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D Ye

Publications and source records attributed to D Ye.

95 records · Page 6Linked to original sources
PubMed1990

[Determination of the benzo(a)pyrene content of 20 Chinese herbal drugs before and after charring].

The B(a)P [Benzo(a)pyrene] contents of 20 Chinese herbal drugs before and after charring were determined for the first time. The result shows that after charring the contents of 6 drugs tend to increase, accounting for only 30% of the total, while those of 14 drugs tend to decrease, making up 70%. This suggests that charred drugs are not carcinogenic if used properly. In the paper a discussion is also made of the factors and technological processes affecting the variation of B(a)P contents.

Benzo(a)pyrene↗
PubMed1990

[Effect of dry ginger and roasted ginger on experimental gastric ulcers in rats].

This paper reports the effect of dry ginger and roasted ginger decoctions on four experimental gastric ulcer models in rats at an oral administration of 4.5 g/kg. The result shows that the roasted ginger has an obvious inhibiting tendency on three gastric ulcer models except the indomethacin induced model while the dry ginger has no such effects. The acute toxicity test has shown that the LD50 of roasted ginger decoction administered orally is 170.6 +/- 1.1 g/kg, but it is over 250 g/kg with dry ginger. This suggests that the water soluble constituents of the dry ginger have changed in the roasting process.

Acetates↗
PubMed1990

[Optimal potassium concentration in cardioplegic solutions].

A model of perfused cardiac papillary muscle from guinea pig was set up in our lab. The procaine-free St. Thomas' Hospital solution was used as the basic cardioplegic solution. The potassium concentration of the solution was designed by optimization (14.6, 22.8, 28.5, 32.6, 57.8 mmol/L). The papillary muscle was undergone anoxic arrest for 60 min in 32 degrees C. The effect of myocardium preservation was assessed with cardiac action potential, contractility and quantitative analysis of ultrastructure. We concluded that; (1) in this research the proper potassium concentration is at the range of 20.7-26.0 mmol/L and the optimal one is 22.8 mmol/L; (2) despite any deviation from the range, phases II and III of the action potential changes first. [K+] greater than or equal to 32.6 mmol/L slows conductivity, weakens contractility and damages the subcellular structure severely; (3) if [K+] is 57.8 mmol/L, anoxic arrest for 60 minutes in 32 degrees C damages of both structure and function of myocardium is irreversible; (4) following the hyperkalemic cardioplegia, there is a secondary change of cardiac action potential in reperfusion period, which shows longer phase II and shorter phase III, smaller Vmax and APA; (5) reperfusion arrhythmia after anoxic hyperkalemic asystole is likely caused by all kinds of conductive disorders resulting from the secondary changes of action potential; (6) arrest time, AT (Y, s) has the negative relativity to the potassium concentration (X, mmol/L) of cardioplegic solution, which can regress to curve 1/Y = 0.10-1.38/X(14.6 less than or equal to X less than or equal to 57.8). The minimum potassium concentration which can guarantee AT within 30 seconds is 20.7 mmol/L.

Action Potentials↗