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At least 19 recordsLinked to original sources
PubMed1978

[Legal and organizational bases of health care in the textile industry of the Congress Kingdom of Poland in the 19th and beginning of 20th cent. (forms of insurance, work safety and sanitary regulations)].

The origins of state interventionism in the field of health care in the textile industry of the Congress Kingdom of Poland go back to the first half of the XIX-th century. It could not, however, be developed earlier than in the last quarter of this century, at which time it resulted from the class struggle of textile workers, mainly in Lódź and Zyraŕdów. Legal and organizational patterns of health care were of foreign origin, but soon they were modified and adapted to the local conditions of the Congress Kingdom of Poland. It has to be underlined that it was the textile industry that stimulated legal and organizational changes in the field of industrial health care, not only in the Congress Kingdom of Poland but also in the whole contemporary Europe.

Accident Prevention↗
PubMed1977

[Mass examination of women employed in the Lódź textile industry (with special reference to the genitalia)].

The complex examinations of the state of health of 468 women workers of the Central Laboratory of Textile Industry in Lodź were performed. In all women were performed physical and gynecological-biochemical examinations as well as hematological-biochemical ones of the selected blood parameters. It has been stated a high (61,8%) percentage of women with morbid changes or genital organs and lower values of examined blood parameters in spinners, comparing with workers of the finishing department and administration.

Female↗
PubMed1977

[Gas chromatographic determination of biphenyl and 2-phenylphenol in the environmental air of dyeing-plant rooms of the textile industry].

A report is made on a method of sampling and analysis of biphenyl and 2-phenylphenol which are present in the environmental air of the dyeing plant rooms of the textile industry. The sampling is carried out by filtering environmental air through small pipes containing two layers of Porapak Q 80-100 mesh, the first one of 150 mg for the analysis and the other, of 50 mg, for the control, with a flux of about 1 l/min. The report includes the description of some tests concerning shifting and displacement with various fluxes, as well as the recovery of known quantities and the stability of the sampling. The saturation for the mixture of the two pollutants was about 130 mg/g of biphenyl and more than 100 mg/g of 2-phenylphenol. The gaschromatographic analysis was carried out on 2 ml of cyclohexane; methylacetate 80:20 by which was obtained the absorbent used for the sampling. An analytical column was used with 3% Apiezon L, 1% phosphoric acid, 2% Carbowax 20 M on Chromosorb W HP 80-100 mesh. These results were also confirmed by other two reference columns. This method was applied successfully to the analysis of the air of the work environment under examination.

Air Pollutants, Occupational↗
PubMed1992

Urinary mutagenicity and N-acetylation phenotype in textile industry workers exposed to arylamines.

Primary aromatic amines have been identified epidemiologically as human carcinogens. It has been suggested that the target organ affected by aromatic amines is dependent on the rate of metabolic activation. Epidemiological studies have shown an association between low acetyl transferase activity and bladder cancer risk. On this basis, our working hypothesis was that the slow acetylators could follow in a higher extent the metabolic pathway independent of N-acetylation, leading to the excretion of conjugates of electrophyles with glucuronic acid. The instability of these glucuronides could be responsible for the association between arylamine-induced bladder cancer and slow acetylator phenotype. A total of 153 individuals were included in this study: 70 exposed to arylamines (working in textile industry) and 83 nonexposed. The following parameters were determined in urine: mutagenic index in the absence of metabolic activation, S9; mutagenic index in the presence of S9; and the mutagenic index after incubation of the urine with beta-glucuronidase. All individuals were phenotyped according to their capacity of N-acetylation by using isoniazid as drug test. The results show that the mutagenic index after incubation of the urine with beta-glucuronidase is statistically higher in exposed subjects when compared with nonexposed individuals (P less than 0.001), this parameter being statistically higher among exposed subjects who were slow acetylators than among rapid metabolizers, independent of the fact that they were smokers or nonsmokers. There were no significant differences between groups for the mutagenicity in urine not incubated with beta-glucuronidase.

Acetylation↗