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Biomedical subjects

V M Sedov

Publications and source records attributed to V M Sedov.

At least 91 records · Page 5Linked to original sources
PubMed1981

[Visceral circulatory state in experimental stenosis of the celiac trunk].

Considerable disturbances of the visceral blood circulation have been found to occur in stenosis of the celiac trunk. Circulatory disorders in the liver, pancreas, stomach and duodenum are dependent on the degree of the celiac trunk narrowing. The collateral vascular bed fails to provide the complete compensation of the reduced organic blood flow. As a result, chronic ischemia of the organs persists.

Animals↗
PubMed1979

[Cancer of the gallbladder].

Clinical observations in 25 patients with cancer of the gallbladder are described. Tumor of this localization, as a rule, is characterized by a latent course with symptoms of cholelithiasis, chronic cholecystitis, manifested by jaundice and signs of neoplasia only ar later stages. Special attention is given to a frequent association of gallbladder cancer with an acute inflammatory process. The prophylaxis against gallbladder cancer is early surgery for cholelithiasis.

Aged↗
PubMed1978

[Results of surgical treatment of acute gastro-duodenal ulcers and erosions complicated by profuse hemorrhage].

25 cases were operated upon at the climax of a massive gastrointestinal bleeding from acute ulcers and erosions. Among them 20 patients underwent stomach resection. One patient died from pancreonecrosis within the early postoperative period. There were no cases of the recurrence of bleeding. Excellent late results were found in 12 of 17 cases, in other 5 cases the results were good.

Acute Disease↗
PubMed1977

[Diagnosis and treatment of internal biliary fistulas].

In the paper the diagnostic and therapeutic policy employed in 11 patients with internal biliary fistulas, most frequently caused by cholelithiasis, is analysed. Some aspects of the preoperative diagnosis are eluciadated, and it is concluded that patients with internal biliary fistulas are to be treated surgically: cholecystectomy, suturing of defects in the gastric and intestinal wall, the drainage of bile tracts.

Aged↗
PubMed1976

[Regeneration of the gastric mucosa under conditions of massive blood loss].

The autoradiographic method was used for a comparative study of regeneration of the gastric mucosa after wounding the rat with a preliminary loss of blood and without it. The H3-thymidin label index was determined in the portions of the mucosa of the wound edge and far from it. A histological analysis of healing the wounds failed to reveal any difference in terms of this process in rats with a loss of blood and without it. A momentary massive loss of blood did not influence the strain and intracellular regeneration processes in the healing wound of the stomach: the amount of cells synthesizing DNA at the edges of the gastrotonic wound proved to be the same in experiments both with a loss of blood and without it. The authors believe that it may serve as an indirect evidence of a somewhat exaggerated danger of surgery at the height of gastroduodenal bleeding.

Animals↗
PubMed1975

[Bone marrow reaction to surgical intervention in gastroduodenal hemorrhage].

Experimentally and clinically, the authors have studied the effect of massive blood loss and various operative procedures (Billroth I and Hofmeister-Finsterer gastrectomies, pyloroplasty with vagotomy) on the hemopietic system. It has been found that the bone marrow response was mainly dependent on blood loss. A surgical intervention renders but insignificant effect on hemopoiesis, similar to that of a moderate hemorrhage, the character of an operative procedure being of no improtance.

Adaptation, Physiological↗
PubMedDate not supplied

[Oxidation of low density lipoproteins in the presence of silica gel treated with fullerene].

This study estimated the effects of low density lipoprotein (LDL) oxidation in the presence of fullerene silicagel in comparison with ions of Cu+2 in the wide region of oxidation times and reagent concentrations. Investigation is directed to reveal the reagent parameters, that promote to increase the oxidation rate of the LDL solution. It revealed only qualitative coincidence of the LDL oxidation kinetics in the presence as Cu+2 as fullerene, that indicated on the identity of the oxidation mechanism in both cases. At the same time the revealed quantitative difference between the oxidation process parameters are interpreted in the terms of oxidation potential and the electron affinity that characterize the reagent activity in the reaction accompanied by the electron transfer. The good correlation between above mentioned parameters and the oxidation rate of the aqueous LDL solution is observed: its increasing takes place at the increase of the electron affinity or at the decrease of the oxidation potential.

Carbon↗