PubMed HealthSearch

PubMed · 7174590

Infant formula physical stability.

Abstract

The physical nature of liquid infant formula products is characterized in terms of emulsion, suspension, and solution processes. Such mixtures inevitably undergo phase separation. A variety of physical and chemical phenomena govern the gradual changes in the appearance of these products. The consequent loss of physical elegance is primarily a question of consumer acceptance. Only in the most extreme cases do the physical characteristics deteriorate to the point that swallowing and digestion are affected. Recently, both public and regulatory attention has been focused on the physical characteristics of liquid infant formula products. Formula appearance is the product aspect most readily evaluated by the consumer. The design and manufacture of products that will have consistent appearance over extended periods is a difficult technical problem. This presentation summarizes the phenomena responsible for changes in the physical characteristics of liquid infant formula products. Product defects are discussed and methods for product evaluation are described. Representative data on the effect of storage on product physical elegance also are presented.

Explore related subjects

Keep this discovery

Explore connections, maps & timelines

BibTeXRIS

J P Miles. 1982. Infant formula physical stability.. https://pubmed.ncbi.nlm.nih.gov/7174590/

Cite the original work for its findings. Save a collection to share your selection of sources.

KEEP EXPLORING

Related citations

Metabolic interactions between glucose and fatty acids in humans.

In vivo energy production results largely from the oxidative metabolism of either glucose or fatty acids. Under diverse physiologic and nutritional conditions, the oxidation of either glucose or fatty acids may predominate. The nature of the control of the availability and oxidation of each substrate has been studied extensively for > or = 30 y. The most popular and enduring hypothesis was proposed by Randle et al in 1963 and is termed the glucose-fatty acid cycle. This proposal places great significance on the regulation of lipolysis as a factor controlling substrate metabolism. Our work has led to an opposite perspective, which could be called the glucose-fatty acid cycle reversed. According to our hypothesis, the rate of glycolysis, determined by the intracellular availability of glucose-6-phosphate, is the predominant factor determining the rate of glucose oxidation. Whereas the rate of lipolysis may have some effect on the availability of glucose, both via a fatty acid-mediated inhibition of plasma glucose uptake and also by supplying glycerol for gluconeogenesis, there is little evidence for a direct inhibitory effect of fatty acid oxidation on the intracellular oxidation of glucose. In contrast, increased glucose oxidation limits oxidation of long-chain fatty acids directly by inhibiting their transport into the mitochondria. Consequently, whereas there is a close coupling between glucose availability and oxidation, fatty acids are generally available in greater quantities than are required for oxidation. We propose that fatty acid oxidation is largely controlled at the site of oxidation, which is in turn determined by the availability of glucose, rather than by its availability via lipolysis.

Dietary Carbohydrates