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Results for “Cerebral Hemorrhage, Traumatic”

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At least 19 recordsLinked to original sources
PubMed1996

Non-traumatic cerebral hemorrhage in Saudi Arabs: a hospital-based study of 243 cases.

This is a retrospective study involving 243 Saudi Arabs with non-traumatic cerebral hemorrhages confirmed by CT scan. Intracerebral hemorrhages accounted for about 20% of all strokes in this population. THe male to female ratio was 2.74 which is slightly higher than those reported from elsewhere. Peak frequency was around 60 years. Hypertension was the most common cause accounting for 64% of the cases, hemopathies/coagulopathies for 10% and arteriovenous malformations (AVMs) for 6%. THe cause was undetermined in 21% of patients. Amyloid angiopathy was probably underrepresented due to lack of autopsy correlation. The etiologies were, however, age-related with a clear predominance of AVMs and coagulation disorders before the age of 40 years. Hypertensive hemorrhages were located in the putamen in 46% of cases compared to 20% in non-hypertensive patients while 52% of non-hypertensive hemorrhages were lobar in location compared to 20.5% in hypertensives. Early mortality (19%) in our series was relatively low compared to reports from the industrialized world.

Adolescent↗
PubMed1985

[Central traumatic cerebral hemorrhage in the computed tomogram].

Traumatically caused central brain haemorrhages are relatively rare. From 1976 through 1984 3598 patients were submitted to computed tomography examination in our department after craniocerebral trauma. Central lesions could only be found in about 3% of the injured. They generally occurred in combination with other severe damage of the skull and skull contents. Most frequently, such central haemorrhages could be found in the basal ganglia occasionally extending into the adjacent medullary layer resulting in large intracerebral haematoma. The right hemisphere was affected significantly more frequently and more severely by such large-size haemorrhages than the left hemisphere. The thalamus region ranking second in localisation of central traumatic haemorrhages was virtually never found to be the origin of large haematomas. In general, no definite distinction could be made between primary and secondary traumatic haemorrhages. We identified, however, a few cases of purely central bleeding without accompanying brain lesions. These haematomas, which were most probably caused primarily by trauma, as well as those with associated damage, preferred the regions of basal ganglia and thalamus. The prognosis of central brain haemorrhages was relatively poor with a 42% lethality rate. However, it depended on the severity of the accompanying brain lesions. Thus, isolated central haemorrhages even had a markedly favourable prognosis. The number of survivors of central bleeding turned out to be approximately the same as the number of deaths, the ratio thus being 1:1. Nevertheless, we think that especially small lesions occur more frequently and have a better survival rate than had been supposed up to now.

Basal Ganglia↗
PubMed1999

[Non-traumatic cerebral hemorrhage in childhood: etiology, clinical manifestations and management].

OBJECTIVE: Non-traumatic intracranial hemorrhage is a rare occurrence in children, with the most frequent etiology being vascular malformation. Our aim was to review the etiology and management of spontaneous cerebral hemorrhage in children. PATIENTS AND METHODS: We have reviewed the patients admitted to our intensive care unit (ICU) with spontaneous intracranial hemorrhage from 1980 to 1998. The etiology, symptoms and their clinical management were analyzed. RESULTS: Forty patients with non-traumatic intracranial hemorrhage were admitted during this time period. The mean age was 6.8 years and the male/female ratio was 2.4/1. This condition presented with a sudden onset in 83%. The most common form of presentation was headache, vomiting and altered consciousness. The most frequent etiology was vascular malformation (32.2%). Initial management was symptomatic and addressed the prevention or treatment of intracranial hypertension and seizures. Mortality was 38% and there were sequelae in 35%. CONCLUSIONS: Management of critical non-traumatic intracranial hemorrhage consists in preventing or treating cerebral hypertension. The monitoring of intracranial pressure, jugular oxygen saturation and transcranial Doppler allows an indirect measurement of cerebral blood flow. Prompt excision of the hematoma improves the outcome. In vascular malformations, endovascular embolization or radiosurgery are possible.

Adolescent↗
PubMed1996

[Stereotaxic therapy of post-traumatic cerebral hemorrhage].

The authors present five case-histories pertaining to stereotactic evacuation of lobar traumatic cerebral haematomas and indicate new ways of their treatment. It ensure already from initial observations of the authors that stereotactic operation of these haemorrhages is justified and useful in haematomas affecting several cerebral lobes simultaneously or in case of symmetrical haematomas.

Adult↗