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At least 19 recordsLinked to original sources
PubMed1978

[Acute renal failure and consumption coagulopathy in the puerperium].

Disseminated intravascular coagulation accompanied 18% of cases of acute renal failure during puerperal disorders. We studied in particular 31 cases of disseminated intravascular coagulation with renal failure due to obstetric disorders : 12 post-abortion infections, 3 retentions of a dead foetus, 5 infections, 6 cases of toxemia, 2 retroplacental hemorrhages, two hemorrhages during the third stage, one uremohemolytic syndrome. For diagnosis one relies on the clinical data (bleeding, purpura, shock, hemolysis, visceral involvement) and laboratory data, (coagulation should be supervised throughout the course). The renal lesions are either cortical necrosis, or interstitial nephritis. The analogies with experimental phenomena suggest a human equivalent of the Schwartzmann-Sanarelli syndrome.

Acute Kidney Injury↗
PubMed1979

An explication of 'wind illness' in northern Thailand.

'Wind illness' is a very common complaint among the Northern Thai, yet is rarely recognized by Thai physicians trained in biomedicine. Persons most susceptible to 'wind illness' are adult women who have ever borne a child. Consequently, data were obtained from 415 everparous women, 43% of whom reported ever having had 'wind illness' and 57%. never having had it. In addition, 20 individuals who had ever had the syndrome were followed for case study, and 13 indigenous healers who traditionally treat clients suffering 'wind illness' were interviewed. Their perceptions of the etiology, symptomatology and treatment of 'wind illness' are reported in Part I. Part II is an attempt to define 'wind illness' in terms of biomedicine and as a consequence of fertility. Part III synthesized the emic and etic accounts with explanations for the perdurance of 'wind illness' despite the advances of biomedicine and the recent fertility decline in Northern Thailand.

Adult↗
PubMed1977

Capgras' syndrome presenting as postpartum psychosis.

A 25-year-old white woman was hospitalized with a postpartum psychosis presenting as an affective disorder with the delusion of Capgras. She was treated with electroconvulsive therapy with resolution of her affective and delusional states.

Adult↗
PubMed1979

Psychobiology of mental disorders associated with childbearing. An overview.

The diagnostic issues, classification, incidence, genetic factors, and theories of etiology of psychiatric illness associated with childbirth are reviewed. Psychologic influences, psychosocial factors and the concept of biologic maladaptation associated with prospective motherhood are discussed. Postpartum mental distress is not a unitary phenomenon. The physiology of the puerperium is thus not a cause in itself of any of the symptoms, but rather must be regarded as a contributing or triggering factor acting upon an underlying predisposition. Clinical research of postpartum psychiatric syndromes (PPS) and animal behavior studies are inconclusive. It is suggested that major neuroendocrine research strategies currently used in studying affective disorders and schizophrenia should be applied in studying PPS.

Adrenal Cortex Hormones↗
PubMed1978

Mothers and children in hospital.

Between 1965-1972 244 individual mothers were admitted on 283 occasions accompanied by 287 children to a general hospital unit. This cohort has been followed up to ascertain re-admission to any New Zealand psychiatric facility for two follow-ups at 1-8 years and 5-12 years. A postal survey of attitudes was also carried out. The content of the replies has been analysed, with statistical and anecdotal accounts of patients' and husbands' expressed feelings and attitudes. These are compared with observations in the literature about themes of hostility and anxiety. The circumstances surrounding the homicide of a baby in this series together with our various rationalisations, value judgements and assessment of the situations are described.

Adult↗
PubMed1977

Autoerthrocyte sensitization---a psychogenic purpura?

There are two schools of thought regarding the pathogenesis of this syndrome. Gardner and Diamond and others demonstrated that these patients have a hypersensitivity to extravasated RBCs. They assumed that a fixed tissue antibody reacted with red blood cell stroma producing edema, increased capillary permeability, and further extravasation of blood into the tissues. Ratnoff, Agle, and others, however, believe the syndrome is a psychiatric disorder and that the ecchymotic lesions represent a form of conversion reaction--a mechanism that could explain the patient's myriad complaints. There is evidence that vascular beds are controlled by psychic processes. Neurogenically elaborated kinin-like agents might serve as humoral mediators between the central nervous system and the local tissue reaction.2,4,12 Regardless of the basic etiology, we think the syndrome is more common than has previously been considered and should be thought of in patients with recurrent purpuric eruptions of the extremities.

Adult↗