[Cooperation between private dentist and dental clinic in the treatment of children with dentist-aversive behavior].
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Biomedical subjects
Publications and source records attributed to W E Wetzel.
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On the basis of the experience gained with 65 children trying to resist dental treatment, the problems causing and influencing this behavior of children having an aversion against the dentist are presented. Apart from factors resulting from specific situations or specific ages, factors were found that are due to parental education, social environment, and serious psychical disturbances of the child.
A fifteen-year-old boy with osteogenesis imperfecta (OI) and dentinogenesis imperfecta also had a big cyst in the mandible and needed surgical therapy. Six months postoperatively we saw a complete regeneration of the bone-structures. We came to the conclusion that cysts which appear independently from the disease of OI can heal after surgical intervention.
Our investigating 186 infants between the ages of one and six with carious destruction of the maxillary primary incisors, it was learned which risk factors were responsible for the condition known as nursing bottle syndrome. One hundred and twenty-eight infants (68.8 percent) were given a nursing bottle, twelve (6.5 percent) a feeding cup or other bottles with bill- shaped extensions, and forty-one (22.0 percent) both a nursing bottle and vessels with bill-shaped extensions; in all cases the feeding was excessive and prolonged beyond the first year of life. An additional five infants (2.7 percent) were breast-fed excessively beyond the first year. The results confirm the risk of tooth destruction, typical of nursing bottle syndrome, by prolonged and frequent consumption of cariogenic beverages from vessels with bill-shaped extensions. It is important, therefore, that a warning regarding dental health hazards of such feeding methods be issued.
The altered structure of "Turner-teeth" was examined under the scanning electron microscope. The enamel surface revealed developmental disorders such as bowl-shaped pits, Tomes' process pits and enamel craters with rounded borders. On the fractured surfaces of yellowish discolored enamel, localized islands of pathological enamel were found. The enamel rods were atypically arranged and poorly mineralized. Surprisingly the prisms formed later by the same ameloblast often exhibited a entirely normal structure.
Three sisters at the ages of seven months, twelve years, and thirteen years presented with the initial damages to the oral tissues and the distinctive long-term effects in conjunction with the congenital analgia syndrome. The severity of this syndrome justifies the consideration of a prophylactic extraction of the primary dentition. A controlled mastication will be more likely with increasing age and eruption of the permanent teeth.
The findings are based on a clinical investigation conducted on forty-nine patients suffering from osteogenesis imperfecta (OI), as well as on a questionnaire study in which 117 osteogenesis imperfecta-affected persons or their parents were involved. The survey established pathological tooth discolorations as well as tooth abrasions. Dentinogenesis imperfecta (DI) was more frequently found in primary teeth than in permanent teeth. There were no gender-specific differences. Radiological abnormalities were found in both, abraded and/or discolored teeth, as well as in clinically normal appearing teeth. In most cases there were club-shaped extensions of the pulp chambers and obliterations of the root canals. The probability that dentinogenesis imperfecta occurs as an accompanying symptom of osteogenesis imperfecta was not dependent on the degree of skeletal severity. The self-assignment according to A and B forms of osteogenesis imperfecta types I and IV in accordance with the presence/absence of dental symptoms was contradictory, since the literature was based on varying classifications.