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PubMed · 7023795

Resurfacing the burned face.

Abstract

Facial burns cause deformities through the permanent effects of wound contracture and scar hypertrophy; they are related to the depth of the burn. The depth of an acute burn can be accurately ascertained only by observation over a period of time. Facial skin burns that do not heal by three weeks are subject to increasingly permanent deformity with the passage of time. The best defense against wound contraction and hypertrophic scar is early and complete wound closure. Early and late grafting must be completely successful in "take" to avoid unsightly irregular scarring; therefore, the receptor bed must be meticulously prepared for optimal take. Both early and late grafts are patches that flatten normal contour. However, if the skin graft conforms to a position in the facial mosaic (aesthetic unit of the face), or some subunit that is harmonious with normal facial lines, the patch of proper color can give a more aesthetic result. Nonreproducible anatomical structures such as the margins of the lids, lips, and nostrils should be preserved whenever found to be normal or only partially destroyed. To minimize the distorting effects of wound contracture, pressure therapy of the face and neck, as well as the positional splinting of the neck, must be maintained for many months following grafting and epithelialization until the deleterious wound forces abate. Burns of mobile structures, such as lids and lips, do not lend themselves to control by pressure, and frequently must be managed secondarily by first totally removing scarred tissues and then applying thick skin grafts of the best color match.

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BibTeXRIS

R L Warpeha. 1981. Resurfacing the burned face.. https://pubmed.ncbi.nlm.nih.gov/7023795/

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Blood transfusions are associated with increased risk for development of sepsis in severely burned pediatric patients.

OBJECTIVE: To determine the risk of developing sepsis following transfusion of blood products in severely burned pediatric patients. DESIGN: Retrospective, cohort study. SETTING: Shriners Hospital for Children and University Hospital. PATIENTS: Severely burned pediatric patients with >30% total body surface area (TBSA) burn. INTERVENTIONS: None. MEASUREMENTS AND MAIN RESULTS: Two hundred seventy-seven pediatric burn patients over a period of 7 yrs (1997-2004) were included in the study, with 25 patients being septic at admission and therefore excluded. Patients were stratified according to TBSA burn and presence or absence of inhalation injury. The amounts of packed red blood cells (RBCs) and fresh frozen plasma (FFP) were recorded during hospital stay before the development of sepsis. Blood product administration was normalized for the number of surgeries and divided into two groups: high (RBCs>20/FFP>5) or low (RBCs<20/FFP<5) amount of blood products. Sepsis was diagnosed based on the criteria set by the Society of Critical Care Medicine in conjunction with positive blood culture or presence of organisms in the organs at autopsy. By stratifying the groups into low and high blood transfusion, we found that patients with >60% TBSA burn with inhalation injury have an 8% risk of developing sepsis in the low RBC group, which increases to 58% in the high RBC group (p<.05). Similar results were found for RBCs per operation, FFP, and FFP per operation (p<.05). There were no differences in age and gender between groups. CONCLUSIONS: Pediatric burn suffering from a 60% TBSA burn with concomitant inhalation injury are more likely to develop sepsis if they are given high amounts of blood products, indicating an immunocompromised state following blood transfusion.

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