PubMed · 14168849
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F A JONES. 1964. BARIUM PROCTITIS.. https://pubmed.ncbi.nlm.nih.gov/14168849/
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Sediments play an important (but still poorly understood) role in the dispersion and final fate of radionuclides and other hazardous materials in aquatic systems. Adopting an experimental point of view, the present work deals with the transfer of a radioactive tracer from water to sediments. Sediments and waters were sampled in the Odiel and Tinto estuaries (South-West Spain) with anthropogenic-enhanced 226Ra concentrations. 133Ba was used as a tracer since it is a gamma emitter and a good analogue of the environmental behaviour of 226Ra. Laboratory experiments have been carried out to quantify the uptake kinetics of 133Ba by sediments in aqueous suspensions and by sediment cores under a water column at rest. Depth distributions of 133Ba in sediments have been studied with different contact times and using sediment samples with different grain sizes. The results reveal an important and fast uptake by suspended sediments (up to 40% in 10 min with a 20 g l(-1) suspension) and sediment cores (up to 70% in a few minutes). The kinetics of the uptake by suspended sediments could be reasonably described by a model of two parallel and reversible reactions followed by a weakly-reversible reaction. The total uptake and the rate of reaction decreased with the increment of grain size. Furthermore, uptake following diffusion through the interstitial water was hardly detectable beyond 1 cm depth. For the case with more experimental results, the depth distribution could be reasonably described by the analytical solution based on the use of an effective diffusion coefficient (4.2 x 10(-12) m2 s(-1)) and the measured intrinsic distribution coefficient (kd = 63 +/- 6 l kg(-1)).
Congenital diaphragmatic hernias, including Morgagni s hernia, usually present in early childhood and are treated by surgical repair. This case report is about an unusual Morgagni s hernia, presenting with dyspepsia and chest pain, at the age of 45 years. For many years the diagnosis remained a dilemma because patient s chest x-ray was not done and she was treated for "angina " and "dyspepsia". Diagnosis was obvious once a chest x-ray was done, however, barium studies were performed for further confirmation.
OBJECTIVE: Activity of single L-type calcium channels (LTCC) is enhanced in human failing myocardium (Circulation 98 (1998) 969.), most likely due to impaired dephosphorylation. Protein phosphatase 2B (calcineurin) has recently been shown to be involved in heart failure pathophysiology. We now focus on the regulation of single LTCC by calcineurin that were prevented by Ca(2+)-free experimental conditions in our previous study. METHODS: Single LTCC currents were recorded in myocytes from human atrium and ventricle. Charge carriers were 70 mM Ba(2+), or a mixture of 30 mM Ca(2+) and 60 mM Ba(2+) to facilitate Ca(2+) permeation through recorded channels. The calcineurin inhibitor cyclosporine (10 microM) was used to reveal a putative role for calcineurin in regulation of LTCC. RESULTS: A mixture of Ca(2+) and Ba(2+) as charge carriers allowed for Ca(2+) permeation through recombinant human embryonic kidney cells and native (atrial and ventricular) human cardiac LTCC. With only Ba(2+) as the charge carrier, activities of both ventricular and atrial LTCC were strongly decreased by cyclosporine. In contrast, channel activity remained constant when Ca(2+) permeation was provided. In the presence of thapsigargin and (S)-BayK 8644, cyclosporine here even increased channel activity. CONCLUSIONS: We propose a dual cyclosporine effect on human cardiac LTCC. A non-specific inhibitory effect prevails with Ba(2+) permeation but can be compensated or overcome by a specific Ca(2+)-dependent stimulation with Ca(2+) permeation. More complete restoration of physiological Ca(2+) movements (e.g., Ca(2+) release from sarcoplasmic reticulum) will help to define even more precisely the involvement of calcineurin in regulation of human cardiac LTCC.