PubMed Health⌕ Search

Biomedical subjects

M Pilarczyk

Publications and source records attributed to M Pilarczyk.

32 records · Page 2Linked to original sources

The role of posttraumatic intracranial cysts in the pathogeny of late epileptic seizures as exemplified by two patients.

Intracranial cysts are most often found in the lateral sulcus between the cerebral hemispheres and at the midline in the posterior cranial cavity. Covered with the clear or turbid arachnoidea, they are filled with a clear, colorless or slightly yellow cerebrospinal fluid, sometimes they have a considerable size and tend to be an unexpected discovery during an operation for symptoms of a brain tumor. The symptoms depend on the cyst's location and size. These are: headaches, epileptic seizures, sometimes deficiency signs. Located in the posterior cranial cavity they can imitate a cerebellar tumor (1, 3, 5). Intracranial cysts can be a result of abnormal ontogenesis or sometimes following a head injury (2). Since there is little information in literature on the incidence of posttraumatic intracranial cysts and their clinical picture, we find it desirable to present patients treated at Neurology Clinic of the Medical Academy in Lublin. All patients underwent a neuropsychological examination.

Adolescent↗

[Effect of piracetam on inorganic phosphates and phospholipids in the blood of patients with cerebral infarction in the earliest period of the disease].

The influence of piracetam on the level of inorganic and phospholipid phosphorus in blood of ischemic stroke patients was evaluated. In healthy patients piracetam (2G, i.v.) diminished the concentration of inorganic phosphorus and essentially lowered the content of ion connected with phospholipids. In stroke patients inorganic phosphorus was primarily enhanced and organic lowered. Treatment with piracetam lowered the concentration of both inorganic and phospholipid phosphorus in blood.

Acute Disease↗

[Effect of long-term administration of low-molecular dextran (40,000) on the blood levels of inorganic phosphorus and lipid-bound phosphorus in patients with cerebral infarction].

The effect of low-molecular dextran given on 6 days after cerebral infarction onset on the content of inorganic phosphorus and lipid-bound phosphorus was studied in 22 patients. The obtained results were compared with those in a control group. Low-molecular dextran caused a statistically significant decrease of the serum lipid-bound phosphorus level in healthy subjects and in patients, which was most pronounced on the sixth day of successive dextran administration, while it had no effect on serum inorganic phosphorus in the studied subjects.

Aged↗