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Biomedical subjects

C F Beckmann

Publications and source records attributed to C F Beckmann.

44 records · Page 3Linked to original sources
PubMedDate not supplied

Entanglement of a pigtail catheter by the chordae tendineae of the tricuspid valve during pulmonary angiography.

Two cases are presented in which a pigtail catheter was entrapped by the chordae tendineae of the tricuspid valve during pulmonary arteriography. A technique for removal of the catheter from its entanglement by the chordae tendineae is described. Caution must be taken when advancing through the right ventricle a catheter that appears to be entrapped by the chordae tendineae. When such an entanglement occurs, measures to reduce the risk of rupturing a papillary muscle must be taken.

Adult↗
PubMedDate not supplied

Selective embolization of bilateral arterial cavernous fistulas for posttraumatic penile arterial priapism.

A 22-year-old man suffered a hiking accident with perineal trauma and developed a nonpainful priapism secondary to bilateral arterial-cavernosal fistulas. To minimize the risk of impotence in this young patient, successive selective embolizations with autologous blood clot were performed to close the fistulas. This led to an uncomplicated full recovery. No fistula was detectable on Doppler ultrasonography at 1-year follow-up. Review of the literature confirms the safety of embolization with autologous clot.

Adult↗
PubMedDate not supplied

Aneurysm formation in distal saphenous vein bypass grafts as a cause of graft failure.

Aneurysms of a reversed saphenous vein femoropopliteal bypass graft developed in two patients. In both patients, the aneurysm led to thrombosis and occlusion of the graft. One graft was reopened by intraarterial thrombolysis followed by local graft revision; the other required complete regrafting. This uncommon cause of graft occlusion should be considered before thrombolysis.

Aged↗
PubMedDate not supplied

Vascular resistance changes distal to progressive arterial stenosis: a critical re-evaluation of the concept of vasodilator reserve.

Previous studies have shown that with progressive arterial stenosis, resting blood flow does not start to diminish until 85-95% luminal area stenosis is reached. However, during hyperemic states, peak flow starts to diminish at only 40-60% stenosis. An autoregulatory mechanism has been postulated, whereby peripheral arterioles undergo compensatory vasodilatation, thereby maintaining resting flow. During hyperemia, some vasodilator reserve is presumably already used up, resulting in flow dropoff at an earlier stage. We measured flow and pressure and calculated peripheral vascular resistance (Rp) distal to progressive iliac artery stenoses in five dogs. Contrast injections proximal to the stenoses allowed precise angiographic quantitation of the lesions and provided reproducible hyperemic stimuli. Flow-stenosis relationships proved similar to those discussed above, but Rp distal to the lesions failed to show progressive decrease as stenosis increased. Thus, compensatory peripheral vasodilatation does not occur during most phases of progressive arterial stenosis. The contours of resting and hyperemic flow-stenosis curves are not related to the concept of vasodilator reserve but instead are readily explained by basic hydrodynamic principles.

Animals↗
PubMedDate not supplied

Balloon-occlusion superior mesenteric arteriography for improved visualization of the mesenteric and portal venous anatomy of dogs.

The superior mesenteric arteries of twelve dogs were occluded with an inflated balloon on the tip of an endhole catheter while diatrizoate meglumine and diatrizoate sodium (Renografin 76) was injected distal to the occlusion. The balloon was immediately deflated at the end of contrast injection. Films taken over a 33-second period showed consistently excellent visualization of the mesenteric arteries and veins as well as the portal veins. Comparison with studies performed by the conventional, nonocclusion technique confirmed the superiority of the films obtained with the balloon occlusion method. Histologic examination of the mesenteric artery at the site of balloon inflation and of the small and large bowel revealed no abnormalities.

Angiography↗
PubMedDate not supplied

Reactive hyperemia as an indicator of peripheral vasodilation associated with arterial stenoses.

Reactive hyperemia occurs when an artery is temporarily occluded and the occlusion is then released. During a period of vascular occlusion, peripheral vascular dilatation develops distally, and is reversed only gradually after release of the occlusion. Reactive hyperemia was measured in the hind limbs of five mongrel dogs at different degrees of arterial stenosis to determine at what degree of stenosis compensatory vasodilatation of the peripheral vascular bed occurs. In the hind limbs of the dogs, which had been anesthetized, reactive hyperemia as an expression of peripheral vasodilatation did not occur until the critical stenosis (the stenosis at which resting flow started to drop) was reached. It began to occur only beyond the critical stenosis, and then increased as the vessel became totally occluded. This study contradicted the vasodilator reserve theory, which states that during progressive arterial stenosis the peripheral vascular bed dilates to maintain flow until the peripheral vascular bed is maximally dilated, when the critical stenosis is reached.

Animals↗
PubMedDate not supplied

Reactive hyperemia vs. pharmacologic hyperemia in the canine iliac circulation. A comparison.

Different methods of selective vasodilation have been described. One method is reactive hyperemia, which occurs when an arterial occlusion is released, and another method involves injection of vasodilator drugs. Since both arteriography and pressure measurements are often performed during hyperemia, it is important to know whether these two methods differ in the degree or duration of hyperemia achieved. We measured reactive hyperemia in the hind limb of five anesthetized mongrel dogs after arterial occlusion times between 20 seconds' and 20 minutes' duration and after selective intra-arterial injection of 5, 10, and 20 cc of Renografin-76. Only 5 cc of Renografin-76 created greater and longer lasting hyperemia than the maximum reactive hyperemia achieved after 10 minutes of arterial occlusion. For this reason, contrast material-induced hyperemia appears to be preferable to reactive hyperemia as an aid to peripheral arteriography and as an aid to the evaluation of aortoiliac stenoses.

Angiography↗