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PubMed · 1520666

[Open-angle glaucoma yesterday and today].

P Cernea
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Abstract

Glaucoma with open angle is now considered as an early optical neuropathy, that is determined by an uneasiness in flowing of the aqueous humour at the trabecular level. The ocular hypertension is the most frequent manifestation of the evolution of glaucoma, but it is not obligatory because there is a glaucoma with open angle that develop with low pressure. The automatically perimetry and the photography of the retinal arcuate fibers in aneritre light, may evidence incipient lesions before increasing of the ocular tension. Stereophotographies of the optic papillae make possible the calculus of the neuroretinal ring surface that replaces the old ratio cup/disk. The investigation of the microcirculation of the papillae, the determination of the papillar blood tide by pneumotonography and the velocimetry laser Doppler have brought new data for determining the incipient lesions of the optic disk. The large utilization of beta-blockings has changed the life of a glaucoma patient and the filtrant surgery has extended and diversified so being prolonged the visual function.

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Glaucoma, Open-AngleHumansPedigreePrevalenceRisk FactorsVisual Field TestsEnglish AbstractJournal ArticleReview

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P Cernea. [Open-angle glaucoma yesterday and today].. https://pubmed.ncbi.nlm.nih.gov/1520666/

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PubMed2006

Screening for prevention of optic nerve damage due to chronic open angle glaucoma.

BACKGROUND: Open angle glaucoma (OAG) is a primary, progressive optic neuropathy; the onset is without symptoms and progression occurs silently until the advanced stages of the disease, when it affects central vision. The blindness caused by OAG is irreversible. It has often been assumed to be a condition that fulfils the criteria for population screening, although this has not been supported by other in-depth non-systematic reviews. The focus of this review was to examine the evidence for the effectiveness of screening for OAG. OBJECTIVES: To determine the impact of screening for OAG compared with opportunistic case findings or current referral practices on the prevalence of and the degree of optic nerve damage due to OAG in screened and unscreened populations. SEARCH STRATEGY: We included any randomised controlled trial (RCT) evaluating population-based screening programmes for OAG with a minimum one year follow up. We searched the Cochrane Central Register of Controlled Trials (CENTRAL) on The Cochrane Library (which contains the Cochrane Eyes and Vision Trials Register) (Issue 1, 2006), MEDLINE (1950 to February 2006) and EMBASE (1988 to February 2006). We also searched the National Research Register (Issue 1, 2006) and Zetoc for grey literature (29 June 2006). There were no language or date restrictions in the electronic searches. SELECTION CRITERIA: We planned to include RCTs, including cluster RCTs. DATA COLLECTION AND ANALYSIS: Two review authors independently assessed the study abstracts identified by the electronic searches. We did not find any trials that met the inclusion criteria. MAIN RESULTS: As no trials were identified, no formal analysis was performed. AUTHORS' CONCLUSIONS: On the basis of current evidence, population-based screening for chronic OAG cannot be recommended, although much can be done to improve awareness and encourage at risk individuals to seek testing. In wealthy countries with equitable access to high quality eye care and health education, blindness from chronic OAG should become increasingly rare; much greater challenges face poor and emerging economies and countries where there are substantial health and wealth inequalities. Effectiveness of screening for OAG can be established only by high quality RCTs.

Glaucoma, Open-Angle↗
PubMed2006

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