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Biomedical subjects

K Gajkowski

Publications and source records attributed to K Gajkowski.

At least 19 recordsLinked to original sources
PubMed1992

[Effect of hemodialysis on somatosensory evoked potentials of medium and long latency].

In 16 patients with terminal renal failure aged from 21 to 74 years maintained on haemodialysis programmes for 2 to 60 months somatosensory evoked potentials were studied after stimulation of the median nerve immediately before and after haemodialysis. The central conduction time, the P 45-N 60, P 100-N 140-P 200 complexes and the P 300 wave were evaluated. The results were compared with those in 16 healthy volunteers aged 20 to 66 years. In the patients on haemodialyses the evoked somatosensory potentials of medium and long latency differed statistically significantly from those in the control group, but not direct effect of haemodialysis on these potentials was found.

Adult↗
PubMed1991

Creutzfeldt-Jakob disease in young people.

Three neuropathologically confirmed cases of Creutzfeldt-Jakob disease in young people (19, 23, and 27 years of age) are described. None had received pituitary hormone therapy. At the onset of illness all patients were suspected of having SSPE or other viral encephalitis, because of the similarity of clinical symptomatology and the shift towards older age of SSPE onsets observed in Poland in recent years.

Adult↗
PubMed1986

Cerebellar atrophy diagnosed by computed tomography and clinical data.

The diagnostic relevance of computed tomography (CT) in the classification of cerebellar atrophy or degeneration is unclear. Twenty-one patients with cerebellar atrophy at CT were studied and the findings were correlated to clinical data. Based upon such data the material was divided into two groups. In the first group (12 patients), with signs of cerebellar deficiency, 6 cases presented familial hereditary ataxia, and olivopontocerebellar atrophy of the Menzl type, 3 ataxia telangiectasia (Louis-Bar syndrome), 2 olivopontocerebellar atrophy of the sporadic type, and 1 adrenoleukodystrophy. In the second group (9 patients), without cerebellar deficit, cerebellar atrophy was found only occasionally. In all of them, there was cerebral atrophy. Clinically manifest cerebellar deficiency and cerebellar atrophy as evident at CT was mainly found in patients with familial genetic disorders. Cerebellar and/or vermian atrophy without clinical signs of cerebellar deficiency were observed only occasionally and were not specific.

Adolescent↗
PubMedDate not supplied

[Surgically treated epidural hematoma of the spinal cord at the Th1-Th2 levels associated with arachnoid cyst].

In a man aged 28 clinical picture, myelography and computerized myelotomography demonstrated epidural haematoma at the level of Th1-Th2 vertebrae with associated arachnoidal cyst. Owing to initially erroneous diagnosis of transverse myelitis the haematoma was diagnosed only after 7 days and removed surgically with simultaneous evacuation of the cyst. The therapeutic result was minimal.

Adult↗
PubMedDate not supplied

[The Tolosa-Hunt syndrome. Case report].

A case of headaches with recurrent paralysis of cranial nerves III, IV, V and VI is reported. A good therapeutic effect was obtained with corticoids. After ruling out other diseases and causes of ophthalmoplegia the Tolosa-Hunt syndrome was diagnosed.

Adult↗
PubMedDate not supplied

[Neurologic symptoms in lithium poisoning].

A 69-year-old female patient is reported who had been treated with lithium salts for bipolar affective disease. During hospital stay due to myocardial infarction in cardiology department signs of intoxication with lithium carbonate developed. Parkinsonian syndrome and amnesia with abnormal EEG tracings were observed. After withdrawal of lithium and correction of water and electrolyte disturbances as well as pharmacological treatment gradual disappearance of neurological abnormalities was observed, with full normalization of EEG tracings.

Aged↗
PubMedDate not supplied

[Familial cerebellar ataxia: clinical, radiological and electrophysiological findings].

A family with cerebellar ataxia of late onset occurring in four generations was observed. Neurological abnormalities included signs of cerebellar ataxia, pyramidal tract damage and damage to the peripheral motor neuron. Computerized tomography demonstrated in five out of six studied patients an image suggesting olivo-ponto-cerebellar atrophy. In the cerebellar structures, brainstem and cerebral hemispheres evidence of atrophy was detected. No correlation was demonstrated between the intensity of the clinical signs and the progression of changes in CT image. Electrophysiological investigations demonstrated changes compatible with damage to the motor and sensory fibres in the peripheral nerves and signs suggesting damage to the spinal motor neurons and pyramidal tract. These observations confirm the multilevel development of the process. The use of similar diagnostic methods will permit a more accurate classification of cerebellar ataxia and obtaining of better information for prognostication of individual cases.

Adult↗
PubMedDate not supplied

[A case of cerebral cysticercosis treated with praziquantel in the postoperative period].

A 51-year-old man with cerebellar signs and raised intracranial pressure had negative indirect and serological tests for cysticercosis. During operation cysticerci were removed from the cisterna magna, fourth ventricle and aqueduct. Histological examination confirmed the diagnosis of cysticercosis. After operation the condition of the patient deteriorated consciousness disturbances, involuntary movements of the left upper extremity and paraparesis appeared. Only after treatment with Praziquantel with simultaneous administration of oedema-reducing agents nearly complete regression of neurological manifestations was achieved.

Cerebral Ventricles↗
PubMedDate not supplied

[Familial occurrence of calcinosis of the basal ganglia].

In two sisters aged 36 and 38 years symmetrical calcifications were found in the vicinity of the pallidum in brain CT. In the younger sister epileptic seizures and transient focal signs were due to arrhythmias of the heart caused by mitral valve leaflet prolapse. In the second case no neurological signs were found. The calcifications were probably genetically determined suggesting an autosomal recessive inheritance. The described cases are another example of familial calcifications in basal ganglia without neurological changes.

Adult↗