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Results for “ULTRAVIOLET RAYS”

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At least 19 recordsLinked to original sources
PubMed2000

[Antioxidants and protection of the skin against the effect of ultraviolet rays].

Ultraviolet radiation causes increase of production of the reactive oxygen species in the human skin, which are responsible for oxidative stress and damage of many important cellular components in the target tissue. The complex of antioxidants represents the major protective system against the oxidative damage of the skin. Many antioxidants, namely vitamin E, can be successfully used in photoprotection.

Antioxidants↗
PubMed1981

[Mechanism of mutant induction in the ade2 gene of diploid Saccharomyces cerevisiae yeasts by ultraviolet rays].

Ultraviolet light (UV) at 3000 ergs/mm-2 induces ade2 mutants with a frequency about 10(-4) in wild-type haploid strains of yeast and about 10(-5) in diploid wild-type strains. UV irradiation effectively induced mitotic segregation of ade2 in the heterozygous diploid (the frequency of segregation is 6%). Interallelic complementation and localization spectra are similar for mutations induced both in haploids and diploids. The occurrence of ade2 mutants in diploids correlated with mitotic segregation of the marker his8 which is situated in the same arm of XY chromosome as ade2 is, distal to the centromere. Our data about the frequency of ade2 mutants in diploids and haploids, the frequency of ade2 mitotic segregation, mitotic segregation of other markers and genetic characteristics of ade2 mutations confirm the suggestion that the major mechanism of diploid ade2 mutants appearance is mutation in one of the two ADE2 alleles and consequent mitotic homozygotisation of mutation as a result of mitotic crossingover between ade2 and the centromere.

Diploidy↗
PubMed1998

[Change in the content of chemical elements in epitheliocytes of the prickle cell layer of granulating skin wounds exposed to ultraviolet rays and ultraviolet laser therapy].

Spinous layer of human epidermis was studied in control group (2 human subjects) and in 16 patients with thermic lesions prior to and following ultraviolet laser therapy (UVLT) of ultraviolet irradiation (UVI). Skin biopsy was performed in each patient on undamaged regions (individual control). Chemical elements (Ca, K, Cl, Mg, Na and Al) content prior to and following treatment was compared. Patients with equal stage (IIIB) and localisation of lesion were selected. Bioptates were obtained for diagnostic purposes. UVLT was established to prompt normalization of the content of chemical elements named in epitheliocytes of human epidermis in IIIB lesions whereas UVI does not cause this effect at the same conditions. It is proposed that this difference is dependent on the origin of appropriate irradiations.

Adult↗