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At least 19 recordsLinked to original sources
PubMedDate not supplied

[The neurogenic aspects of the pathogenesis of glossalgia].

Comprehensive clinical, biochemical, and electrophysiologic examinations of 250 glossalgia patients have revealed disintegration of supra-segmentary vegetative formations: increased activity of sympathoadrenal and serotonin systems, depressed kallikrein-kinin system, changed level of salivary electrolytes, reduced blood supply to the facial and oral tissues and decreased sensitivity of these tissues. Based on these findings, the authors suggest a scheme of glossalgia pathogenesis, with disorders in the autonomic nervous system underlying the development of the condition. Correction of the detected shifts contributes to a more effective therapy of glossalgia.

Adrenal Glands↗
PubMed1988

[Neurophysiological analysis of the central mechanisms of glossalgia].

Bioelectrical activity of the brain was studied in 20 patients with glossalgia characterized by a most severe clinical symptomatology. Neurophysiological analysis has shown a marked imbalance of the leading neuromediator cerebral systems, namely, deficiency of ascending activating reticular effects, tense functioning of syncronizing systems, and involvement into the chain of impaired relationships of the limbic-reticular complex and inhibitory systems of the brain. It has been established that general disorganization of the activity of the cerebral mechanisms is attended by impaired control of ascending afferentation. As a result, any type of chronic stimulation of the peripheral receptors is no longer blocked when entering the central nervous system and manifests in a clinically marked syndrome of glossalgia. On the basis of clinico-physiological correlations the authors draw a conclusion that the syndrome of glossalgia should be considered as one of the links in the chain of chronic emotional stress manifestations.

Adult↗
PubMed1981

[Urinary excretion of vanilmandelic acid in the glossalgia syndrome].

Under observation there were 78 patients aged 41 to 70 years suffering from the glossalgia syndrome, as well as 25 clinically healthy subjects. An activation of the sympathoadrenal system was revealed in the patients. This was ascertained on the basis of the data on vanillylmandelic acid excretion with the urine. The excretion of that acid is known to be dependent on the intensity of the paraesthesias, the duration of the ailment and the character of concurrent visceral diseases. The results obtained are regarded as evidences of the participation of the vegetative nervous system in the mechanism of the glossalgia syndrome development.

Adrenal Medulla↗
PubMedDate not supplied

Use of transcutaneous electrical nerve stimulation in the complex treatment of glossalgia.

Thirty-four patients with glossalgia were studied. The painful part of tongue was electrically stimulated using an 'Elektronika-2M' apparatus with a current of 45 microA for 10-20 min (current strength was patient-controlled to produce the sensation of local prickling): courses consisted of 10-12 treatments. The results of electrical stimulation therapy were compared with results obtained using standard methods of treatment (novocaine blockade, analgesics, etc.), which were used in a control group of 30 patients. Transcutaneous electrical nerve stimulation was highly effective: improvements were noted after the first session, and significant reductions in pain syndrome occurred after 1-3 sessions; therapeutic effects were obtained at the end of treatment in all 34 patients, i.e., in 100% of cases, as compared with 70% in the control group, with remission lasting more than one year in 20 patients and from 3-12 months in 14.

Aged↗
PubMed1995

[The use of transcutaneous electric nerve stimulation in the combined treatment of glossalgia].

34 patients with glossalgia were examined. Tongue's pain zones electrostimulation was performed with the aid of "Elektronika - 2M" device. The current of 45 mkA was applied during 10-20 min (current's power was regulated by patients themselves which had being tried to get the slight pricking sensation). The treatment session consisted of 10-12 exposures. The treatment results were compared with generally accepted methods (novocaine blockade, analgetic, etc.) which were used in control group (30 cases). The high TCES efficiency was demonstrated, exactly the improvement was observed just after the first exposure, the significance decrease of pain syndrome was observed after treatment in all patients, (100% cases as compared with 74% in control group) with remissions more than year in 20 observations and from 3 months to 1 year in 14 individuals.

Aged↗