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Biomedical subjects

J Ananth

Publications and source records attributed to J Ananth.

At least 55 records · Page 3Linked to original sources
PubMed1982

Schizophrenia and psychosomatic illness.

In order to explore the possibility of a mutually exclusive relationship between schizophrenia and certain psychosomatic disorders, 354 adult schizophrenic patients, both acute and chronic, were studied with regard to lifetime prevalence of peptic ulcer, bronchial asthma, neurodermatitis and rheumatoid arthritis. Significant evidence to support the hypothesis of mutual exclusivity was found only in the case of peptic ulcer in the overall sample. Prevalence was higher in younger, more acute patients than in older, chronic patients, but this finding was difficult to interpret. Parallel enquiry into the prevalence of schizophrenia and the same disorders among close family members of the patients yielded inconclusive results.

Adult↗
PubMed1982

Benzodiazepines: selective use to avoid addiction.

When judiciously used, benzodiazepines are therapeutically effective and remarkably safe. Long-term use may result in addiction and physical dependence in some patients. The physician's awareness of this risk helps in the prevention of dependence. Four variables play a part in the development of dependence, ie, dose, duration of treatment, the history, and the patient's personality. A dosage higher than the usual therapeutic dose not only is not needed in most patients but produces more side effects. Short term therapy carries a low risk of dependence and is preferred. It is advisable not to use benzodiazepines in patients with a history of alcohol or drug abuse, as dependent personalities pose a higher risk than other personality types.

Anti-Anxiety Agents↗
PubMed1982

Current psychopathological theories of tardive dyskinesia and their implications for future research.

An understanding of pathophysiology is essential for establishing an etiological relationship between the neuroleptics and tardive dyskinesia, for elucidating specific treatment methods and perhaps for synthesizing new neuroleptics which do not produce this side effect. Our current knowledge has been derived from clues obtained by drug-related factors, therapeutic agents assumed to improve tardive dyskinesia and by neuropathological findings. Among the many theories, dopaminergic hypersensitivity is the most favored and yet unproven. At present the etiology of tardive dyskinesia may be related to the interaction between the exogenous drugs and the endogenous predisposition, the nature of which is so far elusive.

Acetylcholine↗
PubMed1982

International comparison of the prevalence of psychosomatic disorders in schizophrenic patients.

An epidemiological survey was carried out in Montreal (Canada). Plattsburgh (USA) and Lodz (Poland) to test the hypothesis that certain psychosomatic disorders show a low lifetime prevalence in schizophrenic patients (total n = 665). The same method for collection and evaluation of demographic and clinical data was used in order to establish the lifetime prevalence of peptic ulcer (PU), bronchial asthma (BA), neurodermatitis (ND) and rheumatoid arthritis (RA) in the patients as well as in their nuclear families. Low values of lifetime prevalence were found in schizophrenic patients in chronic hospitals as compared with those in general-university hospitals. Similarities were found in age- and sex-related patterns of these psychosomatic disorders in both samples. However, PU showed higher prevalence, and BA, ND and RA showed lower prevalence in the Polish sample.

Adult↗
PubMed1981

Familial tardive dyskinesia.

The authors surveyed 500 inpatients receiving long-term neuroleptics; 8 had first-degree family members who were also being treated with long-term neuroleptics and suffering from a psychiatric disorder. The 8 patient-relative pairs showed concordance for the presence or absence of tardive dyskinesia.

Adult↗