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Biomedical subjects

J F Haas

Publications and source records attributed to J F Haas.

31 records · Page 2Linked to original sources
PubMed1984

Epidemiologic approaches to dietary factors in cancer etiology.

The role of epidemiologic studies in the identification and evaluation of dietary factors associated with altered cancer risk is assessed. Possible mechanisms by which dietary factors may influence cancer development are described. Research strategies for investigating diet and cancer are presented with emphasis on epidemiologic approaches. Methods of nutritional assessment of diet are reviewed and related to selected epidemiologic approaches.

Carcinogens↗
PubMed1983

Ethnic differences in cancer frequency.

Ethnic and racial differences in cancer incidence, mortality and survival are well documented for many tumor sites. For skin cancer and probably for some other sites such differences have their foundation in biological characteristics that presumably reflect genetically determined differences. In other cases, apparent racial differences in fact reflect and must be distinguished from, associated co--variables including socio--economic and cultural factors.

Ethnicity↗
PubMed1979

Prevalence of tardive dyskinesia.

Forty-four epidemiologic studies on tardive dyskinesia were evaluated as to whether they provided information on diagnostic criteria, objective scale and assessment, interobserver reliability, period of observation, and specific interhospital coordination. Studies which met these standards were reviewed for data on class of neuroleptic therapy, dose, duration, continuity of treatment, extrapyramidal toxicity, spontaneous dyskinesias, other drugs and treatment modalities, age and sex. A higher prevalence of tardive dyskinesia has been consistently noted in the elderly and in females. No other predisposing factors for tardive dyskinesia have been conclusively demonstrated thus far. Prevalence of tardive dyskinesia is estimated at 24-56% in chronic neuroleptic users.

Adolescent↗
PubMed1979

Risks to the offspring from parental occupational exposures.

Risks to the offspring of workers with occupational chemical exosures may derive from mutagenic, teratogenic or carcinogenic effects of industrial agents to which the parents are exposed. Evidence for impaired pregnancies and hazards to the offspring of working populations with chemical exposures is, however, very limited. Perhaps the best documented example is increased spontaneous abortion rates in female operating room personnel who have first trimester exposure to waste anesthetic gases. Evidence is reviewed for hazards to the offspring resulting from parental occupational exposure to vinyl chloride, benzene, chloroprene, radiation and petroleum-derived hydrocarbons. It is essential in investigating the role of occupational factors that other environmental and behavioral factors with major effects on pregnancy outcome be accounted for. These include smoking, alcohol, and drug exposures. An approach to surveillance for chromosomal abnormalities in offspring of occupationally exposed parents is outlined.

Alcohol Drinking↗