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Results for “SODIUM CHLORIDE/deficiency”
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The low-salt syndromes in congestive heart failure.
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The low salt syndrome. An extreme example in heart failure.
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Sodium losing nephropathy in the urological patient: presentation of a case associated with renal calculus disease; differential diagnosis and therapy.
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Types of shock-like reaction during transurethral resection and relation to acute renal failure.
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The low salt syndromes in congestive heart failure.
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Dehydration, salt depletion and potassium loss; theoretical considerations.
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Acute salt depletion associated with the nephrotic syndrome developing during treatment with a mercurial diuretic.
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Behavior resulting from sodium chloride deprivation in adrenalectomized rats.
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Delayed effect of ipsilateral intracarotid infusion of sodium chloride on the composition of the parotid saliva of sodium-depleted sheep.
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Influence of salt deficiency on taste sensitivity in human subjects.
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Salt-deficiency heat exhaustion.
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A simple method for detecting the salt depletion syndrome during cardiac therapy.
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Studies on renal juxtaglomerular cells. I. Variations produced by sodium chloride and desoxycorticosterone acetate.
Accumulation of granules in the juxtaglomerular cells occurred in rats which were maintained for 5 to 6 weeks on a diet low in sodium, chloride. Cytological evidence suggests that this was probably a storage phase of secretion following a decrease in the rate of liberation of the granules. Administration of DCA (desoxycorticosterone acetate) to salt-deficient rats did not alter this appearance of the juxtaglomerular cells. Two per cent sodium chloride taken in the drinking water consumed for 4 weeks by similar animals caused degranulation of the juxtaglomerular cells. This effect was enhanced by DCA. DCA administered to animals on a normal salt intake produced a lesser degree of degranulation. Cytological changes in degranulated cells suggested that these represent a stage of hyperactivity in the secretory cycle produced by an increase in the rate of liberation of granules. A hypothesis is suggested that the juxtaglomerular cells are involved in the hormonal regulation of sodium metabolism and/or blood pressure.
Study of some aspects of nervous and muscular activity during experimental human salt deficiency.
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Fibrocystic disease of the pancreas presenting with acute salt depletion.
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A salt wasting syndrome in infancy.
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Cortisone treatment of the low-salt syndrome.
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