Biomedical subjects
Publications and source records attributed to E G Pearson.
Abomasal fluid was collected from adult cows using a specially designed tube device. The device was placed in the abomasum through a rumen fistula. A filter on the end of the tube filtered out the larger particles so the liquid could be aspirated up a smaller tube placed inside a larger one. The smaller tube reduced the amount of dead space created by the distance from the abomasum to the rumen fistula.
A D-xylose absorption curve was determined on 10 apparently healthy adult cows. The D-xylose was placed directly into the abomasum by abomasocentesis. Various doses of D-xylose were placed in the abomasum of cows with rumen fistula to determine the most useful dose. By giving 0.5 gm of D-xylose per dg of body weight, a blood level was produced that peaked around 90 minutes at 13.8 +/- 3 mg/di. The technique for abomasal centesis was developed by first using embalmed cows and then cows with rumen fistulae.
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The clinical signs of tansy ragwort poisoning are variable and in 5 cases included diarrhea, tenesmus, ascites, bloody feces, icterus, paresis, CNS involvement, rectal prolapse, poor appetite and weight loss. It is thus apparent that the disease can be confused clinically with many others, and tansy ragwort poisoning should be considered in animals exhibiting ascites, diarrhea and rectal prolapse. Several cases of tansy ragwort (Senecio jacobaea) poisoning occurring from 1969 to 1976 are reported to illustrate the numerous clinical pictures that might confront a practitioner. The disease is sporadic in this area. The poisonous principles in tansy ragwort are pyrrolizidine alkaloids, which cause gradual alteration and necrosis of liver cells with replacement by fibrous tissue. The development of signs in all of these cases could be attributed to failure of one or more liver functions.
Megaesophagus was diagnosed in 15 llamas on the basis of survey and contrast radiography. Age of onset ranged from 13 months to 9.5 years. Clinical signs varied, with salivation being the most common; regurgitation or dysphagia were noted in only 33% of the cases. Duration of the disorder ranged from 1 week to 5 years. Organophosphate toxicity was the cause of megaesophagus in 1 llama. While most cases were of unknown etiology, the 3 with histological abnormalities included 1 with vagal neuropathy and 2 with a degenerative myopathy of esophageal muscles. The causes in most were undetermined.