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Biomedical subjects

U Brunner

Publications and source records attributed to U Brunner.

At least 37 records · Page 2Linked to original sources
PubMed1989

[Imitation of acute, deep venous thrombosis of the lower leg by a ruptured Baker's cyst].

In case of a swollen leg, differential diagnosis includes deep venous thrombosis. After having ruled out a thrombosis by venography, other causes have to be sought. We report a case where massive swelling of the lower leg was due to the arthrographically proven rupture of a Bakers' Cyst. Had more emphasis been put on recording the patients history, time loss and diagnostic procedures could have been minimized.

Aged↗
PubMed1988

[Trigeminus neuropathy as a rare main symptom of progressive systemic sclerosis].

In addition to the habitual skin manifestations, the following forms of organic involvement were found (with decreasing frequency) in 33 patients with progressive systemic sclerosis treated from 1972-1986 at the Clinic of Rheumatology, University Hospital of Zürich: blood vessels 29, lungs 18, digestive system 11, joints 11, bones 8, lacrimal and salivary glands 7, musculature 3, heart 3, kidneys 2, cranial nerves 1 and peripheral neuropathy 1.-A 65-year-old patient with classic progressive systemic sclerosis and visceral involvement of the esophagus and the lungs showed, as a principal sign, unilateral neuropathy of the trigeminal nerve involving all three branches as well as absent corneal reflex. Electromyographic examination afforded evidence of a sensory neuropathy of the trigeminal nerve. Chemical laboratory tests revealed a typical high titre of antinuclear antibodies. In addition to autoimmunological causes, proliferation of fibrous structures of the perineurium and vascular lesions are discussed in connection with these complications.-The subsequent history of 37 patients with localized scleroderma and 4 patients suffering from progressive systemic sclerosis, treated at the Clinic of Dermatology, University Hospital of Zürich from 1980-1985 provided no evidence of a cranial nerve involvement. The fact that during the period 1975 to 1985 only one patient treated at the Clinic of Neurology of Zürich University Hospital showed signs of disturbances of pupil motility confirms the very rare involvement of the nervous system in progressive systemic sclerosis.

Aged↗
PubMed1987

[Differential indications for surgical and conservative internal medicine procedures in the treatment of acute ischemia of the leg].

Re-opening of the main arterial pathway is the most efficient method for treatment of arterial occlusive disease in stage III and IV. In addition to surgical thromb-embolectomy catheter procedures such as percutaneous transluminal angioplasty, catheter lysis and catheter extraction of fresh clots may give satisfactory results. The following methods are indicated in acute cases: catheter procedures for occlusions of the popliteal trifurcation and profound femoral artery, segmental occlusions of the superficial femoral artery, femoro-popliteal occlusions with impaired run-off, recurrent occlusions after desobliteration with both surgical methods, occlusions of lower arm arteries; surgical thrombectomy for occlusions of the aortic bifurcation, the iliac arteries, the femoral bifurcation, for femoro-popliteal aneurysms and for occlusions of the subclavian and axillary artery. Both procedures do not exclude each other and may be combined according to the relevant indications.

Angioplasty, Balloon↗
PubMed1987

[Compression syndromes of the shoulder and their differential diagnosis].

Compared to other painful conditions on the shoulder suprascapular nerve entrapment is an obscure and uncommon syndrome causing severe shoulder pain and disability, and is easily cured if only it is recognized. The condition was described by Thompson and Kopell in 1959, Schilf reported a case of isolated suprascapular entrapment in 1952. The nerve passes through the suprascapular notch, the roof of the notch is formed by the transverse scapular ligament. The shape of the suprascapular notch may be guilty for entrapment symptoms. Suprascapular nerve compression may result of acute trauma, of transmitted forces, fracture of collum scapulae, of kinking or traction of the nerve over the edge of the foramen, of sling effect, of compression by ganglia, or its cause may be idiopathic. The hallmark of suprascapular nerve entrapment is a deep and poorly delineated pain, localized to the posterior and lateral aspect of the shoulder. Atrophy and weakness of the supraspinatus and infraspinatus may be noted. Adduction of the extended arm across the body tenses the nerve, increasing the pain. Blocking the nerve by local anesthetic a dramatic, but not long lasting pain relief may be achieved. Electromyographically a decrease in the amplitude or marked polyphasicity of evoked potentials is significant as well as an increased latency time, indicating an impaired conductibility. A surgical release is indicated in case of constant pain and pathological changes of EMG-patterns. From a postero-superior approach decompression of the nerve is performed by simple removal of the transverse scapular ligament.

Diagnosis, Differential↗
PubMed1986

Juvenile arteritis of extracranial arteries with hypereosinophilia.

The case history of a 23-year-old man presenting with a progressive tender swelling in both temporal regions is described. Eosinophilia varied between 28% and 48% and IgE was markedly increased (1,380 U/l). Arteriography revealed tortuous, enlarged temporal and occipital arteries intersected by stenoses. The pathology diagnosis was chronic necrotizing panarteritis of the temporal artery with pseudoaneurysms, fibrinoid necroses, scar tissue, occasional giant cells, and abundant hypereosinophilia.

Adult↗
PubMed1986

[Possibilities for vascular reconstruction in the post-thrombotic syndrome].

Reconstructive surgery of deep vein thrombosis is technically established. Results, however, are limited by the evolutive character of thrombotic processes and by the permanently impaired hemodynamics in veins with a postthrombotic condition. Long-term patency and functionally satisfactory reflux are not regularly achieved. Best suited are locally limited interventions in the main confluent venous trunks of the pelvic area, the groin and the popliteal groove. It ought to be kept in mind that every deep vein thrombosis simultaneously impairs the deep and superficial lymphatics. Surgical procedures in deep vein thrombosis should therefore try to prevent any further damage to lymph vessels.

Humans↗
PubMed1986

[CT demonstration of an entrapment syndrome of the popliteal artery].

In a 43-year old patient, in whom a transluminal revascularisation of an occluded popliteal artery had ben performed, computed tomography showed a popliteal artery entrapment syndrome. The popliteal artery coursed on the medial side of a lateralised, medial gastrocnemius muscle and was also compressed from dorsally by an accessory muscle that extended from the medial femoral condylus to the medial gastrocnemius muscle. The findings were confirmed by an operation.

Adult↗