[Characteristics of cyanide-sensitive and cyanide-resistant respiration in the brain in myocardial ischemia and the significance of emotional stress in their occurrence].
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Animals↗
Biomedical subjects
V V Davydov
Publications and source records attributed to V V Davydov.
[Oxidative phosphorylation in the cerebral hemispheres in experimental myocardial infarct].
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Animals↗
[Effect of nootropil on the conditioned reflex activity in acute myocardial infarct].
At the acute stage of myocardial infarction the higher functions of the brain such as fixation and reproduction of temporary connections were shown to be markedly depressed. The use of nootropil in experimental myocardial infarction was followed by the improvement of conditioned-reflex activity.
Animals↗
[Calcium metabolism and 3',5'-AMP levels in various organs in emotional-pain stress].
Emotional stress caused distinct alterations in calcium metabolism and hypocalcemia development was observed. At the same time, negative balance of calcium, redistribution of calcium in tissues as well as alteration of the 3; 5'-AMP content in the tissues were noted.
Adenosine Monophosphate↗
[Effect of adrenaline and acetylcholine on the phosphatidylserine decarboxylase activity of the heart].
The authors have established the presence of phosphatidylserine decarboxylase in the sarcolemma. The capacity of this enzyme to change under the influence of adrenaline and acetylcholine has been brought to light.
Acetylcholine↗
[Protein metabolism in the cerebral hemispheres during the emotional-algesic stress].
Emotional-algesic stress causes essential changes in the protein metabolism of cerebral hemispheres. These changes may be of great importance for the functioning of the brain and cause the disturbances of the higher nervous activity when the organism is influenced by the emotional stress factors.
Animals↗
[Energy metabolism in the cerebral cortex in necrosis of the myocardium].
In ischemic necrosis of myocardium inhibition of energy metabolism occurred in brain, as a result of which an impairment of the brain functional state might take place. Emotional stress, preceding the myocardium necrosis, augmented the impairments of energy metabolism in brain hemispheres.
Adenine Nucleotides↗
[Glucocorticoid function following removal and autotransplantation of the adrenal glands and its alteration under the influence of stress in rats].
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Adrenal Cortex Hormones↗
[Local changes in experimental burn cachexia].
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Animals↗
[The significance of changes in cerebral carbohydrate-phosphorus metabolism in the pathogenesis of burn cachexia in rats].
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Adenosine Triphosphate↗
[Secretion by the adrenal cortex and blood 11-hydroxycorticosteroid levels in different regions following blood loss].
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11-Hydroxycorticosteroids↗
[Alteration of Ca2+, Mg2+-ATPase activity from rat hypothalamus and myocardium in development of the neurotic state].
Simultaneous decrease of Ca2+, Mg2+-ATPase activity occurred in rat hypothalamus and myocardium during experimental neurosis. The decrease of the enzymatic activity was more distinct in hypothalamus than in myocardium. In hypothalamus the decreased enzymatic activity maintained within a week after completion of the excitation of the animals.
Animals↗
[Phospholipids of mitochondria and the activity of their membrane-bound enzymes in development of myocardial necrosis in rats after stress].
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Adenosine Triphosphatases↗
[Na K-dependent ATPase activity of synaptosomes of rat cerebral hemispheres with ischemic necrosis of the myocardium, occurring after emotional-painful stress].
Within 2 days after ligation of the descending branch of left coronary artery activity of Na, K-dependent ATPase was inhibited in heavy synaptosomal fraction of brain hemispheres of rats. Emotional-painful stress, which developed before simulation of acute occlusion of coronary artery, prevented the enzyme inhibition.
Adenosine Triphosphatases↗
[Activity of Krebs cycle dehydrogenase and tissue respiration enzymes in cerebral hemispheres of rats under stress].
It is established that emotional-painful stress and myocardium necrosis evoked in animals after the emotional-painful stress is accompanied by a disturbance in the creative metabolism and partial inhibition of cytochrome oxidase in cerebral hemispheres. A conclusion is made that the found changes may be one of the pathogenetic factors in formation of the neurosis-like states under stress and diseases originated from the effect of the emotional-psychic overstrain.
Animals↗
[Formation, fixation and reproduction of temporary connections in the rat after experimental myocardial infarction].
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Animals↗
[Creatine phosphate content and creatine phosphokinase activity in the cardiomyocyte mitochondria in myocardial infarct in rats subjected to emotional and pain stress].
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Animals↗
[Glycolysis in the myocardium during necrosis induced after the exposure to stress].
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Animals↗