Interocular blur suppression and monovision.

M J Collins; A Goode

doi:10.1111/j.1755-3768.1994.tb02777.x

Home/Archive/PubMed

PubMed · 7976272

Interocular blur suppression and monovision.

M J Collins·A Goode

View original source ↗Export metadata ↓

Abstract

The ability of the binocular system to suppress blur from the defocused eye is an important mechanism underlying the success of monovision correction. With a group of 60 new monovision wearers, we found that blur suppression ability varied substantially between subjects and that crossed dominance influenced blur suppression ability. Symptoms of ghosting amongst the monovision wearers appeared to be influenced by the strength of rivalry dominance. An understanding of the suppression processes underlying monovision may assist in patient selection and management.

Explore related subjects

Anisometropia Contact Lenses Dominance, Cerebral Humans Presbyopia Pupil Vision Disparity Vision, Monocular Journal Article Research Support, Non-U.S. Gov't

Keep this discovery

Explore connections, maps & timelines

BibTeX RIS

M J Collins, A Goode. 1994. Interocular blur suppression and monovision.. https://doi.org/10.1111/j.1755-3768.1994.tb02777.x

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

PubMed2007

Phakic IOL in children.

Explore the source record for details and available documents.

Anisometropia↗

PubMed2006

Anisometropia and migraine: is the link to cortical spreading depression logically defensible?

Explore the source record for details and available documents.

Anisometropia↗

PubMed2006

Development of astigmatism and anisometropia in preterm children during the first 10 years of life: a population-based study.

OBJECTIVE: To assess the development of astigmatism and anisometropia to 10 years of age in preterm children, previously included in a population-based study on the incidence of retinopathy of prematurity. METHODS: Cycloplegic retinoscopies were performed in 198 preterm children at 6 months, 2(1/2) years, and 10 years of age. We analyzed the development of astigmatism of 1 diopter (D) or more and anisometropia of 1 D or more. RESULTS: The amount and prevalence of astigmatism declined between 6 months and 2(1/2) years of age and then remained stable. We found no difference in the course of astigmatism at different ages with regard to stage of retinopathy of prematurity. The amount of anisometropia increased, but its prevalence remained unchanged. Multiple regression analyses showed that astigmatism of 1 D or more at 2(1/2) years of age and cryotreated severe retinopathy of prematurity were risk factors for astigmatism at 10 years of age, and that anisometropia of 2 D or more at 2(1/2) years of age was a risk factor for anisometropia at 10 years of age. CONCLUSIONS: The development of astigmatism and anisometropia showed a similar course, regardless of stage of retinopathy of prematurity. The retinoscopy findings at 6 months of age were of no value in predicting astigmatism and anisometropia at 10 years of age, but the refraction at 2(1/2) years of age was. Retinoscopy at about 2(1/2) years of age in all preterm children may be useful for detecting astigmatism and anisometropia that will persist in children of school age.

Anisometropia↗