallard ducks, Anas platyrhynchos and a black crested night heron, Nycticorax nycticorax were fed trout infected with Myxosoma cerebralis (HOFER) in 2 separate experiments. Feces from the birds were deposited in troughs containing M. cerebralis-free mud as well as in 1 trough without mud. Spore suspensions were also added directly to mud in 1 trough and to another without mud. Susceptible rainbow trout, Salmo gairdneri, developed whirling disease in all troughs containing mud contaminated with M. cerebralis but remained free of infection when exposed to M. cerebralis in troughs without mud. This demonstrates the possibility of bird transmission of the organism causing whirling disease to previously non-contaminated waters.

Animals↗
PubMed1979

Zoonotic diseases in psittacine birds: apparent increased occurrence of chlamydiosis (psittacosis), salmonellosis, and giardiasis.

Between September 1977 and November 1978, chlamydiosis (psittacoisis) was diagnosed in 52 of 128 parrots, 5 of 12 cockatiels, 2 of 5 cockatoos, 3 of 6 macaws, 1 of 22 conures, 2 of 18 lovebirds, and 6 of 76 parakeets; 2 lories and 1 lorikeet were chlamydiosis negative. Two cases of human chlamydiosis were associated with two submissions of parrots subsequently found to have active infection. Twenty parrots (including 13 that were chlamydiosis positive), 2 cockatiels, 1 macaw, 1 lorie, and 1 parakeet yielded salmonella organisms, of which 16 were identified as Salmonella typhimurium, 8 as untypeable monophasic salmonellae of serogroup B, and 1 as S arizonae. Three S typhimurium from parrots that had been treated with chlortetracycline for chlamydiosis were resistant to tetracyclines, streptomycin, and sulfonamides; another isolate was found to be resistant to chloramphenicol only. Severe giardiasis was diagnosed in parakeets originating from six aviaries.

Animals↗
PubMedDate not supplied

Pacheco's parrot disease of psittacine birds.

Pacheco's parrot disease was identified as the cause of death of 3 psittacine birds at an aviary. Confirming a previous report, a herpesvirus was found to be the etiologic agent. The virus induced mortality in embryonated chicken eggs and budgerigars. The virus was characterized by electron microscopy, filtration experiments, and ether-sensitivity. Hepatic lesions in chick embryos and budgerigars were similar by light- and electron-microscope studies. Naked and enveloped nucleocapsids were seen in the nucleus and cytoplasm of infected hepatocytes, and naked virions were present in the sinusoids of the liver. Multinucleated cells in the liver contained virus in both intranuclear and intracytoplasmic inclusions; the latter inclusions were both membrane-bound and not. Vaccination of budgerigars with a commercial lyophilized Marek's disease vaccine did not prevent infection with the Pacheco's disease herpesvirus.

Animals↗
PubMed1975

Natural and experimental infections of Japanese tree sparrows with Japanese encephalitis virus.

20 to 37 per cent of wild Japanese tree sparrows had neutralizing antibodies to Japanese encephalitis virus (JEV). Sparrows free of antibodies were inoculated with 10,000 plaque-forming units of JEV. None of the birds became clinically ill. Virus could be detected in blood plasma during the first 3 days after inoculation but not thereafter. Neutralizing antibodies started to appear at 4 days after inoculation and the response was of variable duration, in some cases extending up to 200 days after inoculation.

Animals↗
PubMed1977

Aspergillosis in imported penguins.

An outbreak of aspergillosis in imported penguins caused by Aspergillus fumigatus is described. The birds probably acquired the infection from the country of origin or during their transportation. Predisposing circumstances are discussed.

Animals↗
PubMed1977

Experimental infection of birds with epidemic Venezuelan encephalitis virus.

Sixty-three birds representing 13 species were inoculated with a strain of epidemic Venezuelan encephalitis (VE) virus from the 1971 Texas outbreak. More than 95% of the birds became infected. Mortality which could be attributed to infection with VE virus was very low. Viremia persisted 2-6 days. Peak viremia levels ranged from 10(3.2) to 10(8.2) suckling mouse intracranial 50% lethal doses per milliliter (SMICLD50/ml). Blood virus levels were highest in juvenile Louisiana Herones, adult Robins and adult Mockingbirds and were lowest in juvenile Common Egrets. Most bird species had blood virus levels about 10(5) SMICLD50/ml (high vector infection potential) for 2-3 days. Neutralizing antibody response was more uniform and frequent in herons (95%) than in passerines (56%). The role of birds in the epidemiology of Venezuelan is discussed.

Aedes↗
PubMed1978

[Study data on ornithosis infection in the Azerbaijan SSR].

Data are presented on the study of epizootology and epidemiology of ornithosis. There were revealed natural foci of ornithosis in the Kyzyl-Agach preserve, at the Sary-Su lakes and at the Glinyany island; these foci were dihostal and polyhostal, as well as conjointed (ornithosis, arboviruses, Q-fever, Asian tick-borne typhus, leptospirosis). Anthropurgic foci were found in 12 populated localities, semi-wild dove serving as the main component. Fowl was found to be infected in 12 poultry-farms; occupational ornithosis was present among the bird-rearers. Immunological structure of the population in respect to ornithosis was studied. Patients with various diagnoses showed positive serological reactions retrospectively; there were 155 cases of ornithosis among them.

Animals↗
PubMedDate not supplied

[Isolation of influenza virus with the antigenic formula Hav4 Nav2 and Hav5 Nav2 during epizootic infection among sea gulls in the Astrakhan district in the summer of 1976].

Virological examinations of nesting colonies of Laridae birds in the avandelta of the Volga, Astrakhan region, during an epizootic in July, 1976, yielded 5 strains of influenza virus: 3 from Sterna albifrons, 1 from S. hirundo and 1 from Larus genei, identical with A/Sterna hirundo/S. Africa/61 (Hav5 Nav2) virus. At the same period two strains were isolated from Larus genei showing a previously unknown composition of the surface antigens: Hav4 Nav2. All the strains were isolated only from cloacal washings.

Animals↗