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[Diagnostic value of myography in lumbar disk protrusion (author's transl)].
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[Transient complete atrioventricular block caused by ventricular myography].
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[Functional anatomy of the feeding apparatus of the mallard (Anas platyrhynchos L.) structure, movement, electro-myography and electro-neurography (author's transl)].
A causal model for the functioning of the beakapparatus of the mallard during straining is formulated on a qualitative basis. The starting point for this model is an abstraction of the analysed structure of the muscle, bone, ligament system. The system was studied by electromyography and cinematography of the movements. It was attempted to quantify the model. Connections with neurography are shown.
[Value of myography in the differential diagnosis of myopathic diseases].
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Automatic recording and evaluation of neuronography.
The introduction in the 60' s of the Nerve Excitability Test (NET) by Laumans and Jongkees for the diagnosis of Bell's palsy stimulated an interest of quantitative testing of the facial nerve. In 1970 Yanagihara reported the Evoced Myography (EMG; i.e. NET is combined with EMG). Starting with electrophysiological basis of the impulse spread of peripheral nerves May, in 1971, extended this concept by including supraliminal stimuli, i.e. Maximal Stimulation Test (MST), for diagnostic purposes. This method eventually developed into what is known as neuronography (Esslen, Huffmann, Ehrenberger, Satoh). It is undoubtedly a refinement in the question of prognosis of Bell's palsy. Continuing the original concept of May we have developed an electronic device with which to carry out automatically the neuronography that demonstrates an evaluation on a numeric display. We use the conventional bipolar negative square wave for a 1 ms stimulation. We stimulate the angle of the mandible, where the ramus marginalis can be readily found in all patients. Neuronography and Evoked Myography provide earlier a more precise prognosis of facial palsy than Nerve Excitability Test (NET). We developed an electronic device for the Maximal Stimulation Test (MST) of May, M., that is automatic. The device counts the spikes of the evoked myogram. A time window is related to the stimulus to exclude stimulus noise and background muscle noise from the counter. Only spikes seen in this window are processed. The number of spikes generated per stimulus is the measurement of the live axons in a stimulated nerve.
[A electromyographic study of the vestibular reflexes. A clinical experimental study on human beings (author's transl)].
It is reported about myography on testpersons with normal vestibular function as well as on patients after operativ dissection of the VIII. nerve, neuropathia nerv. vest. and M. Menière. On the base of these findings it is supposed that myography proofes to be a useful help in the judgement of the specific influence of the vestibular organs on the motoric system. The myographic changes of the muscular activity after caloric stimulation give exact information about the entire vestibular system and especially about the vestibular-spinal-pathways. The best way of testing the vestibular activity is performed on musculars with symmetric, simultaneous and reziproke intented innervation, f.i. on the sternocleidomastoidei muscels in slight deflected position of the head. Most significant for the vestibular influence are an increase of the base-activity and the appearance of groups of potentials which are related to the vestibular stimulus in time and intensity.
[Irritable colon. Possibilities for an objective diagnosis and its value for the therapy].
Diagnosis of functional gastrointestinal disorders consists mainly in exclusion of organic diseases. A more positive diagnosis may be established by the identification of characteristic symptoms. Myography and manometry within the colon sigmoideum reveal significant patterns in irritable bowel patients, which also may allow control of therapy.
Guidelines for evaluating endothelial function in vascular tissue.
The endothelium plays a central role in maintaining vascular homeostasis by orchestrating vascular tone, inflammation, healing, permeability, and thrombosis. Assessing endothelial function in vascular tissue is essential for understanding the cellular and molecular mechanisms underlying cardiovascular physiology and pathology. Traditional approaches, such as wire and pressure myography, have been instrumental in defining endothelium-dependent responses and identifying key pharmacological targets. However, the complexity and heterogeneity of endothelial cells across vascular beds and their dynamic phenotypic changes in health and disease necessitate the incorporation of new investigative strategies. Emerging methodologies, including bulk and single-cell transcriptomics, proteomics, and advanced imaging, now provide unprecedented insights into endothelial cell diversity and function. A team of leading experts in the field, who collectively reached a consensus on the most widely used techniques to evaluate endothelial function, developed these guidelines. The document establishes best practices for assessing endothelial function, from endothelial cell cultures to isolated vascular tissues, integrating conventional functional assays with modern molecular approaches. By fostering methodological consistency and embracing innovation, our goal is to enhance rigor, reproducibility, understanding, and discovery in endothelial biology.
[EMG-findings and nerve conduction velocity in herpes zoster disease (author's transl)].
We have performed a myography on three patients and an additional study of the nerve conduction velocity (CV) on two adult patients suffering from herpes zoster. It could be demonstrated that the affected muscles can suffer at the same time a neuropathy as well as a myopathy. We found fibrillations and CV-reduction also in clinically healthy nerves and muscles. The abnormal findings were most prominent in the clinically affected segments and often suggested an in portio lesion of the motor unit. The infiltration of the spinal ganglion appears to have no paramount significance for the understanding of this disease.
Neurologic complications of thyroid dysfunction.
Until such time as results of more rigorous studies are available, the morbidity rates for thyroid dysfunction cited here must suffice. The 1955 to 1956 outpatient "incidence" for England and Wales was 1.1 per 1,000 for thyrotoxicosis and 1.7 per 1,000 for myxedema (18). United States in-patient "incidence" for 1971 was 0.16 per 1,000 for thyrotoxicosis and 0.13 per 1,000 for myxedema (25). The 1935 to 1967 average annual incidence of Graves' disease for females in Olmsted County, Minnesota, was 30.5 per 100,000 (10). Well over 50% of hyperthyroid patients have clinical evidence of mild or moderate muscle weakness. Usually this weakness is proximal, and electro-myography and muscle biopsy confirm the existence of myopathic process (Table 11). Severe muscular weakness of acute onset is relatively rare and is encountered in approximately 1% of hyperthyroid patients (11,17,40). Ophthalmoplegia and psychosis are reported 4% and 2% of patients, respectively (17). Myasthenia gravis, although well publicized, is estimated to occur in less than 1% of patients (3,30). TPP is virtually nonexistent in the West; in the Orient it is reported in 2 to 8% of hyperthyroid patients and is 20 to 60 times more frequent in the hyperthyroid male than in the hyperthyroid female (Table 12). The neurologic symptomatology of myxedema is more extensive, and agreement among the various series is poor. The only unselected series addressing itself to neuromuscular manifestations of myxedema that is suitable for citation is that of Scarpalezos et al. (36). This comprehensive study was done without apparent patient selection, and it reported 2% of patients with definite carpal tunnel syndrome, 6% with myopathy, and 18% with polyneuropathy (Table 13). Reported percentages of hypothyroid patients found to have neurologic manifestations of cerebellar dysfunction are extremely diverse: ataxic gait was reported in 5 to 32% (6,7,12,27) of patients and dysdiadochokinesia in 6 to 52% (7,12,27). Psychosis is encountered in 2 to 5% (6,14,17,27,39) of myxedematous patients, memory loss in 23 to 55% (6,14,27), and coma in less than 1% (27).
[The importance of electrographic investigations for the diagnosis and treatment of acroparesthesias (author's transl)].
Acroparesthesias may develop as a result of various pathological processes which affect the nerves of the hand. Usually the diagnosis can be made from the history and the clinical findings. If objectifiable neurological findings are lacking, electrographic investigations must be carried out. If several diseases are present, electrography (stimulation and myography) is particularly important for diagnosis and therapy. In compression of the median nerve in the carpal tunnel, the speed of sensory conduction is delayed in about 85%, an anomalous motor latent duction is delayed in about 70%. A distal motor latency of the median nerve of more than 8 m/sec is an absolute indication for a surgical intervention. Signs of denervation of the thenar muscles also required operative treatment.
[Characteristics of disorders of body functions in post-thrombophlebitic disease].
397 patients with chronic diseases of the extremities veins were examined, 256 of them suffered postthrombophlebitic disease. Along with the general clinical examination special investigations were conducted: ECG under orthostatic stress conditions, test tube and instrumental auto-coagulograms, thromboelastograms, biomicroscopy of the anterior ocular segment, studies of the intraocular pressure, ocular fundus, rheovasography, skin thermometry, oscillometry (with nitroglycerin stress), the extremity volume under orthostatic stress conditions, myography, non-contrast roentgenography of the lower extremities, and also the phenomenon of intradermal diffusion and pheblography. It was found that with the progress of postthrombophlebitis trophic disorders are observed in all tissues of the involved extremity. Postthrombophlebitic disease is associated not only with local involvement of vessels, but also with functional disturbances of other organs.
Local cooling in myasthenia. Improvement of neuromuscular failure.
Weak myasthenic muscles were tested by nerve stimulation both at normal temperature and after local cooling that was accomplished by exposing the skin to ice bags or cold paraffin oil. Reduction of intramuscular temperature from 35 C to 28 C increased the voltage of the bellytendon electrical response, the force of the tetanus elicited by 10/sec or 20/sec nerve stimulation. The myasthenic decrement of successive muscle responses was less marked after cooling, as were "delayed rundown" and "postactivation exhaustion." All these effects were reversed on rewarming the muscle. The abnormal neuromuscular jitter in motor-unit components was also reduced by local cooling. These observations may explain why diagnostic application of repetitive stimulation may be false-negative; muscle temperature must be controlled.
Fibrous myopathy. A rheumatic complication of drug abuse.
A progressive fibrous myopathy may result from chronic intramuscular drug abuse. This complication may mimic other rheumatic disorders and early recognition may prevent disability. The patient described here presented with fixed flexion and extension contracture of hips and knees, respectively, after abusing meperidine and other agents for 3 years. Soft tissues of thighs and buttocks were "wood hard," EMG showed absence of action potentials in affected muscles, and biopsy revealed extensive replacement of muscle with dense, acellular fibrous tissue. Possible mechanisms are discussed.
Hill's equation applied to reconstituted contractile collagen.
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A method for mechanographical recording of muscle tone in the rat: the effect of some antiparkinsonian drugs on rigidity induced by reserpine.
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Primary versus delayed-primary neurorrhaphy in rat sciatic nerve.
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