"Learned helplessness," "learned hopefulness," and "learned obsessiveness": effects of varying contingencies on escape responding.
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Conditioning, Operant↗
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Learned helplessness and learned effectiveness: effects of explicit response cues on individuals differing in personal control expectancies.
Female undergraduates (n = 62) who scored as extreme internals or externals on the Mirels Personal Fate Control Scale participated in a partial replication of Hiroto's learned helplessness experiment. Lights were added to the treatment apparatus, which made explicit to subjects the contingency or noncontingency between their responses and the termination of an aversive tone. As predicted, the performance of internals was significantly impaired by uncontrollability (learned helplessness), while that of externals was facilitated by controllability (learned effectiveness). Externals performed as well as internals in the "escapable" condition, but their performance was inferior to that of internals in the control condition. Following "inescapable" treatment, internals performed worse than externals. These results are supportive of Lefcourt's theory of cue explication. Implications for locus of control and learned helplessness research are discussed.
Acoustic Stimulation↗
Stock differences in the susceptibility of rats to learned helplessness training.
Learned helplessness (LH) consists of shock escape deficits evidenced by animals previously exposed to inescapable shock. This phenomenon has shown promise as a behavioral screen for new antidepressant drugs. Unfortunately, some stocks of rats evidence low susceptibility to LH training. Accordingly, male rats from 8 different stocks were tested for susceptibility to LH training. The outbred stocks consisted of Harlan SD, Sasco Holtzman, and Charles River Holtzman. The inbred stocks (i.e. strains) tested were Lewis, Wistar Kyoto, Brown Norway, Fischer F-344, and Buffalo. The Lewis, Brown Norway, Fischer and Sasco Holtzman rats were found to be virtually non-susceptible to LH training. Harlan SD and Buffalo rats evidenced intermediate susceptibilities of 28% and 33%, respectively. Kyoto and Charles River Holtzman rats were the most susceptible at 53% and 55%, respectively. No stock differences between control animals were observed. These results indicate that wide differences in susceptibility to LH training exist in rats from different stocks or suppliers and researchers should be careful to choose subjects from a susceptible stock. Charles River Holtzman and Wistar Kyoto rats appear to be very susceptible to LH training.
Animals↗
Learned helplessness.
1. Nurses described learned helplessness solely in terms of residents not performing the daily activities they were capable of. 2. The relatively strong correlation between the learned helplessness subscale of the MDI and the Beck Depression Inventory suggests that learned helplessness can be maladaptive and dysfunctional. 3. Analysis of attributional style found that personal, stable, and global attributions for negative events were closely associated with LH scores and self-reported depressive symptomatology in this sample. 4. Residents, except in the areas of meals and privacy, generally reported satisfaction with the amount of control they had in their treatment setting.
Aged↗
Hippocampal neuropeptide Y mRNA is reduced in a strain of learned helpless resistant rats.
The learned helpless rat is considered to be one of the better animal models of depression. A genetically inbred strain with a high vulnerability to develop helplessness (LH), as well as a highly resistant strain (NLH) have both been developed. Since the brain peptide neuropeptide Y (NPY) is involved in the regulation of a number of behaviors known to be altered in clinical depression as well as in learned helplessness, we measured the relative level of NPY mRNA in the hippocampus and cortex of control Sprague Dawley (SD), LH and NLH rats. We find that NLH rats have approximately a 30-35% decrease in basal hippocampal NPY mRNA compared with SD and LH rats. By contrast, cortical NPY mRNA and hippocampal pre-proenkephalin and somatostatin mRNA levels were not significantly different in the 3 strains. The data suggest that the regulation of NPY gene expression may be involved in the reduced vulnerability of NLH rats to develop learned helplessness.
Amino Acid Isomerases↗
Effects of bilateral adrenalectomy on the induction of learned helplessness behavior.
In the learned helplessness model of depression, naive Sprague-Dawley rats are exposed to a 40-minute uncontrollable shock training and are subsequently tested in a shock escape paradigm. "Helpless rats" exhibit 11 to 15 failures in a 15-trial test while "nonhelpless" rats and naive controls score 0 to 5 failures in the same test. We report on the effect of bilateral adrenalectomy on the induction of learned helplessness. Most of the adrenalectomized rats (70%) became helpless whereas sham controls responded to the training and testing similarly to naive nonoperated rats (20% to 30% helpless). This increase in behavioral deficits after adrenalectomy was reversed by administration of corticosterone, the naturally occurring glucocorticoid in rat. We conclude that secretion from the adrenal cortex is necessary for the incorporation of a learned response after stress and that a dysregulation of the hypothalamic-pituitary-adrenal axis seems to be involved in helpless behavior.
Adrenalectomy↗
Learned helplessness at work.
The development of theory and research on learned helplessness is reviewed and criticized on some points, e.g., for its reliance on artificial laboratory experiments. Some empirical findings are presented, indicating a connection between certain work characteristics and learned helplessness. Other research traditions have emphasized the importance of job qualifications, freedom of action, and development possibilities for well-being and health. There is, however, hardly and research on learned helplessness at work. Learned helplessness hypotheses should be tested on data from real life; if applied to work environment research, the theory of learned helplessness could generate important results.
Achievement↗
Effect of beta-adrenergic and serotoninergic agents on the stress-induced behaviour of "learned helplessness" in rats.
The phenomenon of "learned helplessness" is characterized by elements of motor, cognitive and emotional deficit, and it is particularly suitable to be used as a "model" of behavioural depression. There is no doubt about the central role of the cerebral noradrenaline and serotonin in the stress-induced behavioural deficit. The effects of two known beta-adrenoceptor agonists--salbutamol and trimetoquinol--and the beta-adrenergic blockers propranolol and compound 3B, with beta-blocking activity equipotent to that of propranolol, were investigated under conditions of "prophylactic" and "therapeutic" administration in rats subjected to stress by modelling of the behaviour of learned helplessness. Among the serotoninergic agents, the effects of 5-hydroxytryptophane and of the inhibitor of serotonin synthesis, parachlorophenylalanine, were studied. The experimental results show a favourable effect on the behavioural deficit, caused by the blocking of the adrenergic transmitter system and by increasing the activity of the serotoninergic system.
Adrenergic beta-Agonists↗
Learned helplessness, depression, and anxiety.
The learned helplessness model of depression predicts that depressives should tend to perceive reinforcement as response-independent in skill tasks. Depressed-anxious, nondepressed-anxious, and nondepressed-nonanxious college students estimated their chances for success in a skill or a chance task. (Virtually no depressed-nonanxious subjects could be obtained.) Depressed-anxious subjects showed less expectancy change in skill than nondepressed-anxious subjects, while these two groups exhibited similar expectancy change in chance. Nondepressed-anxious and nondepressed-nonanxious subjects did not differ in either skill or chance. The results for a discrimination learning problem were mixed. The groups did not differ in latency to shut off an aversive noise. So, depressed subjects perceptually distort the outcomes of skilled responding as being response-independent, and they may, under certain conditions, show deficits at learning the consequences of responses. These deficits may reflect learned helplessness and are specific to depression.
Anxiety↗
Learned helplessness in diabetic youths.
Determined the relationships among the learned helplessness attributional style, depression, regimen adherence, and metabolic control in a sample of diabetic youth. Fifty children (20 male and 30 female) between the ages of 10 and 16 completed the Children's Attributional Style Questionnaire and the Children's Depression Inventory. Regimen adherence was assessed with three 24-hr recall interviews conducted separately with patients and their mothers over a 1-week period. Metabolic control was measured by glycosylated hemoglobin A1 (HbA1). Learned helplessness was significantly associated with depression (p less than .001) and HbA1 (p less than .02), but was not associated with regimen adherence. Depression was unrelated to regimen adherence and metabolic control. The adherence measures together accounted for 24% of the variance of HbA1. Significant correlations were obtained between HbA1 and the exercise (p less than .05) and eating/testing frequency (p less than .05) adherence factors, indicating that lower levels of adherence were associated with worse metabolic control. Age, pubertal status, sex, disease duration, and SES were unrelated to regimen adherence and metabolic control. These results support the notion that the learned helplessness attributional style for negative events is related to both depression and long-term metabolic control problems in diabetic youths.
Adolescent↗
Learned helplessness as conditioned inattention to the target stimulus.
Learned helplessness theory explains the impaired performance that follows exposure to uncontrollable outcomes by assuming learned expectation of response-outcome independence that is transferred between tasks. Recent evidence has shown that introducing a second neutral stimulus, contingent on the offset of the uncontrollable stimulus, removes the subsequent interference. This finding has been claimed to support the view that the interference is a result of conditioned inattention rather than of the expectation of response-outcome independence. These conflicting explanations were examined in a series of four experiments that varied induction procedures (passive exposure or inescapability) and stimulus quality (aversive or nonaversive). All four experiments found the predicted interference, but only one, in which passive exposure was combined with an aversive stimulus, obtained results supporting the conditioned inattention hypothesis. We conclude that learned helplessness probably involves more than a single mechanism and that the passive exposure procedure may not be appropriate for demonstrating genuine helplessness deficits.
Adult↗
[Depression and learned helplessness in humans].
The learned helplessness hypothesis as a model of human depression is rather unknown in the journals of german language. This article wants to remove that ignorance and make known that experimentalclinical model of depressive behavior. We want further to stimulate empirical and experimental research. In recent advances attributional and cognitive reflections and determinants got important. We introduce these new developments. Depressed persons attribute failures and negative experiences in an internal, global, and stabile way. A problematic cognitive circle develops and depression gets more intensive and chronically. Therapeutical and preventive implications are also discussed.
Depression↗
Development of a measure of learned helplessness.
The purpose of this study was to develop a measure of learned helplessness. Based on a review of the literature, 50 items were developed for inclusion in the proposed Learned Helplessness Scale (LHS). On the basis of independent reviews by three experts, 20 items were selected for inclusion in the LHS. An individual's response to each item was graded, using a Likert scale. A standardized alpha reliability coefficient of .85 was obtained for a sample of 241 healthy adults. There was a positive correlation between the LHS scores and Beck's Hopelessness Scale (HS) scores (r = .252) and a negative correlation between the LHS scores and Rosenberg's Self-Esteem Scale (SES) scores (r = -.622). These correlations were in the direction postulated by various theorists. A Varimax-rotated factor analysis of the LHS data yielded five factors. Three of these factors tapped content relevant to the attributional styles of learned helplessness. Clinical data were also obtained on samples of oncology, hemodialysis, and spinal cord patients. Because the alpha reliability coefficients of the LHS and the Pearson product moment correlation coefficients between the LHS and the HS and the LHS and the SES were in the same direction, the instrument is believed to have adequate internal consistency.
Adolescent↗
Learned helplessness, test anxiety, and academic achievement: a longitudinal analysis.
The stability of individual differences in test anxiety and learned helplessness over a 2-year period and their relation to concurrent and future school achievement were examined. Several issues regarding the assessment of learned helplessness are also addressed. 82 children were administered measures of test anxiety and helplessness in the third grade and again in the fifth grade. Teachers also provided reports of learned helpless and mastery-oriented behaviors at these 2 grade levels. It was found that: (a) both self-report and teacher-report measures of helplessness were stable over the 2-year period; (b) helplessness in the third grade was related to achievement test scores in the fifth grade; and (c) teacher reports may be a viable means of identifying helplessness. These findings are discussed in terms of cognitive developmental changes in children's understanding of effort and ability, and their implications for the assessment of learned helplessness are outlined.
Achievement↗
Prevention of learned helplessness: in vivo correlation with cortical serotonin.
Learned helplessness (LH) is prevented by pretreatment with acute benzodiazepines (BDZs), subchronic tricyclic antidepressants (TCAs), or escapable stress (ES). We have investigated the role of serotonin (5-HT) in LH prevention by these three prevention paradigms, using microdialysis to measure in vivo 5-HT release in frontal cortex (FC) after LH testing. Rats receiving pretreatment before inescapable stress with any of the three methods of prevention--BDZs, TCAs, or ES--showed escape behavior in the shuttle-box test for LH comparable to naive unstressed controls. K(+)-stimulated 5-HT release in all three groups receiving pretreatment was also similar to naive unstressed controls. Rats receiving saline before inescapable stress showed significantly more LH behavior in the shuttle-box task and had significantly lower 5-HT release as well. This suggests that LH correlates with a significant decrease in intracellular releasable 5-HT in FC, and that three different techniques for LH prevention, acute BDZs, subchronic TCAs, and ES all similarly prevent this 5-HT depletion.
Animals↗
Frustation and learned helplessness.
This article reports the transfer of learned helplessness from one aversive motivator, shock to another, frustration. In experiment 1, animals were trained to approach food in a runway and concomitantly exposed to either escapable, inescapable, or no shock in a different situation. Extinction was conducted in the runway, and subsequently the animals were tested for hurdle-jump escape from the frustrating goal box. Inescapably shocked rats failed to learn to hurdle-jump, whereas escapably or nonshocked animals learned the frustration escape response. Experiment 2 replicated the basic finidngs of Experiment 1 and showed transfer of learned helplessness from shock to frustration when no running response had been first acquired in the runway.
Animals↗
Learned helplessness and urinary MHPG levels in unipolar depression.
OBJECTIVE: Studies of the learned helplessness paradigm in laboratory animals show increased central noradrenergic activity following exposure to uncontrollable stressors. In clinical studies, depressed patients as a group report higher perceptions of helplessness and powerlessness. The authors examined the relationship between perceptions of powerlessness and noradrenergic activity in depressed patients. METHOD: Twenty drug-free patients (12 women and 8 men) meeting DSM-III criteria for major depressive disorder were given the Kobasa Hardiness Questionnaire, which contains subscales measuring feelings of powerlessness, security, and alientation. Concurrently, 24-hour urine samples were collected for measurement of urinary MHPG. RESULTS: Significant correlations were found between MHPG levels and total hardiness scores as well as between MHPG levels and total powerlessness scores but not between MHPG levels and total security or total alientation scores. CONCLUSIONS: These results suggest that depressed patients with high urinary output of MHPG are more likely to show the cognitive features of learned helplessness.
Adolescent↗
Frustration-instigated behavior and learned helplessness.
This paper compares Seligman's recent work on learned helplessness with N. R. F. Maier's work on frustration-instigated behavior published some 30 years ago. Some striking similarities are noted between the two approaches, and it is concluded that some of the differences are more apparent than real and that the learned helplessness model could provide an explanation for the "abnormal fixations" reported by Maier.
Animals↗