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Biomedical subjects

D A Pass

Publications and source records attributed to D A Pass.

At least 19 recordsLinked to original sources
PubMed1989

Investigations of an enteric infection of cockatoos caused by an enterovirus-like agent.

An enteric infection in cockatoos associated with a 30 nm diameter enterovirus-like agent seen in faeces and intestinal epithelial cells is described. The disease is characterised by intractable, profuse, mucoid diarrhoea, weight loss, dehydration and death. Lesions in the intestine consist of villous atrophy, villous fusion, enterocyte hyperplasia and, in some cases, chronic inflammation. Affected birds so far examined have concurrent psittacine beak and feather disease.

Animals↗
PubMed1985

Detection of two lectins in haemolymph from the oyster Pinctada maxima.

Two lectins were detected in haemolymph from the oyster Pinctada maxima, a macromolecular (approximately 8 X 10(6) dalton) N-acetyl-galactosamine-binding lectin and a lower molecular weight (approximately 4 X 10(5) dalton) alpha-galactose-binding lectin. The macro-molecular lectin agglutinated human type A erythrocytes to a greater extent than human type B or O erythrocytes and was the major lectin detected in the haemolymph. The 4 X 10(5) dalton lectin agglutinated human type A, B and O erythrocytes to the same extent.

Acetylgalactosamine↗
PubMed1985

Canine laryngeal rhabdomyoma.

Three canine laryngeal tumors were diagnosed as oncocytomas by light microscopy, but were determined to be rhabdomyomas following ultrastructural and immunocytochemical examination. Tumors consisted of large eosinophilic cells interspersed with smaller dark cells. Large tumor cells had a granular, intensely eosinophilic cytoplasm. Scattered through the tumors were a few elongated cells with cytoplasmic cross striations and multiple nuclei. Tumor cells from all three dogs contained numerous mitochondria and bundles of myofibrils with electron-dense Z-lines typical of striated muscle cells. Intracellular myoglobin and desmin were detected in the tumors by immunocytochemistry. Comparisons are made with a previous report of canine laryngeal oncocytomas.

Adenoma↗
PubMed1984

The pathology of psittacine beak and feather disease.

Psittacine beak and feather disease is characterised by loss of feathers, abnormally shaped feathers and overgrowth and irregularity of the surface of the beak. The disease occurs in a number of psittacine species including the Sulphur-crested Cockatoo, Lovebirds , Budgerigars and Galahs . The abnormal appearance of feathers and beak is due to a dystrophic process within the epidermis of the feather and beak. The process consists of epidermal cell necrosis, epidermal hyperplasia and hyperkeratosis. Many of the feather abnormalities are due to retention of a hyperkeratotic feather sheath. A characteristic microscopic finding is the presence of macrophages containing purple intracytoplasmic inclusions in affected epidermis and feather pulp. The inclusions consist of aggregates of particles 17 to 22 nm in diameter. Similar but smaller inclusions occur in epidermal cells. In addition, non-suppurative inflammation occurs in the feather pulp. The findings are suggestive of a viral infection.

Animals↗
PubMed1984

Beak and feather dystrophy in wild sulphur-crested cockatoos (Cacatua galerita).

Three flocks of wild sulphur-crested cockatoos in southeastern Australia had a 10-20% prevalence of feather and beak deformities. In affected birds, the crest, tail, contour and down feathers were lost or reduced in length owing to a "pinching off" 0.5-1 cm above the skin. The underlying skin was discolored brown. Flight feathers appeared normal and could sustain flight. In mildly affected birds, only the crest and down feathers were involved. Histological examination of affected feathers showed necrosis of developing cells in their epidermal collars. The epidermis was moderately hyperplastic and contained numerous large macrophages, with large intracytoplasmic granules. Electron microscopic examination showed these granules to consist of regular lattices of 17-20-nm-diameter particles, resembling parvovirus particles. In some birds the upper beak was enlarged and had longitudinal cracks. Histological examination of the beak showed necrosis of its epidermis. It was concluded that a viral infection may have caused feather and beak dystrophy in these birds.

Animals↗