[Distribution of radioactivity in polymorphonuclear leukocytes during phagocytosis and digestion in vivo of denatured gamma globulin-I-131. I. Introduction and methods].
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Animals↗
Biomedical subjects
A Pacini
Publications and source records attributed to A Pacini.
[Distribution of radioactivity in polymorphonuclear leukocytes during phagocytosis and digestion in vivo of denatured gamma globulin-I-131. II. Phagocytosis].
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Animals↗
[Distribution of radioactivity in polymorphonuclear leukocytes during phagocytosis and digestion in vivo of denatured gamma globulin-I-131. 3. Digestion].
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Animals↗
[Distribution of radioactivity in polymorphonuclear leukocytes during phagocytosis and digestion in vivo of denatured gamma globulin-I-131. IV. Fractionation].
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Centrifugation↗
[Catabolism in vitro of denatured gamma globulins. Study with peritoneal macrophages].
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Animals↗
[Catabolism of I-131 serum proteins by polymorphonucleated leukocytes in vitro. II. Study with gamma-globulins and albumin].
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Albumins↗
[Catabolism of I-131 serum proteins by polymorphonucleated leukocytes in vitro. 3. Study with alpha-2 and beta-globulins].
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Alpha-Globulins↗
[Catabolism of I-131 serum proteins by polymrphonucleated leukocytes in vitro. 1. Methods].
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Animals↗
Microfilament organization in human platelets.
Morphological and biochemical studies suggest that actin in human platelets polymerizes from monomers or oligomers into long filaments more tightly structured in activated than in resting platelets. The polymerization and reorganization of actin filaments are regulated by cellular proteins. Profilin prevents actin polymerization by forming tight complexes with monomeric actin; gelsolin acts both severing filaments and inhibiting their elongation by capping at the barbed end; other actin-binding proteins nucleate polymerization and cross-link actin filaments into networks or bundles. The changes in the actin assembly state, which are under the control of calcium, seem essential for pseudopodal formation. Other platelet processes, such as granule centralization and contractile gel formation, are due to calcium-dependent actin-myosin interaction. In addition, Ca++ seems to inhibit through calmodulin the binding of caldesmon to actin, allowing actin linkage to myosin in a 'flip-flop' fashion.
Actins↗
[The 1st Italian family with Scheie's disease or late Hurler's disease as studied by European authors].
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Abnormalities, Multiple↗
Factors regulating plasma protein synthesis. 3. Large scale separation of rabbit fibrinogenolytic products.
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Allergy and Immunology↗
[Thymoleptic activity of a new antidepressive drug benzoxazone (FI 6654). Preliminary clinico-therapeutic observations].
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Adjustment Disorders↗
[Pathogenetic hypothesis and peculiar characteristics of delusions of persecution in the presence of depressive psychosis. Clinical case report].
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Adult↗
[Anorexia nervosa as possible atypical manifestation of endogenous depression. Considerations on a clinical case].
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Adolescent↗
[Clinical considerations on monochemispheric electroshock on the dominant side].
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Adult↗
[Fatty acid composition of cholesterol esters of the cerebral white substance in multiple sclerosis. (Preliminary note)].
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Adult↗
An evaluation of methods for determining the concentration of fibrinogen in rabbit plasma.
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Aminocaproates↗
Metabolism of interferon (facts and fancy).
Although the knowledge of the interferon metabolism is still incomplete, two organs are emerging as elective sites. The kidney and probably the liver may share a major role but only future research will define quantitatively their importance. Many aspects of interferon activity upon cellular processes and its possible function as a 'chalone-type' substance are reviewed.
Animals↗