[Absence seizure--recent physiopathologic data].

D Cuciureanu; Felicia Stefanache

PubMed · 14755927

[Absence seizure--recent physiopathologic data].

Abstract

The progresses in clarifying the normal and pathologic cellular and molecular mechanisms are reflected in the elucidation of the way some of the most common forms of generalized seizure--absence seizures--occur and are produced. Intrinsic properties of the thalamic neurons that give them the ability to release or preserve oscillatory, low-frequency neuronal discharges, and the thalamo-cortical feedback mechanism seem to explain the pathogenesis of absence seizures. The involvement of GABA receptors in the regulation of membrane calcium channels, as well as their genetically-induced changes are new pieces in the pathogenic puzzle.

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Animals Cerebral Cortex Child Epilepsy, Absence Feedback, Physiological Humans Mice Receptors, GABA Thalamus English Abstract Journal Article Review

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D Cuciureanu, Felicia Stefanache. [Absence seizure--recent physiopathologic data].. https://pubmed.ncbi.nlm.nih.gov/14755927/

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