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At least 19 recordsLinked to original sources
PubMed2002

Osteochondroma of the cervical spine--a surprising finding in a liver transplanted patient with polyneuropathy and polyradiculitis: case report.

BACKGROUND: Osteochondroma of the spine is a rare condition. We report a case of a patient with a cervical osteochondroma presenting with a polyneuropathy and polyradiculitis simultaneously. CASE DESCRIPTION: In a liver-transplant patient with progressive neurological deficits a polyneuropathy and a polyradiculitis were diagnosed. Eventually the patient became quadraparetic and an osteochondroma compressing the cervical spinal cord was found. The patient's neurological symptoms markedly improved after gross total tumor resection and antibiotic therapy. CONCLUSIONS: Review of the literature reveals this case to be an unusual presentation of a cervical osteochondroma, its diagnosis being delayed because of concomitant neurological diseases.

Bone Neoplasms↗
PubMed1976

[Cranial polyradiculitis or the so-called Fisher syndrome].

In the light of 3 personally observed cases a description is given of polyradiculitis cranialis or socalled Fisher's syndrome, a separate form of Guillain-Barre-Strohl polyradiculitis. It is a combination of acute ataxia, areflexia and ophthalmoplegia. The cause is probably an immunological reaction to a harmless infectious disease. Reversible or limited demyelination is assumed. The demyelination occurs particularly in the peripheral nerves and their spinal roots, but also in the spinal-cerebellar pathways and possibly in the nuclei of the cranial or optic nerves. Diagnosis is based on detection of "albumino-cytologic dissociation" in the cerebrospinal fluid and the finding of a lesion of the peripheral motor neuron in the EMG. Special drug therapy is unnecessary. The prognosis is good: complete remission is usually observed.

Abducens Nerve↗
PubMed1999

[Progressive muscular weakness due to subacute postinfectious polyradiculitis and myelitis].

In a 67-year-old patient, generalised stable muscular weakness preexisting for several years became rapidly progressive within a few weeks prior to hospitalisation. He died one month after admission from acute cardiocirculatory failure. There was no history of muscular pain, clinical examination showed weak or absent tendon reflexes, hyposensibility of the dorsa of his feet, fasciculations and myocloni of the muscles of the lower limbs as well as a generalised muscular atrophy. Polyneuropathy due to diabetes mellitus and monoclonal IGG-kappa-type gammopathy were preexisting. CSF examination showed inflammatory cerebral fluid changes and further investigations revealed inflammatory polyradiculopathy affecting mainly motor nerve fibres. There was evidence of a reactivated varicella-zoster infection in serum and in the cerebrospinal fluid samples. The search for a tumour, vasculitis or a drug-related cause for this syndrome remained negative. Neuropathological examination at autopsy showed subacute polyradiculitis accompanied by myelitis. The most probable cause of this disorder is immune-mediated polyradiculitis after varicella-zoster infection.

Aged↗
PubMed1991

[Clinical symptoms and diagnostic criteria in polyradiculitis--Landry Guillain Barré].

Acute polyradiculitis ("acute inflammatory demyelinating polyradiculitis--AIDP" or the Landry-Guillain-Barré syndrome--GBS) is an acute inflammatory disease of the peripheral nervous system. Despite extremely severe courses and complications, the prognosis is favourable for the majority of patients. The typical clinical course is featured by non-characteristic sensory symptoms following an infection, with ensuing ascending motor signs and symptoms which, in 80% of the patients, reach a maximum within two weeks. The legs are usually involved before the arms. About 50% of the patients show involvement of cranial nerves. In the acute phase, respiratory insufficiency and autonomous dysfunctions may occur. For diagnosis, predominantly clinical criteria are used according to the criteria summarized by Asbury.

Autonomic Nervous System Diseases↗
PubMed1988

[Findings in the peripheral motor system following stimulation of the motor nerve roots: polyradiculitis, amyotrophic lateral sclerosis and polyneuropathy].

The peripheral nervous system was stimulated over the vertebral column by brief, high-voltage condenser discharges. Potentials from proximal and distal muscles were recorded. We examined healthy subjects (N = 28), patients suffering from ALS (N = 5), from polyradiculitis (N = 6), and polyneuropathies (N = 8). In ALS latencies were slightly (in most cases less than 20%), in polyradiculitis latencies were markedly increased (30-40% on average). The latencies of patients suffering from polyneuropathies showed a variable pattern of vulnerability. Moreover, we could show that this method is frequently capable to quantify deficits in the peripheral nervous system when F-waves are absent.

Amyotrophic Lateral Sclerosis↗
PubMed1987

Unintended plasma exchange therapy in poliomyelitis: difficulties in the differential diagnosis of paralytic poliomyelitis and polyradiculitis.

During the recent polio epidemic in Finland, 3 patients were initially misdiagnosed as Guillain-Barré polyradiculitis, and 1 of them was treated with plasma exchange. The follow-up until 2 years showed no difference in the recovery between this patient and the 2 other poliomyelitis patients treated without plasma exchange. The importance of the differential diagnosis between acute poliomyelitis and acute Guillain-Barré polyradiculitis is emphasized. An evaluation scheme includes sequential physical and cerebrospinal fluid examinations, together with viral antibody determinations.

Adult↗
PubMed1976

Gram-negative bacillary meningitis therapy. Polyradiculitis following intralumbar aminoglycoside administration.

Gram-negative bacillary meningitis is a serious threat to patients with head trauma or altered immune systems, or those who have had neurosurgical procedures. The aminoglycoside antibiotics administered systemically and into the cerebrospinal fluid (CSF) have proved useful in the treatment of these infections. Intralumbar and parenteral gentamicin sulfate and tobramycin sulfate were administered to an acromegalic woman with Klebsiella meningitis. She had objective evidence of adhesive arachnoiditis and symptoms of polyradiculitis. Cessation of the aminoglycoside administration resulted in symptomatic improvement, and reinstitution of therapy caused a recurrence of neurologic symptoms. Polyradiculitis may be a complication of the intralumbar administration of aminoglycosides.

Adult↗
PubMed2002

[Problems of differential diagnosis in tick-borne encephalitis-induced polyradiculitis].

In about 10% of cases, tick-borne encephalitis (TBE) presents with additional myeloradiculitic features mimicking acute poliomyelitis, which can rarely appear as the sole symptom. We report on a 59-year-old man infected with TBE in Thuringia,Germany, who developed polyradiculitis with rapidly progressive, predominantly proximal tetraparesis and respiratory failure. We discuss the differential diagnosis and the epidemiological relevance in conjunction with a second typical case of TBE acquired in the same region and time period.

Antibodies, Viral↗
PubMed1994

Recurrent acute inflammatory demyelinating polyradiculitis after allogeneic bone marrow transplantation.

We report a patient who developed recurrent acute inflammatory demyelinating polyradiculitis (AIDP) receiving immunosuppressive treatment with cyclosporin and prednisone for secondary chronic graft versus host disease (GVHD) following allogeneic bone marrow transplantation (BMT) from an unrelated donor for chronic myelogenous leukemia (CML). After the second relapse of AIDP, cyclosporin was discontinued and a rapid, sustained improvement of his neurological deficits occurred. The role of cyclosporin and systemic CMV infection in the pathogenesis of AIDP in this patient are discussed.

Acute Disease↗
PubMed1982

Reversible bladder denervation in acute polyradiculitis.

A case of reversible bladder denervation in acute polyradiculitis is presented, in which both motor and sensory bladder involvement could be demonstrated using cystometry and denervation-hypersensitivity testing. Attention is drawn to the differential diagnosis to cauda equina syndromes of other etiologies, and treatment with prompt catheter drainage is emphasized. Catheter removal should be guided by disappearance of denervation supersensitivity, and residual urine control by non-invasive methods is recommended.

Humans↗
PubMed1976

[Acute polyradiculitis. Experiences in an intensive care unit].

30 patients with acute polyradiculitis treated in an intensive care unit between 1960 and 1974 are described. Intensive care in these patients was indicated mainly for respiratory problems necessitating tracheotomy and, frequently, artificial ventilation. The main complications encountered are described in detail: they are infections of the respiratory and urinary tracts; cardiovascular problems (mainly instability of blood pressure and sinus tachycardia, and sometimes lifethreatening arrhythmias); frequent thromboembolic complications in patients not on anticoagulants; and bleeding (especially from the gastrointestinal tract) even in the absence of anticoagulants. A careful search should be made for glomerulonephritis, "Schwartz-Bartter-syndrome" or difficulties in regulating calcium metabolism. The prognosis is independent of the time course of the disease. The mortality of 23% in this seriously ill group of patients is fairly low, but the results as far as healing is concerned are less good. In only 23% was there complete rehabilitation, whereas 34% had minor and 20% major residual symptoms.

Acute Disease↗
PubMed1989

[A case of multiple sclerosis with polyradiculitis and spinal subarachnoid block].

A case of 53-year-old female with multiple sclerosis was reported. In August 1987, she suffered from weakness in her legs and urinary retention. These signs had progressed with incomplete remissions and exacerbations. In March 1988, she developed sensory loss of all modalities below C2 level, spastic paralysis of upper extremities and flaccid paraplegia of lower extremities. Electromyography showed evidence of denervation in affected muscles of all extremities and paraspinal muscles. On lumber puncture, spinal fluid pressure fell to 0 mmH2O after removal of the spinal fluid of 12 ml, and the CSF protein was 740 mg/dl, indicating subarachnoid space block. The oligoclonal band was positive. MRI showed swelling in the cervical and upper thoracic cord, and multiple lesions in the periventricular white matter in the cerebrum. We diagnosed this case as multiple sclerosis in combination with acute polyradiculitis. The spinal subarachnoid block was considered to be caused by the swelling of the spinal cord.

Acute Disease↗
PubMed1977

[Comparison of orthodromic and antidromic sensory nerve conduction. 2. Patients with polyneuropathies and polyradiculitis (author's transl)].

The results of the orthodromically and the antidromically recorded nerve action potentials from a median nerve in 30 patients with clinically slight or beginning polyneuropathies have been compared. Antidromic conduction velocity was slowed in 8 cases, in 8 patients it was normal and in 14 patients no definite antidromic nerve action potential could be recorded. This last finding is not considered as being definitely abnormal. In all but one patient orthodromic nerve action potentials could be elicited. In 28 out of 30 patients the orthodromic method gave pathological findings. Duration of the nerve action potentials and the number of their potential component were the most sensitive single parameters, which were abnormal in more cases than conduction velocity of the fastest fibres. For the detection of slight or beginning polyneuropathies the diagnostic yield of the orthodromic method turned out to be much greater than the antidromic. In contrast, in a group of 19 young patients, which previously had suffered from a polyradiculitis, no significant difference between the two methods was found.

Action Potentials↗
PubMed1980

[Postural blood pressure variations in primary acute polyradiculitis (author's transl)].

We have explored the baroreceptor reflex on nineteen patients free of all cardiovascular diseases having contracted acute primary polyradiculitis. The tilt test (at 30 degrees from the horizontal line) demonstrates that, at the acute point of the disease, there is a dysregulation of the arterial pressure characterised by an orthostatic hypotension with a lack of normal increase of the peripheric arterial resistances. Once the motor activity restituted, this dysregulation disappears without sequelae. The clinical and pharmacological tests prove a failure in the baroreceptor arc and shows that sympathetic and parasympathetic pathways to the heart and vessels are functionally normal. Thus, this study localizes the defect on the afferent pathways conducting the local information perceived on the baroreceptors.

Acute Disease↗
PubMed1980

[Chronic polyradiculitis with cutaneous and endocrine signs suggesting a plasmocytic dyscrasia to IgA (author's transl)].

A case of chronic disabling sensory-motor polyradiculitis occurring in a female patient and followed up for 18 months is reported. Clinical findings were papilledema, hypertrichosis, abnormal pigmentation of the skin, generalized edema, and spontaneous cutaneous necrosis. The diagnosis of a plasmocytic dyscrasia to IgA Lambda was made at a late stage of the disease. There were no signs of a solitary plasmocytoma or of diffuse myeloma. At autopsy, there were mild lesions in the peripheral nervous system, but no plasmocytic infiltration or amylosis was seen on optical or electron microscopy. This case is similar to other clinical cases reported mainly by Japanese authors.

Chronic Disease↗
PubMed1995

[Demyelinating polyradiculitis in neuro borreliosis].

Demyelinating neuropathy is considered a rare complication of Lyme borreliosis. We report a case of meningoradiculitis due to Borrelia burgdorferi in which the neurophysiological analysis showed evidence of restricted demyelinating involvement of the nerve roots and plexus, with no peripheral involvement or signs of distal axonal lesions. Lyme disease, therefore, can in fact be associated with demyelinating polyradiculitis with no peripheral nerve damage.

Aged↗
PubMed1996

[Fulminant cerebello-brainstem encephalitis with polyradiculitis following probable Epstein-Barr virus infection].

Epstein-Barr virus (EBV) infection is occasionally accompanied by acute neurological impairment. The pathogenesis of neurological manifestations with EBV infection consists of primary inflammations of EBV infection, and secondary immunologic reactions. However, their clinical course and prognosis are usually favorable. Here we report a patient with fulminant neurological involvement in association with EBV infection. The patient was a 44-year-old man. One morning he developed ataxic gait and speech following flu-like symptoms. He noticed double vision in the afternoon. He had disturbance of consciousness, bilateral ptosis with mydriasis, opthalmoplegia, facial diplegia, bulbar palsy, and weakness of muscles in extremities and respiratory system on the next day. He required mechanical ventilatory support for a month. His symptoms began to improve gradually two weeks after the onset. Two month later, neurological examinations disclosed severe cerebellar ataxia of the four extremities and ocular movement, cerebellar speech, and moderate weakness in his limbs. Moderate cerebellar ataxia and diminished deep tendon reflexes remained for 8-months. Although he had no physical manifestations of infectious mononucleosis, DNA of EBV was identified in the cerebrospinal fluid (CSF) by the polymerase chain reaction method. From these results, we diagnosed his condition as a cerebello brainstem encephalitis with polyradiculitis associated with EBV infection. The cell counts and protein content of CSF gradually normalized in the early stage of his illness, but CSF protein increased again, and had the peak of 275 mg/dl in about one month. In spite of normalized CSF cell counts, his neurological symptoms persisted. CT scan and MRI studies of the brain and the spinal cord were repeated, but demonstrated no significant abnormalities. Clinical course and CSF findings revealed that his fulminant neurological symptoms were most likely produced by the secondary immunologic reactions following the primary inflammations by EBV infection.

Adult↗