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[Lung embolism in a cow with pus forming mastitis].
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The challenge of controlling mastitis.
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[Biochemistry, serology, pathogenicity and antibiotic resistance of an Escherichia coli strain from healthy sows and from animals with mastitis-metritis-agalactia syndrome].
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Estimation of the costs of mastitis, using National Animal Health Monitoring System and milk somatic cell count data.
Data collected by the National Animal Health Monitoring System in Ohio for a 12-month period during 1986 and 1987 were used to determine the relative magnitude of costs associated with mastitis in the following categories: milk production loss, veterinary services, drugs, producer labor, and "other" factors. The cost of milk loss associated with mastitis that was reported by producers cooperating in the National Animal Health Monitoring System program was compared with estimates based on bulk tank somatic cell counts and individual cow milk somatic cell counts. Using producer-reported estimates, milk loss accounted for about one third of the total cost associated with mastitis. When estimates of milk loss were replaced by estimates based on bulk tank somatic cell counts, milk loss accounted for over 80% of the total cost of mastitis. Estimates of the cost of milk loss based on studies relating milk yield to somatic cell counts differed considerably. Consequently, it was unclear how to best estimate the relative magnitude of the milk loss component of mastitis costs.
Report of the panel of the colloquium on bovine mastitis.
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[UGCN warns for Klebsiella mastitis].
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Cryptococcal mastitis in dairy animals.
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Influence of liver fat on experimental Escherichia coli mastitis in periparturient cows.
Eleven cows with a wide range of liver fat (5.7 to 51.4 per cent) at seven days post partum were experimentally infected in a single quarter with a capsular Escherichia coli at 10 days post partum. The results suggested that a fatty liver in itself does not influence the severity of mastitis. All animals had clinical mastitis 10 hours after infection but no animals became severely ill and no treatment was given. Four out of five animals in the group with less than 20.2 per cent liver fat had bacteria in their milk at 10 hours after infection but these bacteria were eliminated by 12 hours. The six animals in the group with more than 28.3 per cent fat in their liver retained viable bacteria in the udder for much longer; with two animals bacteria were shed and abnormal milk was secreted for up to four months despite antibiotic therapy.
[Giant cells in milk sediment as a diagnostic aid in the determination of tuberculosis of the udder and of brucellar mastitis].
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[Mammary tip condition scoring--case report of clinical mastitis].
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[Epithelial-cell metaplasia in the udder of a cow with chronic Staphylococcus mastitis].
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[Pathogenicity and biochemical activity of staphylococci isolated from udders of cows belonging to large herds affected with prolonged mastitis].
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Low bulk milk SCC and toxic mastitis.
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