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Correction of entropion from Stevens-Johnson syndrome: use of nasal septum and mucosa for severely cicatrized eyelid entropion.

The resistant entropion, especially of the upper lid, that results from the persistent contraction of the conjunctiva after Stevens-Johnson syndrome is difficult to correct. Grafts of buccal mucosa have relieved this condition for only a few weeks or months. Because of the rigidity of the nasal septum, a sector of this structure, with the perichondrium and mucosa intact on one side, has been grafted into the posterior layer of the upper lid at the margin to turn the lashes and skin away from the globe. For more than two years postoperatively, this graft of septal mucosa has relieved patients of their entropion, and from all indications this correction will be permanent.

Conjunctiva

Tarsotomy for the treatment of cicatricial entropion with trichiasis.

Transverse tarsotomy and lid margin rotation is a simple procedure that is effective in repositioning the entropic lid margin without requiring external incisions or grafting. We report the results of this procedure in 81 eyelids of 58 patients with cicatricial entropion and trichiasis who were followed up for a minimum of 6 months after surgery. Fifty-nine (94%) of sixty-three eyelids with mild to moderate cicatricial entropion were cured with this procedure. Patients with severe cicatricial entropion had a lower success rate with initial tarsotomy (55%), but in these patients the procedure had minimal complications and repeating the operation resulted in a higher success rate. Tarsotomy and lid margin rotation produces excellent cosmetic and functional results when used to treat patients with mild to moderate cicatricial entropion. In cases of more severe cicatricial entropion, we still recommend it as the initial procedure after which more complex modalities may be used if needed.

Adolescent

Tarsal grafting for correction of cicatricial entropion.

A technique of tarsal grafting was used as a simple yet effective procedure for the correction of upper-lid cicatricial entropion. A tarsal graft, 3 to 4 mm in height, excised from the superior tarsus of the donor upper eyelid and placed in a marginal tarsotomy, vertically expands the posterior lamella of the involved lid. This graft also provides a base for suture fixation of the everted lid margin. One case of unilateral cicatricial entropion corrected with a contralateral tarsal graft and one case of bilateral cicatricial entropion corrected by ipsilateral tarsal grafting demonstrate results obtained with this technique. Functional and cosmetically satisfactory results have been achieved with this technique. No recurrence of cicatricial entropion has been noted. With this procedure, tarsus provides excellent graft material for correction of the eyelid deformities associated with cicatricial entropion.

Adult

Spastic entropion after cataract surgery.

Spastic entropion is an acute eyelid condition seen in patients with acute inflammatory ocular conditions. It has been reported after cataract surgery. We describe three cases of spastic entropion after cataract surgery that did not resolve after the ocular irritation subsided. All were associated with eyelid and/or cul-de-sac injection of antibiotics and corticosteroids or anesthetic solution. All had dehiscence of the capsulopalpebral fascia. Spastic entropion is an evolving stage toward permanent entropion.

Aged

Senile ectropion and entropion: a comparative histopathological study.

A microscopic histopathological study was done on 500 full-eyelid-thickness surgical specimens: 25 with the diagnosis of senile ectropion and 25 with that of senile entropion. Five different staining techniques were used. There appears to be significantly more orbicularis and Riolan's muscle ischemia, atrophy, and collagen fragmentation with ectropion than with entropion. Entropion shows more septal and tarsal atrophy. In both conditions, the skin and conjunctiva show chronic inflammation and scarring as a constant feature. Statistical significance at the 1% level was present for all six characteristics studied. These histopathological changes, if not etiological, are at least concomitant features differentiating senile ectropion from entropion at the microscopic tissue level.

Aged

Senile entropion. Pathogenesis and treatment.

Lower lid entropion is owing to a defect of the lower eyelid retractors that causes attenuation of the aponeurosis. Twelve patients with lower lid entropion underwent surgery in which a procedure that reapproximates normal lower lid anatomy was used. There were two recurrences of the entropion condition in the twelve patients.

Aged

Senile entropion.

A new surgical procedure has been developed for the correction of senile entropion of the lower lid. The procedure consists of horizontal shortening of the lower lid by a pentagonal wedge resection combined with an infratarsal eyelid suture technique. Twenty eyelids of 13 patients with entropion have been operated on over the last 4 1/2 years with use of various modificatons of this procedure. The average follow-up period is two years three months, with the longest being 4 1/2 years and the shortest being three months. There has only been one temporary recurrence and no overcorrections. The technique is simple and effective when applied to chronic senile entropion unassociated with severe enophthalmos or any cicatricial component.

Aged

Senile (atonic) entropion.

The etiology and surgery of senile entropion are reviewed. Many of the presumptive causes of this clinical entity including the vague neurological etiology which caused it to be labeled "spastic" have been found baseless, and the explanation of the elder Fuchs, later confirmed by Duke-Elder, that the cause of senile entropion is due to degenerative tissue changes has been found to be more logical and more compatible with the anatomic findings. There have always been two methods of surgical repair of senile entropion: (1) unwinding the lid by resection of horizontal strips of skin or skin and muscle, and (2) by resection of vertical spindles and triangles of tissue to tauten the lids horizontally. I prefer the latter technique.

Aged

Involutional entropion. A review with evaluation of a procedure.

The pathophysiologic changes that lead to involutional entropion are discussed. The Quickert entropion operation gives good results because it rectifies the effect of enophthalmos; it restricts the upward movement of the preseptal muscle; it repairs the relaxation or disinsertion of the lower eyelid retractors; and it corrects the buckling of the upper tarsal border. Using this procedure, the recurrence rate of 3.7% is comparable with that of other procedures.

Adolescent

Ectropion following entropion surgery: an unhappy patient and physician.

Ectropion following entropion surgery is an uncommon situation that creates an unhappy patient and physician when it occurs. The precipitating events that lead to this situation may be related to a faulty understanding of the pathophysiology of the entropic process, utilization of an inappropriate technique, overcorrection of entropion repair, bleeding into the operative field with subsequent scarring of the lid, or a combination of the above. Correction of this situation requires restoration of the normal anatomy where possible, release of any scar bands, and often a lid-tightening procedure.

Aged

Cautery for lower lid entropion.

A prospective study was undertaken to evaluate a simple cautery technique for the correction of involutional lower lid entropion in 50 patients. After a 12 month follow-up period all patients were free of entropion. Only one patient needed to have the procedure repeated because of recurrence. The technique was found to be simple, effective, safe, and required very little time and skill.

Aged

Neglected lid deformities causing progressive corneal disease. Surgical correction of entropion, trichiasis, marginal keratinization, and functional lid shortening.

The potentially blinding abrasive deformities of the lids--trichiasis, districhiasis, entropion, and marginal keratinization--and the defects in lid closure resulting from functionally shortened upper or lower lids should be corrected surgically before contact lenses are fitted or corneal grafting undertaken. The choice of procedure for surgical correction of various degrees of trichiasis-entropion is indicated by the various associated findings, such as thickness of tarsal plate, presence of functional shortening of the upper lid, and involvement of the lower lid (Table II).

Congenital Abnormalities

A tarsal resection procedure for senile entropion with lid retraction.

1. A method of tarso-conjunctival resection for correction of spastic entropion associated with lid retraction has been presented which prevents a) the presence of suture knots against the cornea, b) localized trichiasis, and c) tissue reaction to absorbable sutures. 2. These advantages are achieved through the use of several figure-of-8 silk sutures which are tied on the skin surface. 3. By keeping the apex of the excised triangle of tarsus 2 mm below the lid margin trichiasis may be avoided. 4. Twelve operations have been followed for 1 to 6 years. The retraction and entropion were markedly improved in eleven lids. One under-correction resulted. There were no other complications.

Entropion

Senile entropion: modified Schimek operation.

Senile entropion is a common cause of ocular discomfort. Medical treatment is ineffective for it and a definitive operation generally is required. Many surgical procedures are available, but most of them are not effective. The operation described in this paper was devised by one of us (H.G.S.) 9 years ago, employing a modification of the Schimek procedure. It has the advantage, however, of fixation of the sutures to the periosteum of the lateral orbital rim and thus provides firm support for the lid septum. The operation is simple, safe, and takes little more. Even if entropion recurs, the operation can be repeated with no damage to the lid.

Aged

Congenital entropion in a litter of rabbits.

Conjunctivitis and blepharospasm were observed in a litter of four, 2-week-old New Zealand white rabbits. Corneal opacity and neovascularization of the cornea and entropion of the upper eyelids were observed when the rabbits were examined 10 weeks later. Conjunctival cultures of these rabbits yielded normal bacterial flora. Treatment with a steroid-antibiotic ophthalmic ointment did not alter the ocular abnormalities. Surgical correction of the entropion resulted in a complete regression of all clinical signs.

Animals

Experiences with the Fox technique for the repair of senile entropion.

The author reports on the advantages of Fox's technique in the surgery of senile entropion on the basis of experiences with 30 interventions on 26 patients. In the evolution of senile entropion the structural changes in the orbicularis oculi muscle plays a decisive role, which leads to the malfunction of the muscle. The degenerative changes of the muscle are shown with electron microscopy. The author recommends the Fox technique because this type of intervention aims to abolish the cause of the aging process and not only the consequences of it as other methods do.

Aged

Senile entropion.

129 cases of senile entropion operated on by the Oculo-Plastic Service of the Manhattan Eye, Ear and Throat Hospital were reviewed. There was an overall success rate of 84 percent. Age, sex, race and suture material were of no importance to the eventual outcome of the surgery. A Basedown-Tarso-Conjunctival resection greater than 7 mm had 100 percent results. The orbicularis transplant with insertion at the infero-lateral border of the orbit was found to be the most effective procedure for re-operations for non-cicatricial entropion.

Age Factors