Malignant glaucoma after diode laser cyclophotocoagulation.

A Azuara-Blanco; H S Dua

doi:10.1016/s0002-9394(98)00359-6

Home/Archive/PubMed

PubMed · 10218707

Malignant glaucoma after diode laser cyclophotocoagulation.

A Azuara-Blanco·H S Dua

View original source ↗Export metadata ↓

Abstract

PURPOSE: To report a case of malignant glaucoma after diode laser cyclophotocoagulation. METHOD: Case report. RESULTS: A 45-year-old man with uncontrolled secondary glaucoma in his right eye after corneoscleral graft and cataract extraction underwent diode laser cyclophotocoagulation. The right eye was aphakic, with an intact posterior capsule. Two weeks later, the patient presented with blurred vision, edematous cornea, and flat anterior chamber. The posterior capsule was touching the endothelium. Intraocular pressure was 20 mm Hg. Repeated Nd:YAG laser capsulotomy was effective in reversing the malignant glaucoma attack, and the anterior chamber deepened. CONCLUSION: Malignant glaucoma can occur after diode laser cyclophotocoagulation.

Explore related subjects

Anterior Chamber Burns, Chemical Cataract Extraction Ciliary Body Corneal Transplantation Eye Burns Glaucoma Humans Intraocular Pressure Laser Coagulation Lens Capsule, Crystalline Male Middle Aged Reoperation Sclera Case Reports Journal Article

Keep this discovery

Explore connections, maps & timelines

BibTeX RIS

A Azuara-Blanco, H S Dua. 1999. Malignant glaucoma after diode laser cyclophotocoagulation.. https://doi.org/10.1016/s0002-9394(98)00359-6

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed2007

Modified technique for management of LIDRS.

Explore the source record for details and available documents.

Anterior Chamber↗

PubMed2007

Errata in printed Hoffer Q formula.

Explore the source record for details and available documents.

Anterior Chamber↗

PubMed2007

Model of pulsatile-flow of aqueous humor through the iris-lens canal.

PURPOSE: To present a model of pulsatile-flow of aqueous humor from posterior (PC) to anterior chamber (AC) and to analyze the sensitivity of this novel model in detecting typical high risk conditions predisposing to pupillary block. METHODS: The model assumes noncontinuous flow of aqueous through the iris-lens canal. Aqueous that fills the canal will be ejected toward the AC-side of the canal at certain time intervals, and between 2 events of aqueous ejection there is no actual flow through this canal. Pupillary pumping rate (PPR) was calculated from the aqueous flow rate and the calculated volume of iris-lens canal. RESULTS: PPR values were generated by incorporating pupillary diameter (1 to 8 mm), aqueous flow rate (1 to 2.5 microL/min), and iris-lens canal width (0.5 to 2 mm) and height (3-9 microm) in numerical experimentation with the present model. PPR showed inverse dependence on iris-lens canal height and pupillary diameter and was directly proportional to aqueous flow rate, in agreement with the steady-flow model. However, contrary to the steady-flow model, PPR showed inverse dependence on iris-lens canal width and predicted the anticipated PC-AC pressure gradient changes at simulated light-dark transition in eyes of patients with clinically narrow angles and ultrasound biomicroscopy evidenced pupillary block. CONCLUSIONS: Upon the incorporation of real ultrasound biomicroscopy measurements in numerical experimentations with both models, the present pulsatile-flow model, contrary to the steady-flow model, showed good predictability of PC-AC pressure gradient changes in a typical condition predisposing to pupillary block.

Anterior Chamber↗